Search results for 'pathogenesis' (try it on Scholar)

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  1. Olli S. Miettinen & Kenneth M. Flegel (2003). Elementary Concepts of Medicine: VI. Genesis of Illness: Pathogenesis, Aetiogenesis. Journal of Evaluation in Clinical Practice 9 (3):325-327.score: 21.0
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  2. Reinhart Koselleck (1988). Critique and Crisis: Enlightenment and the Pathogenesis of Modern Society. Mit Press.score: 15.0
    In this way progressive bourgeois philosophy, which seemed to offer the promise of a unified and peaceful world, in fact produced just the opposite.The book ...
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  3. Vadim S. Rotenberg (2004). The Ontogeny and Asymmetry of the Highest Brain Skills and the Pathogenesis of Schizophrenia. Behavioral and Brain Sciences 27 (6):864-865.score: 15.0
    The most developed and the latest-to-mature mental skills represented in the creation of mono- versus polysemantic contexts are related respectively to the left and right frontal lobe. A polysemantic way of thinking is responsible for the subject's successful integration in the polydimensional world. The functional insufficiency of this right-hemispheric way of thinking displays a predisposition toward the development of mental disorders, including schizophrenia.
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  4. Susann Schweiger & Rainer Schneider (2003). The MID1/PP2A Complex: A Key to the Pathogenesis of Opitz BBB/G Syndrome. Bioessays 25 (4):356-366.score: 15.0
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  5. Anne Joutel (2011). Pathogenesis of CADASIL. Bioessays 33 (1):73-80.score: 15.0
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  6. Louis Arnorsson Sass (2001). Pathogenesis, Common Sense, and the Cultural Framework: A Commentary on Stanghellini. Philosophy, Psychiatry, and Psychology 8 (2):219-224.score: 15.0
  7. Anthony A. Nash (1985). The Viral Enigma. Viral Pathogenesis and Immunology. By CEDRIC A. MIMS and DAVID O. WHITE, Blackwell Scentific Publications, 1984. Pp. 398. £14.80. [REVIEW] Bioessays 3 (5):237-237.score: 15.0
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  8. Dominik Wodarz & Martin A. Nowak (2002). Mathematical Models of HIV Pathogenesis and Treatment. Bioessays 24 (12):1178-1187.score: 15.0
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  9. P. Burke (1988). Critique and Crisis: Enlightenment and the Pathogenesis of Modern Society Reinhart Koselleck (Leamington Spa, Hamburg and New York: Berg, 1988), X + 204pp., £25. [REVIEW] History of European Ideas 9 (6):762.score: 15.0
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  10. Peter Burke (1988). Critique and Crisis: Enlightenment and the Pathogenesis of Modern Society. History of European Ideas 9 (6):762-762.score: 15.0
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  11. V. Fóti (1982). Thought, Affect, Drive and Pathogenesis in Spinoza and Freud. History of European Ideas 3 (2):221-236.score: 15.0
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  12. June E. Osborn (1986). Co‐Factors and HIV: What Determines the Pathogenesis of AIDS? Bioessays 5 (6):287-289.score: 15.0
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  13. Francesco Pansera (1987). Pathogenesis of Osteoarthritis From an Evolutionary Perspective. Acta Biotheoretica 36 (4).score: 15.0
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  14. J. Wayne Streilein & Wade P. Parks (1986). Challenges: On the Pathogenesis of Immune Incompetence in the Acquired Immune Deficiency Syndrome. Bioessays 4 (6):286-291.score: 15.0
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  15. H. T. Wright (2000). Alzheimer's Disease and Related Disorders. Etiology, Pathogenesis, and Therapeutics. Bioessays 22 (7):682-683.score: 15.0
  16. J. S. Abramson (1987). The Pathogenesis of Bacterial Infections in Infants and Children: The Role of Viruses. Perspectives in Biology and Medicine 32 (1):63-72.score: 15.0
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  17. Hobson & Bishop (2004). The Pathogenesis of Autism: Insights From Congenital Blindness. In Uta Frith & Elisabeth Hill (eds.), Autism: Mind and Brain. Oup Oxford.score: 15.0
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  18. Andrea Cossarizza, Cristina Mussini & Alessandra Viganò (2001). Mitochondria in the Pathogenesis of Lipodystrophy Induced by Anti‐HIV Antiretroviral Drugs: Actors or Bystanders? Bioessays 23 (11):1070-1080.score: 15.0
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  19. Graham S. Jackson (2001). Spontaneous Conformational Change Within the Prion Protein—Implications for Disease Pathogenesis? Bioessays 23 (9):772-774.score: 15.0
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  20. Sabra L. Klein (2011). Implications of X‐Linked Gene Regulation for Sex Differences in Disease Pathogenesis (Comment on DOI 10.1002/Bies. 201100047). [REVIEW] Bioessays 33 (11):789-790.score: 15.0
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  21. R. A. McCance & E. M. Widdowson (1976). Obesity: Pathogenesis and Management. Edited by Trevor Silverstone. Pp 240. (Medical and Technical Publishing Co, Lancaster, 1975.) Price £8.50. [REVIEW] Journal of Biosocial Science 8 (2):175-176.score: 15.0
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  22. Laurence D. Moore (1984). Stimulating Toxins Toxins and Plant Pathogenesis J. M. Daley B. J. Deverall. BioScience 34 (10):662-662.score: 15.0
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  23. [deleted]Boguslaw Lipinski & Etheresia Pretorius (2013). The Role of Iron-Induced Fibrin in the Pathogenesis of Alzheimer's Disease and the Protective Role of Magnesium. Frontiers in Human Neuroscience 7.score: 15.0
  24. R. D. Terry (1992). The Pathogenesis of Alzheimer's Disease. What Causes Dementia? In Y. Christen & P. S. Churchland (eds.), Neurophilosophy and Alzheimer's Disease. Springer-Verlag. 123--130.score: 15.0
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  25. [deleted]Gerd Lehmkuhl Volker Sturm, Oliver Fricke, Christian P. Bührle, Doris Lenartz, Mohammad Maarouf, Harald Treuer, Jürgen K. Mai (2012). DBS in the Basolateral Amygdala Improves Symptoms of Autism and Related Self-Injurious Behavior: A Case Report and Hypothesis on the Pathogenesis of the Disorder. Frontiers in Human Neuroscience 6.score: 15.0
    We treated a thirteen year old boy for life-threatening self-injurious behavior (SIB) and severe Kanner’s autism with Deep Brain Stimulation (DBS) in the amygdaloid complex as well as in the supra-amygdaloid projection system. Two DBS-electrodes were placed in both structures of each hemisphere. The stimulation contacts targeted the paralaminar, the basolateral, the central amygdala as well as the supra-amygdaloid projection system. DBS was applied to each of these structures, but only stimulation of the baso-lateral part proved effective in improving SIB (...)
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  26. Per Westermark & Kenneth H. Johnson (1988). The Pathogenesis of Maturity‐Onset Diabetes Mellitus: Is There a Link to Islet Amyloid Polypeptide? Bioessays 9 (1):30-33.score: 15.0
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  27. Víctor de Lorenzo (2014). From Theselfish Genetoselfish Metabolism: Revisiting the Central Dogma. Bioessays 36 (3):226-235.score: 6.0
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  28. Stephen V. Gordon, Daria Bottai, Roxane Simeone, Timothy P. Stinear & Roland Brosch (2009). Pathogenicity in the Tubercle Bacillus: Molecular and Evolutionary Determinants. Bioessays 31 (4):378-388.score: 6.0
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  29. George R. Young, Jonathan P. Stoye & George Kassiotis (2013). Are Human Endogenous Retroviruses Pathogenic? An Approach to Testing the Hypothesis. Bioessays 35 (9):794-803.score: 6.0
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  30. Jakob Hohwy & Raben Rosenberg (2005). Unusual Experiences, Reality Testing and Delusions of Alien Control. Mind and Language 20 (2):141-162.score: 3.0
    Some monothematic types of delusions may arise because subjects have unusual experiences. The role of this experiential component in the pathogenesis of delusion is still not understood. Focussing on delusions of alien control, we outline a model for reality testing competence on unusual experiences. We propose that nascent delusions arise when there are local failures of reality testing performance, and that monothematic delusions arise as normal responses to these. In the course of this we address questions concerning the tenacity (...)
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  31. H. Nederbragt (2000). The Biomedical Disciplines and the Structure of Biomedical and Clinical Knowledge. Theoretical Medicine and Bioethics 21 (6):553-566.score: 3.0
    The relation between biomedical knowledge and clinicalknowledge is discussed by comparing their respectivestructures. The knowledge of a disease as a biologicalphenomenon is constructed by the interaction of factsand theories from the main biomedical disciplines:epidemiology, diagnostics, clinical trial, therapydevelopment and pathogenesis. Although these facts andtheories are based on probabilities andextrapolations, the interaction provides a reliableand coherent structure, comparable to a Kuhnianparadigma. In the structure of clinical knowledge,i.e. knowledge of the patient with the disease, notonly biomedical knowledge contributes to the structurebut (...)
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  32. P. Thagard (1998). Ulcers and Bacteria I: Discovery and Acceptance. Studies in History and Philosophy of Science Part C 29 (1):107-136.score: 3.0
    In 1983, Dr. J. Robin Warren and Dr. Barry Marshall reported finding a new kind of bacteria in the stomachs of people with gastritis. Warren and Marshall were soon led to the hypothesis that peptic ulcers are generally caused, not by excess acidity or stress, but by a bacterial infection. Initially, this hypothesis was viewed as preposterous, and it is still somewhat controversial. In 1994, however, a U. S. National Institutes of Health Consensus Development Panel concluded that infection appears to (...)
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  33. Ruth Condray & Stuart R. Steinhauer (2002). The Residual Normality Assumption and Models of Cognition in Schizophrenia. Behavioral and Brain Sciences 25 (6):753-754.score: 3.0
    Thomas & Karmiloff-Smith’ (T&K-S’) argument that the Residual Normality assumption is not valid for developmental disorders has implications for models of cognition in schizophrenia, a disorder that may involve a neurodevelopmental pathogenesis. A limiting factor for such theories is the lack of understanding about the nature of the cognitive system (modular components versus global processes). Moreover, it is unclear how the proposal that modularization emerges from developmental processes would change that fundamental question.
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  34. Josef Egger (1986). Psychological Risk Factors in Cardiovascular Diseases. Theoretical Medicine and Bioethics 7 (3).score: 3.0
    Recent research has shown that psychological risk factors play an important role in the pathogenesis of cardiovascular diseases. The so-called coronary prone behaviour pattern predominates, an important part of which is the Type A behaviour pattern. This is characterized by a marked ambition, a constant feeling of being under pressure, due to latent aggression and to a striving to dominate. For cerebrovascular diseases the so-called pressured pattern as a risk factor has been found to be typical which is comparable (...)
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  35. Russell Gardner (1997). Sociophysiology as the Basic Science of Psychiatry. Theoretical Medicine and Bioethics 18 (4).score: 3.0
    The medical specialty of psychiatry should possess a basic science in which pathologies are considered deviations from normal brain physiology. Historically, psychoanalytic pathogenesis was considered separately from brain physiology. It was not scientific because observations could not be refuted. Countering this, Eli Robins's legacy stemmed partly from his having been damaged by a psychoanalyst. It eschewed pathogenesis. Attempting to integrate psychiatry with medicine more generally, Robins and colleagues refocused on empiricism, although they acknowledged the brain's centrality. Here I (...)
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  36. Paul Thagard, Discovery and Acceptance.score: 3.0
    In 1983, Dr. J. Robin Warren and Dr. Barry Marshall reported finding a new kind of bacteria in the stomachs of people with gastritis. Warren and Marshall were soon led to the hypothesis that peptic ulcers are generally caused, not by excess acidity or stress, but by a bacterial infection. Initially, this hypothesis was viewed as preposterous, and it is still somewhat controversial. In 1994, however, a U. S. National Institutes of Health Consensus Development Panel concluded that infection appears to (...)
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  37. Paolo Vezzoni, Maria Rosa Pozzi & Anna Villa (1989). The Rise of a Microparadigm in Oncology. Biology and Philosophy 4 (1):57-67.score: 3.0
    The study of the history of ideas is usually devoted to big problems and to concluded debates. We have attempted to analyze a current theory whose fate and explanatory power is still not determined. The term microparadigm is used to define a currently and widely accepted theory limited in time and in the field of application, compared to the greater problems usually investigated by historians of science. Among the characteristics defining a microparadigm we found: 1) the status of an accepted (...)
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  38. Giovanni Felice Azzone (1998). The Cement of Medical Thought. Evolutionary Emergence and Downward Causation. History and Philosophy of the Life Sciences 20 (2):163 - 187.score: 3.0
    The aetio-pathogenetic sequences and the physio-pathological patterns of diabetes, emphysema, cholera, circulatory shock and thrombosis have been analysed with respect to an evolutionary interpretation. The diseases, although reflecting alterations of processes that can always be described in physico-chemical language, occur only at the level of biological systems which reflects the decodification of genomic project: the teleonomic projects that have been developed during evolution. The concepts of evolutionary emergence and of downward causation have been used to discuss the relationship between the (...)
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  39. Wim Dekkers & Marcel Olde Rikkert (2006). What is a Genetic Cause? The Example of Alzheimer's Disease. Medicine, Health Care and Philosophy 9 (3):273-284.score: 3.0
    This paper focuses on the causation of diseases, particularly on the idea of a “genetic cause” taking Alzheimer’s Disease (AD) as an example. We (1) provide some historical information and a synopsis of the current knowledge on the etiology and pathogenesis of AD, (2) analyse some conceptual problems related to the notion of “genetic disease” (3) elaborate on the alleged (genetic) cause of AD, and (4) place the discussion on the cause of AD in a broader philosophical context, paying (...)
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  40. Jochen Schaefer (1980). The Case Against Coronary Artery Surgery. Theoretical Medicine and Bioethics 1 (2):155-176.score: 3.0
    Coronary by-pass surgery has been performed in hundreds of thousands of patients in the last 15 years with a high standard of technical and surgical perfection. The indications for this kind of surgery, however, are still controversial because in spite of many retrospective and several prospective studies it cannot be proven convincingly that in a given patient this surgical procedure will prolong life or prevent myocardial infarction. The present attempt to analyze the causes for this controversy shows that the main (...)
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  41. Ganesan Venkatasubramanian (2008). Evolutionary Perspectives on Psychoses and Autism: Does Genomic Imprinting Contribute to Phenomenological Antithesis? Behavioral and Brain Sciences 31 (3):281-282.score: 3.0
    Crespi & Badcock (C&B) have presented a novel view that the influence of genomic imprinting causes diametrically opposite disorders: namely, psychoses and autism. I propose an extended hypothesis that while genomic imprinting is likely to have an influence on the pathogenesis of psychoses and autism, it might contribute to phenomenological antithesis between as well as within these disorders.
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  42. Christoph Gradmann (2001). Isolation, Contamination, and Pure Culture: Monomorphism and Polymorphism of Pathogenic Micro-Organisms as Research Problem 1860-1880. Perspectives on Science 9 (2):147-172.score: 3.0
    : This article analyzes German debates on the microbiology of infectious diseases from 1865 to 1875 and asks how and when organic pollution in tissues became noteworthy for aetiology and pathogenesis. It was with Ernst Hallier's pleomorphistic microbiology that the organic character of alien material in tissues came to be regarded as important for pathology. The process that followed saw both vigorous biological critique and a number of medical applications of Hallier's work. Around 1874 contemporaries reached the conclusion that (...)
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  43. Sabine Brauckmann (2000). Steps Towards an Ecology of Cognition. Sign Systems Studies 28:397-419.score: 3.0
    The essay infonns on Gregory Bateson's holistic approach towards an epistemic view of nature. The ecology of mind relies upon a biological holism serving as a methodic tool to explain living "phenomena", like, e.g., communication, learning, and cognition. Starting from the idea, the smallest unit of information, Bateson developed a type hierarchy of learning that is based on a cybernetic view of mind. The communication model focuses on paradoxa caused by false signification. It leads to a pathogenesis of sckizophrenia (...)
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  44. Camilo A. L. S. Colaco & Bruce J. Roser (1994). Atherosclerosis and Glycation. Bioessays 16 (2):145-147.score: 3.0
    Atherosclerosis is the major cause of death in the industrialised world. Though much work on the pathogenesis of atherosclerosis points to 'oxidised' low density lipoprotein (LDL) as a key aetiological feature in the generation of the atherosclerotic plaque, the nature of this 'oxidised' LDL in vivo remains an enigma. We argue here that glycated LDL shows many of the characteristics attributed to 'oxidised LDL' and may be the source of the latter in vivo. These include the increased uptake and (...)
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  45. S. Camporesi & G. Boniolo (2008). Fearing a Non-Existing Minotaur? The Ethical Challenges of Research on Cytoplasmic Hybrid Embryos. Journal of Medical Ethics 34 (11):821-825.score: 3.0
    In this paper we address the ethical challenges of research on cytoplasmic hybrid embryos, or “cybrids”. The controversial pronouncement of the UK’s Human Embryology and Fertilisation Authority of September 2007 on the permissibility of this area of research is the starting point of our discussion, and we argue in its favour. By a rigorous definition of the entities at issue, we show how the terms “chimera” and “hybrid” are improper in the case of cybrids, and how their use can bias (...)
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  46. Edmond A. Murphy (1997). The Logic of Medicine. Johns Hopkins University Press.score: 3.0
    When first published twenty years ago, The Logic of Medicine presented a new way of thinking about clinical medicine as a scholarly discipline as well as a profession. Since then, advances in research and technology have revolutionized both the practice and theory of medicine. In this new, extensively rewritten edition, Dr. Murphy includes changes to show how these different areas of scholarship may affect details of "the logic of medicine" without compromising its fundamental coherence. New to this edition are discussions (...)
     
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