Online research in philosophy

Recently Stephen Barker has raised stimulating objections to the thesis that, roughly speaking, if two events stand in a relation of counterfactual dependence, they stand in a causal relation. As Ned Hall says, however, this thesis constitutes the strongest part of the counterfactual analysis of causation. Therefore, if successful, Barker’s objections will undermine the cornerstone of the counterfactual analysis of causation, and hence give us compelling reasons to reject the counterfactual analysis of causation. I will argue, however, that they do not withstand scrutiny.

David Lewis modified his original theory of causation in response to the problem of ‘late preemption’ (see 1973b; 1986b: 193-212). However, as we will see, there is a crucial difference between genuine and preempted causes that Lewis must appeal to if his solution is to work. We argue that once this difference is recognized, an altogether better solution to the preemption problem presents itself.

Most versions of the psychological-continuity approach to personal identity (PCA) contain a 'non-branching' requirement. Recently, Robert Francescotti has argued that while such versions of PCA handle Parfit's standard fission case well, they deliver the wrong result in the case of an intact human brain. To solve this problem, he says, PCA-adherents need to add a clause that runs contrary to the spirit of their theory. In this response, I argue that Francescotti's counterexample fails. As a result, the revision he suggests is not needed.

Eigenfeatures are created by the principal component approach (PCA) used on objects described by a low-level code (i.e., pixels, Gabor jets). We suggest that eigenfeatures act like the flexible features described by Schyns et al. They are particularly suited for face processing and give rise to class-specific effects such as the other-race effect. The PCA approach can be modified to accommodate top-down constraints.

On David Lewis's original analysis of causation, c causes e only if c is linked to e by a chain of distinct events such that each event in the chain (counter-factually) depends on the former one. But this requirement precludes the possibility of late pre-emptive causation, of causation by fragile events, and of indeterministic causation. Lewis proposes three different strategies for accommodating these three kinds of cases, but none of these turn out to be satisfactory. I offer a single analysis of causation that resolves these problems in one go but which respects Lewis's initial insights. One distinctive feature of my account is that it accommodates indeterministic causation without resorting to probabilities.

Several different approaches to the conceptual analysis of causation are guided by the idea that a cause is something that makes a difference to its effects. These approaches seek to elucidate the concept of causation by explicating the concept of a difference-maker in terms of better-understood concepts. There is no better example of such an approach than David Lewis’ analysis of causation, in which he seeks to explain the concept of a difference-maker in counterfactual terms. Lewis introduced his counterfactual theory of causation with these words: 'We think of a cause as something that makes a difference, and the difference it makes must be a difference from what would have happened without it. Had it been absent, its effects—some of them, at least, and usually all—would have been absent as well.' (Lewis 1973b: pp. 160-1) According to Lewis, a cause c makes a difference to an effect e in the sense that if the cause c had not occurred, the effect e would not have occurred either. All we shall see in section 2, Lewis’ theory says there is more to the concept of causation than this counterfactual condition. Lewis is on the right track, I think, in saying that we think of a cause as something that makes a difference and that this thought is best explicated in terms of counterfactual concepts. However, I shall argue that the particular way in which Lewis spells out the concept of a cause as difference-maker is unsatisfactory. For Lewis’ articulation of this concept is distorted by a specific metaphysical assumption: specifically, that causation is an absolute relation, specifiable independently of any contextual factors. The distortion induced by this assumption is reflected in the undiscriminating manner in which his theory generates countless causes for any given effect. However, commonsense judgement is much more discriminating about causes than Lewis’ theory. Accordingly, I claim that Lewis' analysis faces the problem of profligate causes and I outline some specific problem cases in section 3..

It is shown that for invariance under the action of special groups the statements "Every invariant PCA is decomposable into (1 invariant Borel sets" and "Every pair of invariant PCA is reducible by a pair of invariant PCA sets" are independent of the axioms of set theory.

In response to counterexamples involving late preemption, David Lewis (1986) revised his original (1973) counterfactual analysis of causation to include the notion of quasi-dependence. Jonardon Ganeri, Paul Noordhof and Murali Ramachandran (1998) argue that their ‘PCA*-analysis’ of causation solves the problem of late preemption and is superior to Lewis’s analysis. I show that neither quasi-dependence nor the PCA*-analysis solves the problem of late preemption.