Online research in philosophy

The idea that causation can be reduced to transmission of an amount of some conserved quantity between events is spelled out and defended against important objections. Transmission is understood as a symmetrical relation of copresence in two distinct events. The actual asymmetry of causality has its origin in the asymmetrical character of certain irreversible physical processes and then spreads through the causal net. This conception is compatible with the possibility of backwards causation and with a causal theory of time. Genidentity, the persistence of concrete objects, can be given an explanation in causal terms. The transmission theory is shown to escape difficulties faced by two important alternative theories of causation: Salmon's (1984) Mark Transmission Theory and Dowe's (1992a) Conserved Quantities Theory.

This paper examines the Transference Theory of causation, developed originally by Aronson (1971) and Fair (1979). Three difficulties for that theory are presented: firstly, problems associated with the direction of transference and causal asymmetry; secondly, the case of persistence as causation, for example where a body's own inertia is the cause of its motion; and thirdly the problematic notion of identity through time of physical quantities such as energy or momentum. Finally, the theory is compared with the Conserved Quantity Theory (Dowe 1992c), and it is shown that that account embodies the modifications that the transference theory needs to adopt.

Much of the philosophical literature on causation has focused on the concept of actual causation, sometimes called token causation. In particular, it is this notion of actual causation that many philosophical theories of causation have attempted to capture.2 In this paper, we address the question: what purpose does this concept serve? As we shall see in the next section, one does not need this concept for purposes of prediction or rational deliberation. What then could the purpose be? We will argue that one can gain an important clue here by looking at the ways in which causal judgments are shaped by people‘s understanding of norms.

In this paper I wish to argue that counterfactual analyses of causation are inadequate. I believe the counterfactuals that are involved in counterfactual analyses of causation are often false, and thus the theories do not provide an adequate account of causation. This is demonstrated by the presentation of a counterexample to the counterfactual analyses of causation. I then present a unified theory of causation that is based upon probability and counterfactuals. This theory accounts for both deterministic and indeterministic causation, and is not subject to many of the traditional problems facing theories of causation.

Transference theorists propose to explain causation in terms of the transference of a physical element. I argue, in two steps, that this is not possible. First, I show that available accounts of ‘transference’ ultimately convey that transference -and, consequently, causation- is the (non-relational) identity over time of the transferred element (a universal, a trope, or even an absolute substance). But, second, I try to defend, it is conceptually impossible that causation is (non-relational) identity.

I propose a non-Humean theory of causation with “tendencies” as causal connections. Not, however, as “necessary connexions”: causes are not sufficient, they do not necessitate their effects. The theory is designed to be, not an analysis of the concept of causation, but a description of what is the case in typical cases of causa-tion. I therefore call it a metaphysical theory of causation, as opposed to a semantic one.

The purpose of this paper is to lay bare the major problems underlying the concept of downward causation as discussed within the perspective of the present interest for phenomena that are characterized by self-organization. In our Discussion of the literature, we have focussed on two questions: (1) What sorts of things are said to be, respectively, causing and caused within the context of downward causation? And (2) What is the meaning of 'causing' in downward causation? We have concluded that the concept of 'downward causation' is muddled with regard to the meaning of causation and fuzzy with regard to the nature of the causes and the effects. Moreover, we have concluded that 'causation' in respect of 'downward causation' is usually understood in terms of explanation and determination rather than in terms of causation in the sense of 'bringing about'. Thus, the term 'downward causation' is badly chosen.

. An attempt is made to identify a concept of ‘downward causation’ that will fit the claims of some recent writers and apply to interesting cases in biology and cognitive theory, but not to trivial cases. After noting some difficulties in achieving this task, it is proposed that in interesting cases commonly used to illustrate ‘downward causation’, (a) regularities hold between multiply realizable properties and (b) the explanation of the parallel regularity at the level of the realizing properties is non-trivial. It is argued that the relation between a realizable property and the property that realizes its effect in a particular case is not usefully regarded as a species of causation and that use of the concept of downward causation deflects our attention from our central explanatory tasks.

One part of the true theory of actual causation is a set of conditions responsible for eliminating all of the non-causes of an effect that can be discerned at the level of counterfactual structure. I defend a proposal for this part of the theory.

Ehring shows the inadequacy of received theories of causation, and, introducing conceptual devices of his own, provides a wholly new account of causation as the persistence over time of individual properties, or "tropes.".