Online research in philosophy

Blackwell, 2000 Review by George Graham, Ph.D on Oct 27th 2000 Volume: 4, Number: 43.

Mad belief (in analogy with Lewisian mad pain ) would be a belief state with none of the causal role characteristic of belief—a state not caused or apt to have been caused by any of the sorts of events that usually cause belief and involving no disposition toward the usual behavioral or other manifestations of belief. On token-functionalist views of belief, mad belief in this sense is conceptually impossible. Cases of delusion—or at least some cases of delusion—might be cases of belief gone half-mad, cases in which enough of the functional role characteristic of belief is absent that the subject is in an in-between state regarding the delusive content, such that it is neither quite right to say the subject determinately believes the delusive content nor quite right to say that she determinately fails to believe that content. Although Bortolotti (2010) briefly mentions such sliding scale approaches to the relationship of delusion and belief, she dismisses such approaches on rather thin grounds and then later makes some remarks that seem consonant with sliding scale approaches.

Cognitive neuropsychiatry (CN) is the explanation of psychiatric disorder by the methods of cognitive neuropsychology. Within CN there are, broadly speaking, two approaches to delusion. The first uses a one-stage model, in which delusions are explained as rationalizations of anomalous experiences via reasoning strategies that are not, in themselves, abnormal. Two-stage models invoke additional hypotheses about abnormalities of reasoning. In this paper, I examine what appears to be a very strong argument, developed within CN, in favor of a twostage explanation of the difference in content between the Capgras and Cotard delusions. That explanation treats them as alternative rationalizations of essentially the same phenomenology. I show, however, that once we distinguish the phenomenology (and the neuroetiology), a one-stage model is adequate. In the final section I make some more general remarks on the oneand two-stage models.

The papers in this volume are drawn from a workshop on delusion and self-deception, held at Macquarie University in November of 2004. Our aim was to bring together theorists working on delusions and self-deception with an eye towards identifying and fostering connections—at both empirical and conceptual levels—between these domains. As the contributions to this volume testify, there are multiple points of contact between delusion and self-deception. This introduction charts the conceptual space in which these points of contact can be located and introduces the reader to some of the general issues that frame the discussion of subsequent chapters.

We provide a battery of examples of delusions against which theoretical accounts can be tested. Then, we identify neuropsychological anomalies that could produce the unusual experiences that may lead, in turn, to the delusions in our battery. However, we argue against Maher’s view that delusions are false beliefs that arise as normal responses to anomalous experiences. We propose, instead, that a second factor is required to account for the transition from unusual experience to delusional belief. The second factor in the aetiology of delusions can be described superficially as a loss of the ability to reject a candidate for belief on the grounds of its implausibility and its inconsistency with everything else that the patient knows. But we point out some problems that confront any attempt to say more about the nature of this second factor.

The present article proposes a logical account of delusions, which are regarded as conclusions resulting from fallacious arguments. This leads to distinguish between primary, secondary, ..., n-ary types of delusional arguments. Examples of delusional arguments leading to delusion of reference, delusion of influence, thought-broadcasting delusion and delusion of grandeur are described and then analyzed. This suggests finally a way susceptible of improving the efficiency of cognitive therapy for delusions.

Subjects with delusions profess to believe some extremely peculiar things. Patients with Capgras delusion sincerely assert that, for example, their spouses have been replaced by impostors. Patients with Cotard’s delusion sincerely assert that they are dead. Many philosophers and psychologists are hesitant to say that delusional subjects genuinely believe the contents of their delusions.2 One way to reinterpret delusional subjects is to say that we’ve misidentified the content of the problematic belief. So for example, rather than believing that his wife is has been replaced by an impostor, we might say that the victim of Capgras delusion believes that it is, in some respects, as if his wife has been replaced by an impostor. Another is to say that we’ve misidentified the attitude that the delusional subject bears to the content of their delusion. So for example, Gregory Currie and co-authors have suggested that rather than believing that his wife has been replaced by an impostor, we should say that the victim of Capgras delusion merely imagines that his wife has been replaced by an impostor.3.

Some otherwise rational people appear to believe strange things. Sometimes people believe that someone, usually a near relative or member of their family - often their spouse - has been replaced by an impostor. Sometimes people believe that they are dead. These two delusions – known as the Capgras and Cotard delusion respectively – are instances of monothematic delusions, for they are limited to very specific topics. Other monothematic delusions involve the delusion that one is being followed by known people in disguise (the Frégoli delusion), or that the person one sees in the mirror is someone else (mirrored-self misidentification). We will focus on the Capgras delusion.

Current models of delusion converge in proposing that delusional beliefs are based on unusual experiences of various kinds. For example, it is argued that the Capgras delusion (the belief that a known person has been replaced by an impostor) is triggered by an abnormal affective experience in response to seeing a known person; loss of the affective response to a familiar person’s face may lead to the belief that the person has been replaced by an impostor (Ellis & Young, 1990). Similarly, the Cotard delusion (which involves the belief that one is dead or unreal in some way) may stem from a general..