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Lesion location and aphasic syndrome do not tell us whether a patient will have an isolated deficit affecting the coindexation of traces

Published online by Cambridge University Press:  28 September 2001

David Caplan
David Caplan
Affiliation:
Department of Neurology, Harvard Medical School, Boston, MA 02225 and Neuropsychology Lab, Massachusetts General Hospital, Boston, MA 02116 caplan@helix.mgh.harvard.edu
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Abstract

Data from published case and group studies bear on the trace deletion hypothesis. The deficit-lesion correlational literature does not support Grodzinsky's claim that lesions in and around Broca's area inevitably lead to comprehension deficits specifically related to coindexation of traces or his claim that other lesions spare this function.

Type
Brief Report
Information
Behavioral and Brain Sciences , Volume 23 , Issue 1 , February 2000 , pp. 25 - 27
Copyright
© 2000 Cambridge University Press

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