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  1. What neurodevelopmental disorders can reveal about cognitive architecture.Helen Tager-Flusberg - 2005 - In Peter Carruthers, Stephen Laurence & Stephen P. Stich (eds.), The Innate Mind: Structure and Contents. New York, US: Oxford University Press USA. pp. 272--288.
    This chapter begins with an overview of the controversy surrounding the study of children and adults with neurodevelopmental disorders, and how these inform theories of neurocognitive architecture. It weighs the arguments for and against what we might learn from studying individuals who have fundamental biological impairments. It then discusses the example of research on theory of mind in two different disorders — autism and Williams syndrome — which has highlighted a number of important aspects of how this core cognitive capacity (...)
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  • Social Cognition in Williams Syndrome: Relations between Performance on the Social Attribution Task and Cognitive and Behavioral Characteristics.Faye van der Fluit, Michael S. Gaffrey & Bonita P. Klein-Tasman - 2012 - Frontiers in Psychology 3.
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  • Object recognition with severe spatial deficits in Williams syndrome: sparing and breakdown.Barbara Landau, James E. Hoffman & Nicole Kurz - 2006 - Cognition 100 (3):483-510.
  • Dissociating intuitive physics from intuitive psychology: Evidence from Williams syndrome.Frederik S. Kamps, Joshua B. Julian, Peter Battaglia, Barbara Landau, Nancy Kanwisher & Daniel D. Dilks - 2017 - Cognition 168 (C):146-153.
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  • Social Brain Development in Williams Syndrome: The Current Status and Directions for Future Research.Brian W. Haas & Allan L. Reiss - 2012 - Frontiers in Psychology 3.
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  • Ontogeny, Genetics, and Evolution: A Perspective from Developmental Cognitive Neuroscience.Annette Karmiloff-Smith - 2006 - Biological Theory 1 (1):44-51.
    The study of genetic developmental disorders originally seemed to hold the promise for those of a nativist persuasion of demonstrating pure dissociations between different cognitive functions, as well as the existence of innately specified modules in the brain and the direct mapping of mutated genes to specific cognitive-level outcomes. However, more recent research within a neuroconstructivist perspective has challenged this promise, arguing that earlier researchers lost sight of one fundamental explanatory factor in both the typical and atypical case: the actual (...)
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