Online research in philosophy

The idea that causation can be reduced to transmission of an amount of some conserved quantity between events is spelled out and defended against important objections. Transmission is understood as a symmetrical relation of copresence in two distinct events. The actual asymmetry of causality has its origin in the asymmetrical character of certain irreversible physical processes and then spreads through the causal net. This conception is compatible with the possibility of backwards causation and with a causal theory of time. Genidentity, the persistence of concrete objects, can be given an explanation in causal terms. The transmission theory is shown to escape difficulties faced by two important alternative theories of causation: Salmon's (1984) Mark Transmission Theory and Dowe's (1992a) Conserved Quantities Theory.

In this paper, I explore the notion of a “causal power”, particularly as it is relevant to a theory of properties whereby properties are individuated by the causal powers they bestow on the objects that instantiate them. I take as my target certain eliminativist positions that argue that certain kinds of properties (or relations) do not exist because they fail to bestow unique causal powers on objects. But the notion of a causal powers is inextricably bound up with our notion of what an event is, and not only is there disagreement as to which theory of events is appropriate, but on the three prevailing theories, it can be shown that the eliminativists arguments do not follow.

Recent research on “causal cognition” in adults and infants shows that we can perceive singular causal relations not previously experienced. In particular, infants that are able to perceive causality seem to rely on innate beliefs and principles that allow a priori inference of a connection between cause and effect. Can causal cognition in infants justify the thesis of causal realism? On the one hand, it weakens the central pillar of the Humean arguments: the impossibility of a synthetic a priori causal inference. On the other hand, if perception is the privileged way of justifying the reality of objects of the external world, that is valid in the case of causal relations as well. Moreover, the perception of causal relations, based on innate principles and beliefs, reflects the selective results of the interaction between the real constraints of the physical structure of the world and the evolution of the human mind.

Minimally, causal realism (as understood here) is the view that accounts of causation in terms of mere, regular or probabilistic conjunction are unsatisfactory, and that causal phenomena are correctly associated with some form of de re necessity. Classic arguments, however, some of which date back to Sextus Empiricus and have appeared many times since, including famously in Russell, suggest that the very notion of causal realism is incoherent. In this paper I argue that if such objections seem compelling, it is only because everyday expressions concerning causal phenomena are misleading with respect to certain metaphysical details. These expressions generally make reference to the relations of events or states of affairs, but ignore or obscure the role played by causal properties. I argue that on a proposed alternative, an analysis in terms of causal processes, more refined descriptions of causal phenomena escape the charge of incoherence. Causal necessity is here located in the relations of causal properties. I distinguish this view from the recent process theories of Salmon and Dowe, which are disinterested in causal realism.

Explanation is usually taken to be a relation between certain entities. The aim of this paper is to discuss what entities are suitable as explanatory relata of singular causal explanations, i.e., explanations concerning singular causality relating particular events or other appropriate entities. I outline three different positions. The purely causal approach stipulates that the same entities that are related in the singular causal relation are also linked by the explanatory relation. This position, however, has a problem to distinguish between causation and explanation, two distinct relations allegedly obtaining between the same entities. The linguistic approach states that explanatory relata are linguistic entities of some sort, e.g., statements, propositions, etc. There are various versions of this position. I deal with two of them and try to show that they are unsatisfactory because they transform explanation into some other type of relation. On the first version, explanation is very close to interpretation or clarification of intension and on the second version it seems to be indistinguishable from an evidential relation or justification. I consider these transformations in understanding explanation unnecessary, and consequently reject linguistic views of explanatory relata. The most promising proposal concerning explanatory relata seems to be the mixed view, according to which propositions explain events or other fitting extra-linguistic entities.

Hume argued that experience could not justify commonly held beliefs in singular causal effcacy, according to which individual or singular causes produce their effects or make their effects happen. Hume's discussion has been influential, as motivating the view that Causal reductionism (denying that causal efficacy is an irreducible feature of natural reality) requires Causal generalism (according to which causal relations are metaphysically constituted by patterns of events). Here I argue that causal reductionists---indeed, Hume himself---have previously unappreciated resources for making sense of Causal singularism, associated with a relation that has been curiously underexploited in the causation debates: resemblance. The core idea I explore here is that causation may be metaphysically and epistemologically indicated by the coming-to-be of a resemblance. Comings-to-be of resemblances are epistemically available in the singular instance, even by Hume's strict lights, and, I argue, can justify (albeit fallibly) belief in the holding of singular causal relations; hence Hume's general argument for generalism fails. More to the contemporary metaphysical point, comings-to-be of resemblances provide valuable resources for existing singularist accounts: while neither changes (Ducasse) nor transfers of physical quantities (Fair, Dowe, Salmon) provide a suffciently fine-grained basis for the individuation of causes, either changes or transfers, in combination with comings-to-be of resemblances, can do so.

Causality is a concept which is sometimes claimed to be easy to illustrate, but hard to explain. It is not quite clear whether the former part of this claim is as obvious as the latter one. I will not present any specific theory of causation. Our aim is much less ambitious; to investigate the formal counterparts of causal relations between events, i.e. to propose a formal framework which enables us to construct metamathematical counterparts of causal relations between singular events. This should be a good starting point to define formal counterparts for concepts like causal law, causal explanation and so on.

One of the cornerstones of modern medicine is the search for what causes diseases to develop. A conception of multifactorial disease causes has emerged over the years. Theories of disease causation, however, have not quite been developed in accordance with this view. It is the purpose of this paper to provide a fundamental explication of aspects of causation relevant for discussing causes of disease.The first part of the analysis will discuss discrimination between singular and general causality. Singular causality, as in the specific patient, is a relation between a concrete sequence of causally linked events. General causation, e.g. as in disease etiology, means various categories of causal relations between event types. The paper introduces the concept of a reference case serving as a source for causal inference, reaching beyond the concept of general causality.

The object of this paper is to offer a conception of singular causality that lies between two main views in the literature, which I take to be paradigmatically represented by David Armstrong (1997) and by Michael Tooley (1987, 1990) respectively. Armstrong maintains that there is singular causation wherever there are singular facts that instantiate causal laws; these facts are otherwise independent regularities. Tooley maintains that singular causation is independent of causal laws together with any other non-causal fact. My own view is that Armstrong’s account is too weak to conform a singularist position, as in his view singular causal relations are finally dependent on universal causation. On the other hand, Tooley’s account is too strong to be causal, as causation dissolves into a purely external and mysterious connection that is not even Humean -not even regularities play a role in its establishment. I want to maintain that there exists a middle way that correctly characterizes the spirit of singular causation, and that can be stated by (CS): (CS) Singular causality is metaphysically independent of universal causality. Neither Tooley nor Armstrong would admit (CS) as an appropriate defining thesis for causal singularism. In what follows, I consider different causal situations existing in the literature, with the purpose of analysing singular causation, arguing against Tooley and Armstrong's views, and defending (CS) instead. If my reasons are accepted, Armstrong’s position, rather than being singular in spirit, results into a form of causal universalism. I will also reject Tooley’s singularism as an extreme counterintuitive form of hyperrealism. Some consequences for causation are immediate. In particular, that singular causal relations are tropes, i.e., individual relations.

The literature on causation distinguishes between causal claims relating properties or types and causal claims relating individuals or tokens. Many authors maintain that corresponding to these two kinds of causal claims are two different kinds of causal relations. Whether to regard causal relations among variables as yet another variety of causation is also controversial. This essay maintains that causal relations obtain among tokens and that type causal claims are generalizations concerning causal relations among these tokens.