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- David Chalmers (1992). The Evolution of Learning: An Experiment in Genetic Connectionism. In Connectionist Models: Proceedings of the 1990 Summer School Workshop. Morgan Kaufmann.This paper explores how an evolutionary process can produce systems that learn. A general framework for the evolution of learning is outlined, and is applied to the task of evolving mechanisms suitable for supervised learning in single-layer neural networks. Dynamic properties of a network’s information-processing capacity are encoded genetically, and these properties are subjected to selective pressure based on their success in producing adaptive behavior in diverse environments. As a result of selection and genetic recombination, various successful learning mechanisms evolve, including the well-known delta rule. The effect of environmental diversity on the evolution of learning is investigated, and the role of different kinds of emergent phenomena in genetic and connectionist systems is discussed.
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Why do societies collapse? We use an individual-based evolutionary model to show that, in environmental conditions dominated by low-frequency variation (“red noise”), extirpation may be an outcome of the evolution of cultural capacity. Previous analytical models predicted an equilibrium between individual learners and social learners, or a contingent strategy in which individuals learn socially or individually depending on the circumstances. However, in red noise environments, whose main signature is that variation is concentrated in relatively large, relatively rare excursions, individual learning may be selected from the population. If the social learning system comes to lack sufficient individual learning or cognitively costly adaptive biases, behavior ceases tracking environmental variation. Then, when the environment does change, fitness declines and the population may collapse or even be extirpated. The modeled scenario broadly fits some human population collapses and might also explain nonhuman extirpations. Varying model parameters showed that the fixation of social learning is less likely when individual learning is less costly, when the environment is less red or more variable, with larger population sizes, and when learning is not conformist or is from parents rather than from the general population. Once social learning is fixed, extirpation is likely except when social learning is biased towards successful models. Thus, the risk of population collapse may be reduced by promoting individual learning and innovation over cultural conformity, or by preferential selection of relatively fit individuals as models for social learning. © 2009 Elsevier Inc. All rights reserved.
The deep formal and conceptual link existing between artificial life and artificial intelligence can be highlighted using conceptual tools derived by Karl Popper's evolutionary epistemology.Starting from the observation that the structure itself of an organism embodies knowledge about the environment which it is adapted to, it is possible to regard evolution as a learning process. This process is subject to the same rules indicated by Popper for the growth of scientific knowledge: causal conjectures (mutations) and successive refutations (extinction). In the field of machine learning such a paradigm is represented by genetic algorithms that, simulating biological processes, emulate cognitive processes. From a practical viewpoint, that perspective allows to identify the two different kinds of learning considered by artificial intelligence, knowledge acquisition and skill improvement, and to get a different view of the problem of heuristic knowledge in learning systems. From a theoretical point of view, these considerations can shade a new light on an old epistemological problem: why do we live in a learnable world?
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Many analogies exist between the process of evolution by natural selection and of learning by reinforcement and punishment. A full extension of the evolutionary analogy to learning to include analogues of the fitness, genotype, development, environmental influences, and phenotype concepts makes possible a single theory of the learning process able to encompass all of the elementary procedures known to yield learning.
Gene targeting has generated a great deal of data on the molecular mechanisms of long-term potentiation and its potential role in learning and memory. However, the interpretation of some results has been questioned. Compensatory mechanisms and the contribution of genetic background may make it difficult to unequivocally prove the existence of a causal (genetic) link between LTP and learning.
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The paper considers the problems involved in getting neural networks to learn about highly structured task domains. A central problem concerns the tendency of networks to learn only a set of shallow (non-generalizable) representations for the task, i.e., to miss the deep organizing features of the domain. Various solutions are examined, including task specific network configuration and incremental learning. The latter strategy is the more attractive, since it holds out the promise of a task-independent solution to the problem. Once we see exactly how the solution works, however, it becomes clear that it is limited to a special class of cases in which (1) statistically driven undersampling is (luckily) equivalent to task decomposition, and (2) the dangers of unlearning are somehow being minimized. The technique is suggestive nonetheless, for a variety of developmental factors may yield the functional equivalent of both statistical AND informed undersampling in early learning.
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