The etiology of a perceptual belief can seemingly affect its epistemic status. There are cases in which perceptual beliefs seem to be unjustified because the perceptual experiences on which they are based are caused, in part, by wishful thinking, or irrational prior beliefs. It has been argued that this is problematic for many internalist views in the epistemology of perception, especially those which postulate immediate perceptual justification. Such views are unable to account for the impact of an experience’s etiology on (...) its justificational status, McGrath, Siegel, and Vahid ). Our understanding of what we have been told can also be affected by, for example, wishful thinking or irrational background beliefs. I argue that testimonial beliefs based on such states of understanding can thus be rendered unjustified. This is problematic not only for internalist immediate justification views of testimony, but also for some externalist views, such as the form of proper functionalism endorsed by Burge, and Graham. The testimonial version of the argument from etiology, unlike the perceptual variant, does not rest on the controversial hypothesis that perception is cognitively penetrable. Furthermore, there is a stronger case for the claim that testimonial justification can be undermined by etiological effects since, I argue, testimonial beliefs can be based on the background mental states which affect our understanding of what is said, and our states of understanding are rationally assessable. (shrink)

In this article, I will reconstruct the monocausal model and argue that modern 'multifactorial diseases' are not monocausal by definition. 'Multifactorial diseases' are instead defined according to a constitutive disease model. On closer analysis, infectious diseases are also defined using the constitutive model rather than the monocausal model. As a result, our classification models alone cannot explain why infectious diseases have a universal etiology while chronic and noncommunicable diseases lack one. The explanation is instead provided by the nineteenth-century germ theorists.

Aristotle’s biological teleology is rooted in an epigenetic account of reproduction. As such, it is best interpreted by consequence etiology. I support this claim by citing the capacity of consequence etiology’s key distinctions to explain Aristotle’s opposition to Empedocles. There are implications for the relation between ancient and modern biology. The analysis reveals that in an important respect Darwin’s account of adaptation is closer to Aristotle’s than to Empedocles’s. They both rely on consequence etiological considerations to evade attributing the purposiveness (...) of organisms to chance. Two implications follow: Darwinian explanations of adaptation are as teleological as Aristotle’s, albeit differently; and these differences show how deeply resistant Aristotle’s version of biological teleology is to descent from a common ancestor. (shrink)

People sometimes try to call others’ beliefs into question by pointing out the contingent causal origins of those beliefs. The significance of such ‘Etiological Challenges’ is a topic that has started attracting attention in epistemology. Current work on this topic aims to show that Etiological Challenges are, at most, only indirectly epistemically significant, insofar as they bring other generic epistemic considerations to the agent’s attention. Against this approach, we argue that Etiological Challenges are epistemically significant in a more direct and (...) more distinctive way. An Etiological Challenge prompts the agent to assess whether her beliefs result from practices of indoctrination, and whether she should reduce confidence in those beliefs, given the anti-reliability of indoctrination as a method of belief-acquisition. Our analysis also draws attention to some of the ways in which epistemic concerns interact with political issues—e.g. relating to epistemic injustice, identity-based discrimination, and segregation—when we’re thinking about the contingent causal origins of our beliefs. (shrink)

It can be shown that considerable common ground exists between indigenous or traditional theories of contagious disease in Africa, and modern medicine, whether human or veterinary. Yet this is not recognized because of the generally low regard in which the medically trained – whether African or expatriate – hold African traditional medicine. This attitude seems to result from the assumption that African health beliefs are based on witchcraft and related “supernatural” thinking. I argue that this may not be so in (...) the important domain of diseases biomedically classified as contagious; such diseases tend to be understood naturalistically. An accurate understanding of how Africans traditionally interpret contagious diseases of humans and livestock is the foundation for the design and implementation of more effective health programs. (shrink)

A fundamental aspect of human beings is that they learn. The process of learning and what is learned are impacted by a number of factors, both cognitive and social; that is, humans are boundedly rational. Cognitive and social limitations interact, making it difficult to reason about how to provide information to impact what humans know, believe, and do. Herein, we use a multi‐agent dynamic‐network simulation system, Construct, to conduct such reasoning. In particular, we ask, What media should be used to (...) provide information to most impact what people know, believe, and do, given diverse social structures? All simulated agents are boundedly rational both at the cognitive and social level, and so are subject to factors such as literacy, education, and the breadth of their social network. We find that there is no one most effective intervention; rather, to be effective, messages and the media used to spread the message need to be selected for the population being addressed. Typically, a multimedia campaign is critical. (shrink)

We reply to Artiga and Martinez’s claim according to which the organizational account of cross-generation functions implies a backward looking interpretation of etiology, just as standard etiological theories of function do. We argue that Artiga and Martinez’s claim stems from a fundamental misunderstanding about the notion of “closure”, on which the organizational account relies. In particular, they incorrectly assume that the system, which is relevant for ascribing cross-generation organizational function, is the lineage. In contrast, we recall that organizational closure refers (...) to a relational description of a network of mutual dependencies, abstracted from time, in which production relations are irrelevant. From an organizational perspective, ascribing a function to an entity means locating it in the abstract system that realizes closure. In particular, the position of each entity within the relational system conveys an etiological explanation of its existence, because of its dependence on the effects exerted by other entities subject to closure. Because of the abstract relational nature of closure, we maintain that the organizational account of functions does not endorse a backward looking interpretation of etiology. As a consequence, it does not fall prey of epiphenomenalism. (shrink)

It is argued there are features which distinguish causal conditions from necessary or sufficient conditions in general; that causal conditions are those which serve our instrumental interests, i.e., interests in producing or preventing states of affairs. It is further argued that the context of our instrumental interests determines what causal condition is designated as "the cause" in scientific contexts. It is argued that this is true of the health sciences in particular and the particular instrumental interests characteristic of the health (...) sciences are further shown to govern the choice of what causal condition of a disease is accorded the status of etiologic agent. (shrink)

There has been a general failure among mental health theorists and social psychologists to understand the etiology of work-engendered depression. Yet the condition is increasingly prevalent in highly industrialized societies, where an exclusionary focus upon work, money, and the things that money can buy has displaced values that traditionally exerted a liberating and humanizing influence. Social critics have called the result an impoverishment of the spirit, a state of cultural bankruptcy, and an incapacity for genuine leisure. From a clinical perspective, (...) the condition has been diagnosed as widespread narcissism and obsessive work. -/- Acedia, a concept developed by the Scholastics, throws clarifying light on the origin of this form of depression. (shrink)

Modern medicine is often said to have originated with nineteenth century germ theory, which attributed diseases to bacterial contagions. The success of this theory is often associated with an underlying principle referred to as the “doctrine of specific etiology”. This doctrine refers to specificity at the level of disease causation or etiology. While the importance of this doctrine is frequently emphasized in the philosophical, historical, and medical literature, these sources lack a clear account of the types of specificity that it (...) involves and why exactly they matter. This paper argues that nineteenth century germ theory involves two types of specificity at the level of etiology. One type receives significant attention in the literature, but its influence on modern medicine has been misunderstood. A second type is present in this model, but it has been completely overlooked in the extant literature. My analysis clarifies how these types of specificity led to a novel conception of etiology that continues to figure in medicine today. (shrink)

Institutions generate cooperative benefits that explain why they exist and persist. Therefore, their etiological function is to promote cooperation. The function of a particular institution, such as money or traffic regulations, is to solve one or more cooperation problems. We go on to argue that the teleological function of institutions is to secure values by means of norms. Values can also be used to redesign an institution and to promote social change. We argue, however, that an adequate theory of institutions (...) should not be ‘moralized’ in that they should not be defined in terms of the values they are supposed to promote. (shrink)

The aim of teleosemantics is to give a scientifically respectable, or ‘naturalistic’ theory of mental content. In the debates surrounding the scope and merits of teleosemantics a lot has been said about the concept of indication (or carrying information). The aim of this paper is to focus on the other key concept of teleosemantics: biological function. It has been universally accepted in the teleosemantics literature that the account of biological function one should use to flesh out teleosemantics is that of (...) etiological function. My claim is that if we replace this concept of function with an alternative one (that we have independent reasons to accept) and if we also restrict the scope of teleosemantics, we can arrive at an account of biologizing mental content that is much less problematic than the previous attempts. (shrink)

Conscious existence is the product of a neural brain mechanism, which is largely identical with Immanuel Kant's Oneness Function, a service performed by 200 million neurons in the prefrontal lobe, & makes possible our interior cosmos, the record of our interconnected, or general, experience. Essential for us humans is the well-being of our interior cosmos, or Saint Teresa of Avila's interior castle, in all interactions with each other \& the greater environment. Any disorders of our cosmos are liable to make (...) us fall ill, setting our existence on a path of psychosomatic etiology of a large class of mysterious undiagnosed diseases. (shrink)

Cummins (1982) argues that etiological considerations are not onlyinsufficient butirrelevant for the determination offunction. I argue that his claim of irrelevance rests on a misrepresentation of the use of functions in evolutionary explanations. I go on to suggest how accepting anetiological constraint on functional analysis might help resolve some problems involving the use of functional explanations.

Cognitive science of religion is sometimes portrayed as having no bearing on the theological doctrines of particular religious traditions, such as Christianity. In this paper, I argue that the naturalistic account of the etiology of religious beliefs offered by the cognitive science of religion undermines the important Christian doctrine of the grace of faith, which teaches that the special gift of divine grace is a necessary precondition for coming to faith. This has some far-reaching ramifications for Christian theology. -/- .

In this paper I clarify a major affinity between Nietzsche and Spinoza that has been neglected in the literature—but that Nietzsche was aware of—namely a tendency to what I call etiology. Etiologies provide second- order explanations of some opponents’ first-order views, but not in order to decide first-order matters. The example I take up here is Nietzsche’s and Spinoza’s rejections of free will—and especially their etiologies concerning how we wrongly come to think that we may boast of such a capacity. (...) In working through the former (i.e., their rejections of free will) in order to make my central metaphilosophical point regarding the latter (i.e., their accounts of why we generally affirm that we have free will), I shed important new light on Nietzsche’s relation to Spinoza. I also further our understanding of what role such second-order accounts play within each of their larger projects on their own terms. (shrink)

In this paper I offer a theory of what makes certain influences on visual experiences by prior mental states (including desires, beliefs, moods, and fears) reduce the justificatory force of those experiences. The main idea is that experiences, like beliefs, can have rationally assessable etiologies, and when those etiologies are irrational, the experiences are epistemically downgraded.

Some philosophers associated with the post-Kantian Continental traditions in philosophy (for example, Marx and Nietzsche) think that the etiology of a belief can impugn the epistemic status of that belief, leading us, correctly, to be “suspicious” of it; let us call them “Etiological Critics. Many analytic philosophers, responding to these and related etiological critiques within Anglophone philosophy are unimpressed. These analytic philosophers agree that facts about the etiology of belief might bring to one’s attention epistemically relevant considerations—for example, the fact (...) that other possible epistemic peers disagree with one’s beliefs—but they deny that the etiology itself has any direct bearing on the epistemic status of belief (in particular, whether it is doxastically justified). I argue that etiology is directly relevant to the epistemic status of belief, arguing against White and Srinivasan, using examples from Marx and Nietzsche. (shrink)

In one approach to classifying island phenomena, there is a group that answers to the following description. ADJUNCT ISLAND CONDITION If an XP is in an adjunct position, nothing may move out of it. In the influential approach to this condition in Huang, “adjunct” position is defined in terms that reference argument structure and its reflection in phrasemarker geometry. This definition groups together subject phrases and modifying phrases, contrasting them with phrases in “complement” position. The subsequent bounding theories in Lasnik (...) and Saito and Chomsky build on this basic idea, but attempt to spread it to a wide variety of island effects, including those characterized by early versions of Chomsky’s Subjacency condition. Central to their approaches is the notion of “lexical governor,” which is responsible for making the complement/non-complement cut — only phrases that are governed by a suitably lexical Xo are “complements,” and the island conditions are defined, then, over all the others. This part of the system has fallen into disuse partly, I suspect, because characterizing the “lexical” versus “non-lexical” distinction never found itself grounded in something more general, and partly because it became unwieldy in the increasing richness of post-Pollock representations of phrase-markers. This paper adopts the view that there is an island condition like that in, which groups together subjects and adjuncts, but it does not attempt to define these phrases on the basis of a “lexical governor.” Instead, let us adopt a characterization of “adjunct” that is wholly geometric: An adjunct is a phrase whose sister is also a phrase and whose mother is not its projection. This will put together “subject” phrases and modifier phrases under the standard assumption that these are both necessarily sisters to phrases rather than heads. Thus it will single out the boxed phrases in. (shrink)

Ramathal et al. have employed an elegant xenotransplantation technique to study the fate of human induced pluripotent stem cells (hiPSCs) from fertile males and from males carrying Y chromosome deletions of the azoospermia factor (AZF) region. When placed in a mouse testis niche, hiPSCs from fertile males differentiate into germ cell‐like cells (GCLCs). Highlighting the crucial role of cell autonomous factors in male sterility, hiPSCs derived from azoospermic males prove to be less successful under similar circumstances. Their studies argue that (...) the agametic “Sertoli cell only” phenotype of two of the AZF deletions likely arises from a defect in the maintenance of germline stem cells (GSCs) rather than from a defect in their specification. These observations underscore the importance of the dialogue between the somatic niche and its inhabitant stem cells, and open up interesting questions concerning the functioning of the somatic niche and how it communicates to the GSCs. (shrink)

Can heritability estimates provide causal information? This paper argues for an affirmative answer: since a non-nil heritability estimate satisfies certain characteristic properties of causation (i.e., association, manipulability, and counterfactual dependence), it increases the probability that the relation between genotypic variance and phenotypic variance is (at least partly) causal. Contrary to earlier proposals in the literature, the argument does not assume the correctness of any particular conception of the nature of causation, rather focusing on properties that are characteristic of causal relationships. (...) The argument is defended against Lewontin's (1974) locality objection and Kaplan and Turkheimer's (2021) recent critique of Genome-Wide Association Studies (GWAS). (shrink)

The proper function of the heart is pumping the blood. According to what we call the type etiological view, this is because previous tokens of the type HEART were selected for pumping the blood. Nanay :412–431, 2010) argues that the type etiological view is viciously circular. He claims that the only plausible accounts of trait type individuation use proper functions, such that whenever the type etiological view is supplemented with a plausible account of trait type individuation, the result is a (...) view that uses proper functions to explain proper functions. We refine this objection, and argue that Nanay at most establishes a potentially benign definitional circularity. However, we show that the type etiological view’s reliance on types nevertheless generates a vicious regress. Hence the type etiological view is false. We reject dispositional and modal alternatives to the type etiological view for the reason that they either cannot accommodate malfunction, or do so at the cost of proliferation; and then formulate a novel token etiological view that overcomes both problems because it makes no reference to trait types. (shrink)

This article elaborates on Putnam's ''discrete behavioral states'' model of dissociative identity disorder (Putnam, 1997) by proposing the involvement of the orbitalfrontal cortex in the development of DID and suggesting a potential neurodevelopmental mechanism responsible for the development of multiple representations of self. The proposed ''orbitalfrontal'' model integrates and elaborates on theory and research from four domains: the neurobiology of the orbitalfrontal cortex and its protective inhibitory role in the temporal organization of behavior, the development of emotion regulation, the development (...) of the self, and experience-dependent reorganizing neocortical processes. The hypothesis being proposed is that the experience-dependent maturation of the orbitalfrontal cortex in early abusive environments, characterized by discontinuity in dyadic socioaffective interactions between the infant and the caregiver, may be responsible for a pattern of lateral inhibition between conflicting subsets of self-representations which are normally integrated into a unified self. The basic idea is that the discontinuity in the early caretaking environment is manifested in the discontinuity in the organization of the developing child's self. (shrink)

Objective. We investigated associations between adults’ beliefs about the heritability of virtue and endorsements of the efficacy of specific parenting styles. Design. In Studies 1 (N = 405) and 2 (N = 400), beliefs about both the genetic etiology of virtuous characteristics and parenting were assessed in samples of parents and non-parents. In Study 3 (N = 775), participants were induced to view virtue as determined by genes or as determined by social factors. Heritability beliefs and authoritarian parenting endorsements were (...) subsequently measured. Results. Study 1 and Study 2 converged to reveal that tendencies to view characteristics as determined by genes were positively associated with endorsement of authoritarian parenting styles. This association occurred independent of individual differences in essentialism and rightwing authoritarianism. Study 3 revealed that exposure to genetic accounts of virtue increased beliefs that virtue is caused by genes, which in turn was positively associated with endorsements of authoritarian parenting responses to child problem behavior. Exposure to genetic accounts of virtue increased endorsement of authoritarian parenting among parents, but was unrelated to authoritarian parenting among non-parents. Conclusions. These studies suggest that genetic accounts of virtuous characteristics reliably relate to more positive beliefs about harsh and controlling parenting practices, illuminating an unrecognized cognitive factor associated with authoritarian parenting endorsement. (shrink)

Is the traditional understanding of cause and effect in aetiology so certain that Arthur Eddington therefore proposed in 1927 "the arrow of time, or time's arrow" involving "the 'one-way direction' or 'asymmetry' of time", such that "a cause precedes its effect: the causal event occurs before the event it affects. Thus causality is intimately bound up with time's arrow"? (WK 2014) This certain view on cause and effect can be contrasted with an opposing view by Michael Dummett, who suggested instead, (...) back in 1957, that "there was no philosophical objection to effects preceding their causes", or what is now known as "retrocausality". (WK 2014a) Contrary to these opposing views (and other ones as will be discussed in the book), aetiology (in relation to cause and effect) are neither possible (or impossible) nor desirable (or undesirable) to the extent that the respective ideologues (on different sides) would like us to believe. Of course, this questioning of different opposing views on cause and effect does not mean that the study of aetiology is useless, or that those diverse fields (related to aetiology) -- like physics, engineering, biology, philosophy, medicine, epidemiology, government, geography, spatial analysis, psychology, statistics, mathematics, economics, management, history, law, sociology, theology, and so on -- are worthless. (WK 2014b & 2014c) In fact, neither of these extreme views is plausible. Rather, this book offers an alternative (better) way to understand the future of aetiology in regard to the dialectic relationship between cause and effect -- while learning from different approaches in the literature but without favouring any one of them (nor integrating them, since they are not necessarily compatible with each other). More specifically, this book offers a new theory (that is, the pluralist theory of aetiology) to go beyond the existing approaches in a novel way, and is organised in four chapters. (shrink)

Kengo Miyazono’s Delusions and Beliefs: A Philosophical Inquiry is an attempt to provide a unified account of the nature, pathology, and etiology of delusions. The strength of his book resides in the clarity of arguments and its consistent adoption of a biological explanation of delusions, based on teleo-functionalism about mental states. However, there are some weaknesses in each of his arguments regarding the nature, pathology, and etiology of delusions. Regarding the nature of delusions, teleo-functionalism makes it difficult to confirm that (...) delusions are beliefs. Regarding the pathology of delusions, I suggest that the existence of harmful malfunction is a necessary but not a sufficient condition for a belief to be considered pathological, and that additional evidence is needed to justify the separation of the belief and the holder’s personhood. Regarding the etiology, it is not reasonable to incorporate the prediction error theory into a kind of two-factor theory. Therefore, the details of his arguments require further modification. (shrink)

Cilj je studije bio utvrditi učiteljsku procjenu znanja o simptomima, etiologiji i tretmanu ADHD-a te njenu povezanost sa sociodemografskim obilježjima učitelja, obilježjima škole i učiteljevoj samoprocjeni općeg znanja o ADHD-u. Prigodan uzorak činilo je 233 učitelja. Primijenjen je Upitnik učiteljskih znanja o ADHD-u trofaktorske strukture: Simptomi, Etiologija i Tretman ADHD-a, odgovarajuće pouzdanosti. Učitelji poznaju važnost tretmana za akademske i socijalne ishode učenika s ADHD-om te simptome ADHD-a, dok iskazuju nedovoljno poznavanje etiologije ADHD-a. Prisutne su zablude u identificiranju uzroka ADHD-a u (...) obiteljskim stresnim okolnostima. Učiteljska procjena općeg znanja o ADHD-u povezana je s višom razinom znanja o simptomima i tretmanu, radnim mjestom učitelja razredne nastave, dok je viša razina znanja o tretmanu povezana s nižom kronološkom dobi učitelja. Implikacije dobivenih nalaza ukazuju na značaj stjecanja kompetencija učitelja za poučavanje učenika s ADHD-om tijekom inicijalnog obrazovanja i trajnog profesionalnog razvoja, kao i osiguravanja podrške stručnjaka edukacijskih rehabilitatora. The aim of this study was to determine the teachers’ level of their perceived knowledge about ADHD and the connection between the teachers’ perceived knowledge and their sociodemographic characteristics, characteristics of the school they are affiliated with and their self-assessment of their general knowledge about ADHD. The sample was convenient and included 233 teachers. We used the Questionnaire of Teachers’ Knowledge of ADHD with three factored structure: Symptoms, Etiology of ADHD and Treatment of ADHD, with the adequate internal consistency. Teachers are aware of the importance of the treatment for the academic and social outcomes of students with ADHD. They also recognize the symptoms of ADHD. Their knowledge of the etiology of ADHD is insufficient. There are misconceptions related to identifying the cause of ADHD in family stressful circumstances. The teachers’ assessment of their general knowledge of ADHD is connected to higher level of knowledge about the symptoms and treatment, and position of primary school teacher. Higher level of knowledge about the treatment is connected to lower age of the teachers. Implications of these findings point out to the importance of obtaining the relevant competences during the initial study of the teachers as well as during their further professional development. The importance of professional support provided by educational rehabilitators is also highlighted. (shrink)

Reincarnation and Biology:. Contribution to the Etiology of Birthmarks and Birth Defects. Ian Stevenson. Westport, CT, and London: Praeger, 1997. volumes. 1192 pp. $195.00 (cloth).