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- L. A. Paul (1998). Problems with Late Preemption. Analysis 58 (1):48–53.In response to counterexamples involving late preemption, David Lewis (1986) revised his original (1973) counterfactual analysis of causation to include the notion of quasi-dependence. Jonardon Ganeri, Paul Noordhof and Murali Ramachandran (1998) argue that their ‘PCA*-analysis’ of causation solves the problem of late preemption and is superior to Lewis’s analysis. I show that neither quasi-dependence nor the PCA*-analysis solves the problem of late preemption.
Similar books and articles
Extant counterfactual accounts of causation (CACs) still cannot handle preemptive causation. I describe a new variety of preemption, defend its possibility, and use it to show the inadequacy of extant CACs. Imagine that it is a law of nature that the first spell cast on a given day match the enchantment that midnight.
Preemption games are widely used to model patent races, innovation adoption and market entry problems. A previously neglected feature of these problems is that the agents’ states (e.g. R&D …rms’ technological improvements) are kept secret and stochastically change over time. We fully characterize equilibrium in preemption games where private information evolves according to Poisson processes, and provide a strategic rationale for the common wisdom that ‘big things happen fast.’ In the context of patent races we surprisingly …nd that strengthening patent rights need not increase innovation disclosure. Furthermore, we clarify a basic welfare tradeo¤ between duplication costs and preemption: the former likely take place in early stages of the race, and preemption in later stages.
No categories
No categories
Causation is a deeply intuitive and familiar relation, gripped powerfully by common sense. Or so it seems. But as is typical in philosophy, deep intuitive familiarity has not led to any philosophical account of causation that is at once clean, precise, and widely agreed upon. Not for lack of trying: the last 30 years or so have seen dozens of attempts to provide such an account, and the pace of development is, if anything, accelerating. (See Collins, Hall and Paul 2003a for a comprehensive sampling of the latest work.) It is safe to say that none has yet succeeded. It is also safe to say that the effort put into their development has yielded a wealth of insights into causation. And it is, arguably, from the study of causal preemption—cases that feature multiple competing candidates for the title of “cause” of some given effect—that the greatest such wealth has flowed. These cases come in a number of varieties: so-called early and late preemption, symmetric overdetermination, and trumping preemption. Collectively, they place extremely severe constraints on any philosophical account of causation that can successfully handle them. One of the lessons they have to teach, then, is a lesson about the form that a successful analysis of causation must have. There is a deeper lesson, a lesson about the nature of causation itself, or if you like, about the workings of our causal concept. It emerges from close study of the struggles that extant accounts face in trying to provide even remotely attractive treatments of causal preemption. It is this: There appears to be a significant and perhaps intractable tension between one strand in our thinking about causation—a strand that emphasizes the need for causes to be connected to their effects via intervening processes with the right intrinsic character—and another—one that emphasizes the claim that effects in some sense depend on their causes. By the end of this essay, this tension will be vividly apparent..
On David Lewis's original analysis of causation, c causes e only if c is linked to e by a chain of distinct events such that each event in the chain (counter-factually) depends on the former one. But this requirement precludes the possibility of late pre-emptive causation, of causation by fragile events, and of indeterministic causation. Lewis proposes three different strategies for accommodating these three kinds of cases, but none of these turn out to be satisfactory. I offer a single analysis of causation that resolves these problems in one go but which respects Lewis's initial insights. One distinctive feature of my account is that it accommodates indeterministic causation without resorting to probabilities.
Counter factual theories of Causation have had problems with cases of probabilistic causation and preemption. I put forward a counterfactual theory that seems to deal with these problematic cases and also has the virtue of providing an account of the alleged asymmetry between hasteners and delayers: the former usually being counted as causes, the latter not. I go on to consider a new type of problem case that has not received so much attention in the literature, those I dub catalysts and anti-catalysts, and show how my account needs to be adjusted to deliver the right verdicts in these cases. The net result is a particular conception of a cause that I try to spell out in the closing section of the paper. In that section, I also briefly discuss causal asymmetry and the purpose behind providing a counterfactual theory of causation.
David Lewis’s latest theory of causation defines the causal link in terms of the relation of influence between events. It turns out, however, that one event’s influencing another is neither a necessary nor sufficient condition for its being a cause of that event. In the article one particular case of causality without influence is presented and developed. This case not only serves as a counterexample to Lewis’s influence theory, but also threatens earlier counterfactual analyses of causation by admitting a particularly troublesome type of preemption. The conclusion of the article is that Lewis’s influence method of solving the preemption problem fails, and that we need a new and fresh approach to the cases of redundant causation if we want to hold on to the counterfactual analysis of causation.
In his ‘Causation as Influence’,1 David Lewis proposed a counterfactual theory of cause which was designed to improve on his previous account.2 Here I offer counter-examples to this new account, involving early preemption and late preemption, and a revised account, which is no longer an influence theory, that handles those counter-examples.
Jonardon Ganeri, Paul Noordhof, and Murali Ramachandran (1996) have proposed a new counterfactual analysis of causation. We argue that this – the PCA-analysis – is incorrect. In section 1, we explain David Lewis’s first counterfactual analysis of causation, and a problem that led him to propose a second. In section 2 we explain the PCA-analysis, advertised as an improvement on Lewis’s later account. We then give counterexamples to the necessity (section 3) and sufficiency (section 4) of the PCA-analysis.
David Lewis modified his original theory of causation in response to the problem of ‘late preemption’ (see 1973b; 1986b: 193-212). However, as we will see, there is a crucial difference between genuine and preempted causes that Lewis must appeal to if his solution is to work. We argue that once this difference is recognized, an altogether better solution to the preemption problem presents itself.
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