Search results for 'Adenosine' (try it on Scholar)

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  1.  1
    L. Sobrevia, Simon M. Jarvis & D. L. Yudilevich, Adenosine Transport in Cultured Human Umbilical Vein Endothelia-Cells is Reduced in Diabetes.
    Adenosine transport in cultured human umbilical vein endothelial cells (HUVEC) was characterized and shown to be mediated by a single facilitated diffusion mechanism. Initial rates of adenosine influx at 22 degrees C were saturable [apparent Michaelis constant, 69 +/- 10 mu M; maximum velocity (V-max), 600 +/- 70 pmol.10(6) cells(-1).s(-1)] and inhibited by nitrobenzylthioinosine (NBMPR). Formycin B had an unusually high affinity [inhibitory constant K-i), 18 +/- 4.3 mu M], whereas inosine had a low affinity (K-i, 440 +/- (...)
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  2. Wai Yiu Cheung (1972). Adenosine 3´, 5´-Monophosphate: On Its Mechanism of Action. Perspectives in Biology and Medicine 15 (2):221-236.
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  3. Jacob Sacks (1964). The Role of Adenosine Triphosphate in Muscular Contraction. Perspectives in Biology and Medicine 7 (3):285-296.
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  4. Randi J. Ulbricht & Ronald B. Emeson (2014). One Hundred Million Adenosine-to-Inosine RNA Editing Sites: Hearing Through the Noise. Bioessays 36 (8):730-735.
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    Koscak Maruyama (1991). The Discovery of Adenosine Triphosphate and the Establishment of Its Structure. Journal of the History of Biology 24 (1):145 - 154.
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  6.  24
    David J. Mellor, Tamara J. Diesch, Alistair J. Gunn & Laura Bennet (2005). The Importance of 'Awareness' for Understanding Fetal Pain. Brain Research Reviews 49 (3):455-471.
  7.  4
    Geoffrey Burnstock (2012). Purinergic Signalling: Its Unpopular Beginning, its Acceptance and its Exciting Future. Bioessays 34 (3):218-225.
    Adenosine 5′-triphosphate (ATP) was identified in 1970 as the transmitter responsible for non-adrenergic, non-cholinergic neurotransmission in the gut and bladder and the term ‘purinergic’ was coined. Purinergic cotransmission was proposed in 1976 and ATP is now recognized as a cotransmitter in all nerves in the peripheral and central nervous systems. P1 (adenosine) and P2 (ATP) receptors were distinguished in 1978. Cloning of these receptors in the early 1990s was a turning point in the acceptance of the purinergic signalling (...)
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    John N. Prebble (2010). The Discovery of Oxidative Phosphorylation: A Conceptual Off-Shoot From the Study of Glycolysis. Studies in History and Philosophy of Science Part C 41 (3):253-262.
    The origins of oxidative phosphorylation, initially known as aerobic phosphorylation, grew out of three research areas of muscle metabolism, creatine phosphorylation, aerobic metabolism of lactic acid in muscle, and studies on the nature and role of adenosine triphosphate . Much of this work centred round the laboratory of Otto Meyerhof, and most of those contributing to the study of aerobic phosphorylation were influenced by that laboratory: particularly Lipmann and also Ochoa. The work of Engelhardt on ATP levels in blood (...)
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  9. L. F. Barros, D. L. Yudilevich, Simon M. Jarvis, N. Beaumont, J. D. Young & S. A. Baldwin, Immunolocalisation of Nucleoside Transporters in Human Placental Trophoblast and Endothelial Cells: Evidence for Multiple Transporter Isoforms.
    Polyclonal antibodies raised against the human erythrocyte nucleoside transporter were used to investigate the distribution of the nucleoside transporters in the placenta. Immunoblots of brush-border membranes isolated from the human syncytiotrophoblast revealed a cross-reactive species that co-migrated with the erythrocyte nucleoside transporter as a broad band of apparent M(r) 55,000. In contrast, no labelling was detected in basal membranes containing a similar number of equilibrative nucleoside transporters as assessed by nitrobenzylthioinosine (NBMPR)-binding. The absence of cross-reactive epitopes in basal membranes and (...)
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