I argue against a priori objections to the view that causation may be reducible to some micro-structural process in principle discoverable by physics. I distinguish explanation from causation, and argue that the main objections to such a reduction stem from conflating these two notions. Explanation is the collection of pragmatically relevant, possibly counterfactual information about causation; and causation is to be identified in a necessary a posteriori way with whatever physical processes underwrite our explanatory claims.

Advocates of the conserved quantity (CQ) theory of causation have their own peculiar problem with conservation laws. Since they analyze causal process and interaction in terms of conserved quantities that are in turn defined as physical quantities governed by conservation laws, they must formulate conservation laws in a way that does not invoke causation, or else circularity threatens. In this paper I will propose an adequate formulation of a conservation law that serves CQ theorists' purpose.

In this paper I offer an 'integrating account' of singular causation, where the term 'integrating' refers to the following program for analysing causation. There are two intuitions about causation, both of which face serious counterexamples when used as the basis for an analysis of causation. The 'process' intuition, which says that causes and effects are linked by concrete processes, runs into trouble with cases of 'misconnections', where an event which serves to prevent another fails to do so on a particular (...) occasion and yet the two events are linked by causal processes. The chance raising intuition, according to which causes raise the chance of their effects, easily accounts for misconnections but faces the problem of chance lowering causes, a problem easily accounted for by the process approach. The integrating program attempts to provide an analysis of singular causation by synthesising the two insights, so as to solve both problems. In this paper I show that extant versions of the integrating program due to Eells, Lewis, and Menzies fail to account for the chance-lowering counterexample. I offer a new diagnosis of the chance lowering case, and use that as a basis for an integrating account of causation which does solve both cases. In doing so, I accept various assumptions of the integrating program, in particular that there are no other problems with these two approaches. As an example of the process account, I focus on the recent CQ theory of Wesley Salmon (1997). (shrink)

This paper examines Wesley Salmon's "process" theory of causality, arguing in particular that there are four areas of inadequacy. These are that the theory is circular, that it is too vague at a crucial point, that statistical forks do not serve their intended purpose, and that Salmon has not adequately demonstrated that the theory avoids Hume's strictures about "hidden powers". A new theory is suggested, based on "conserved quantities", which fulfills Salmon's broad objectives, and which avoids the problems discussed.

Causation has traditionally been analyzed either as a relation of nomic dependence or as a relation of counterfactual dependence. I argue for a third program, a physicalistic reduction of the causal relation to one of energy-momentum transference in the technical sense of physics. This physicalistic analysis is argued to have the virtues of easily handling the standard counterexamples to the nomic and counterfactual analyses, offering a plausible epistemology for our knowledge of causes, and elucidating the nature of the relation between (...) causation and physical science. (shrink)

This paper presents an attempt to integrate theories of causal processes—of the kind developed by Wesley Salmon and Phil Dowe—into a theory of causal models using Bayesian networks. We suggest that arcs in causal models must correspond to possible causal processes. Moreover, we suggest that when processes are rendered physically impossible by what occurs on distinct paths, the original model must be restricted by removing the relevant arc. These two techniques suffice to explain cases of late preëmption and other cases (...) that have proved problematic for causal models. (shrink)

Concerning any object of philosophical analysis, we can ask several questions, including the two posed in the title of this paper. Despite difficulties in formulating a precise criterion to distinguish causal processes from pseudoprocesses, and causal interactions from mere spatiotemporal intersections, I argue that Salmon answered the first of these questions with extraordinary clarity. The second question, by contrast, has received very little attention. I will present two problems: in the first, it seems that Salmon has provided exactly the conceptual (...) resources needed to solve the problem; in the second, it is difficult to see how causal processes and interactions may be used to shed any light. In general, the way to carry Salmon's program forward will be to demonstrate that these resources can be made to do real philosophical work. (shrink)

Showcasing original arguments for well-defined positions, as well as clear and concise statements of sophisticated philosophical views, this volume is an ...

The idea that causation can be reduced to transmission of an amount of some conserved quantity between events is spelled out and defended against important objections. Transmission is understood as a symmetrical relation of copresence in two distinct events. The actual asymmetry of causality has its origin in the asymmetrical character of certain irreversible physical processes and then spreads through the causal net. This conception is compatible with the possibility of backwards causation and with a causal theory of time. Genidentity, (...) the persistence of concrete objects, can be given an explanation in causal terms. The transmission theory is shown to escape difficulties faced by two important alternative theories of causation: Salmon's (1984) Mark Transmission Theory and Dowe's (1992a) Conserved Quantities Theory. (shrink)

The volume offers an overview of current research in ontology, distinguishing basic conceptual issues, domain applications, general frameworks, and mathematical ...

argues that the success of the backward causation hypothesis in quantum mechanics would provide strong support for a version of Reichenbach's account of the direction of causal processes, which takes the direction of causation to rest on the fork asymmetry. He also criticises my perspectival account of the direction of causation, which takes causal asymmetry to be a projection of our own temporal asymmetry as agents. In this reply I take issue with Dowe's argument at three main points: his claim (...) that the backward causation hypothesis in QM is incompatible with my perspectival approach to the direction of causation; his defence of the fork asymmetry approach against a general criticism of mine based on the time-symmetry of microphysics; and his application of his preferred account of the direction of causal processes to the relevant cases in QM. (shrink)

This paper presents a drastically revised version of the theory of causality, based on analyses of causal processes and causal interactions, advocated in Salmon (1984). Relying heavily on modified versions of proposals by P. Dowe, this article answers penetrating objections by Dowe and P. Kitcher to the earlier theory. It shows how the new theory circumvents a host of difficulties that have been raised in the literature. The result is, I hope, a more satisfactory analysis of physical causality.

The propagation of causal influences through space-time seems to play a fundamental role in scientific explanation. Taking as a point of departure a basic distinction between causal interactions (which are localized in space-time) and causal processes (which may extend through vast regions of space-time), this paper attempts an analysis of the concept of causal propagation on the basis of the ability of causal processes to transmit "marks." The analysis rests upon the "at-at" theory of motion which has figured prominently in (...) the resolution of Zeno's arrow paradox. It is argued that this explication does justice to the concept of the ability of causal processes to transmit causal influence without invoking anti-Humean "powers" or "necessary connections.". (shrink)

Negative causation occurs when an absence serves as cause, effect, or causal intermediary. Negative causation is genuine causation, or so I shall argue. It involves no physical connection between cause and effect. Thus causes need not be physically connected to their effects.

Causation, according to David Hume, is one of the three fundamental conceptual relations (along with resemblance and contiguity), and is the foundation of all reasoning concerning matters of fact. Causation, according to various contemporary philosophers, is required for the analysis of metaphysical concepts such as persistence, scientific concepts such as explanation and disposition, epistemic concepts such as perception and warrant, ethical concepts such as action and responsibility, legal concepts such as homicide and negligence, mental concepts such as functional role and (...) conceptual content, and linguistic concepts such as reference, to name just a salient few. Yet the nature of the causal relation itself remains mysterious. (shrink)

Causation, says David Lewis now, is to be understood as the ancestral of counterfactual influence, where C influences E (roughly) iff little changes in C map onto big changes in E. I argue that the influence account provides neither necessary nor sufficient conditions for causation, and suggest that what is missing is the notion of effluence, or physical connection.

Phil Dowe, in Physical Causation, addresses such questions as 'What are causal processes and interactions?', 'What is the connection between causes and effects?', and 'What distinguishes a cause from its effect?' Dowe not only provides explicit and original answers to these questions, but, en route, provides important critiques of alternative answers as well as sophisticated discussions of negative causation, the fork asymmetry, and quantum mechanics.

The leading regularity, counterfactual, and agential accounts of causation converge on the idea that causation is probability-raising. While the necessity of probability-raising for causation remains in dispute, the sufficiency of probability-raising for causation is generally assumed, at least in the direct (no intermediaries involved) and precisely described case. I offer a class of counterexamples: overlappings.

Causation is of undeniable importance to our understanding of, and interaction with our surroundings. Despite this, the correct understanding of causation remains subject to considerable philosophical controversy. In this article, I introduce the most influential philosophical theories of causation, and provide an overview of the main difficulties that has led to the currently most popular versions of these theories.