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Why animals have tumours

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Abstract

From the viewpoint of an evolutionary biologist, carcinogenesis should be looked upon as a protective mechanism against destruction of DNA. Because genes expressed in embryonic cells are covered and protected by heterochromatinization, they are the most appropriate ‘alternate genes’ compared to genes that are expressed already in somatic cells. When DNA-damage occurs, the embryonic genes can be activated. Some somatic cells exhibit some features of embryonic cells.

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References

  • Blood, C.H. and B.R. Zetter (1990). Tumor interactions with the vasculature: angiogenesis and tumor metastasis. Biochimica et Biophysica Acta 1032: 89–118.

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  • Little, J.W. and D.W. Mount (1982). The SOS regulatory system of Escherichia coli. Cell 29: 11–22.

    Article  Google Scholar 

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Niu, D.K., Wang, Y.F. Why animals have tumours. Acta Biotheor 43, 279–280 (1995). https://doi.org/10.1007/BF00707275

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  • DOI: https://doi.org/10.1007/BF00707275

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