Switch to: References

Add citations

You must login to add citations.
  1. Do Humans Have Two Systems to Track Beliefs and Belief-Like States?Stephen Andrew Butterfill & Ian A. Apperly - 2009 - Psychological Review 116 (4):953-970.
    The lack of consensus on how to characterize humans’ capacity for belief reasoning has been brought into sharp focus by recent research. Children fail critical tests of belief reasoning before 3 to 4 years (Wellman, Cross, & Watson, 2001; Wimmer & Perner, 1983), yet infants apparently pass false belief tasks at 13 or 15 months (Onishi & Baillargeon, 2005; Surian, Caldi, & Sperber, 2007). Non-human animals also fail critical tests of belief reasoning but can show very complex social behaviour (e.g., (...)
    Direct download (4 more)  
    Export citation  
    Bookmark   157 citations  
  • Cognitive? Science?J. Ignacio Serrano, M. Dolores del Castillo & Manuel Carretero - 2014 - Foundations of Science 19 (2):115-131.
    Cognitive Science is a promising field of research that deals with one of the most fundamental questions ever: how do beings know? However, despite the long and extensive tradition of the field it has not yet become an area of knowledge with scientific identity. This is primarily due to three reasons: the lack of boundaries in defining the object of study, i.e. cognition, the lack of a precise, robust and consistent scientific methodology and results, and the inner problems derived from (...)
    Direct download (3 more)  
    Export citation  
    Bookmark   1 citation  
  • Modularity in Philosophy, the Neurosciences, and Psychiatry.Jürgen Zielasek & Wolfgang Gaebel - 2008 - Poiesis and Praxis 6 (1-2):93-108.
    The neurosciences are generating new findings regarding genetic and neurobiological aspects of the pathophysiology of mental disorders. Especially, certain genetic risk factors like neuregulin-1 seem to predispose individuals to a psychotic phenotype beyond the limits of traditional classificatory boundaries between organic psychoses in Alzheimer’s disease, bipolar affective disorder and schizophrenia. Little, however, is known about how such genetic risk factors actually confer an increased risk for psychosis in an individual patient. A gap between neuroscientific findings and psychopathological phenomena exists. The (...)
    Direct download (6 more)  
    Export citation