Online research in philosophy

Empiricists have sought to follow Hume in claiming that causality is a relation between events reducible to something more basic, e.g., regularities or counterfactuals. But all such attempts fail through their inability to distinguish cause from effect. The alternative is that causation is irreducible. Regularities are evidence of causation but do not constitute it. We understand what causation is through performing intentional actions which necessarily involve trying, which in turn just is exercising causal power.

I IT is often declared or evidently assumed that causality is some kind of necessary connexion, or alternatively, that being caused is — non-trivially ...

One part of the true theory of actual causation is a set of conditions responsible for eliminating all of the non-causes of an effect that can be discerned at the level of counterfactual structure. I defend a proposal for this part of the theory.

This volume presents a selection of the most influential recent discussions of the crucial metaphysical question: What is it for one event to cause another? The subject of causation bears on many topics, such as time, explanation, mental states, the laws of nature, and the philosophy of science. Contributors include J.L Mackie, Michael Scriven, Jaegwon Kim, G.E.M. Anscombe, G.H. von Wright, C.J. Ducasse, Wesley C. Salmon, David Lewis, Paul Horwich, Jonathan Bennett, Ernest Sosa, and Michael Tooley.

The aim of this paper is to develop an adequate version of the D-N theory of explanation for particular events and to show how the resulting D-N model can be used as a tool in articulating a regularity theory of causation and an analysis of the truth conditions for counterfactual conditionals. Starting with a basic model that is largely the product of other workers in this field, two new restrictions are formulated in order to construct a version of D-N explanation that does not yield the counterintuitive results that have plagued all earlier versions. An additional condition is then developed that is indispensable for utilizing the D-N model as a tool in formulating a regularity theory of causation. Finally, it is shown how a suitable model of potential D-N explanation facilitates the formulation of a theory of counterfactual conditionals.

In this paper I wish to argue that counterfactual analyses of causation are inadequate. I believe the counterfactuals that are involved in counterfactual analyses of causation are often false, and thus the theories do not provide an adequate account of causation. This is demonstrated by the presentation of a counterexample to the counterfactual analyses of causation. I then present a unified theory of causation that is based upon probability and counterfactuals. This theory accounts for both deterministic and indeterministic causation, and is not subject to many of the traditional problems facing theories of causation.

This paper presents ConR (Conditional R), a logic of conditionals based on Anderson and Belnap''s system R. A Routley-Meyer-style semantics for ConR is given for the system (the completeness of ConR over this semantics is proved in E. Mares and A. Fuhrmann, A Relevant Theory of Conditionals (unpublished MS)). Moreover, it is argued that adopting a relevant theory of conditionals will improve certain theories that utilize conditionals, i.e. Lewis'' theory of causation, Lewis'' dyadic deontic logic, and Chellas'' dyadic deontic logic.

The basic idea of counterfactual theories of causation is that the meaning of causal claims can be explained in terms of counterfactual conditionals of the form “If A had not occurred, C would not have occurred”. While counterfactual analyses have been given of type-causal concepts, most counterfactual analyses have focused on singular causal or token-causal claims of the form “event c caused event e”. Analyses of token-causation have become popular in the last thirty years, especially since the development in the 1970's of possible world semantics for counterfactuals. The best known counterfactual analysis of causation is David Lewis's (1973b) theory. However, intense discussion over thirty years has cast doubt on the adequacy of any simple analysis of singular causation in terms of counterfactuals. Recent years have seen a proliferation of different refinements of the basic idea to achieve a closer match with commonsense judgements about causation.

Counterfactual analysis of causation between particular events, combined with standard semantics for counterfactual conditionals, cannot express the idea that the cause is sufficient for the effect. Several authors have suggested that a more complex pattern of nested counterfactual conditionals is a better candidate for expressing the idea of causal connection. The most systematic account is developed by Kadri Vihvelin. She argues that a complex pattern of causal dependence, expressed by embedded conditionals, covers all the cases of causation and still yields an account of causal asymmetry. But the new account relies heavily on the use of backtracking conditionals, and no criterion is given for their evaluation. I will try to show that Vihvelin’s proposal is not superior to the standard account because it overlooks the disadvantages of a liberal theory of causal relata.

If we seek to analyse causation in terms of counterfactual conditionals then we must assume that there is a class of counterfactuals whose members (i) are all and only those we need to support our judgements of causation, (ii) have truth-conditions specifiable without any irreducible appeal to causation. I argue that (i) and (ii) are unlikely to be met by any counterfactual analysis of causation. I demonstrate this by isolating a class of counterfactuals called non-projective counterfactuals, or NP-counterfactuals, and indicate how counterfactual analyses of causation must appeal to them to account for the correct causal judgements we make. I show that the truth-conditions of NP-counterfactuals are specifiable only by irreducible appeal to causation. A dilemma then holds: if counterfactual analyses of causation eschew appeal to NP-counterfactuals they are empirically inadequate, but if they appeal to NP-counterfactuals they are circular and thus conceptually inadequate.