Causality offers the first comprehensive coverage of causal analysis in many sciences, including recent advances using graphical methods. Pearl presents a unified account of the probabilistic, manipulative, counterfactual and structural approaches to causation, and devises simple mathematical tools for analyzing the relationships between causal connections, statistical associations, actions and observations. The book will open the way for including causal analysis in the standard curriculum of statistics, artificial intelligence, business, epidemiology, social science and economics.

In this important book, Ellery Eells explores and refines philosophical conceptions of probabilistic causality. In a probabilistic theory of causation, causes increase the probability of their effects rather than necessitate their effects in the ways traditional deterministic theories have specified. Philosophical interest in this subject arises from attempts to understand population sciences as well as indeterminism in physics. Taking into account issues involving spurious correlation, probabilistic causal interaction, disjunctive causal factors, and temporal ideas, Professor Eells advances the analysis of (...) what it is for one factor to be a positive causal factor for another. A salient feature of the book is a theory of token level probabilistic causation in which the evolution of the probability of a later event from an earlier event is central. This will be a book of crucial significance to philosophers of science and metaphysicians; it will also prove stimulating to many economists, psychologists, and physicists. (shrink)

In this classic, David Bohm was the first to offer us his causal interpretation of the quantum theory. _Causality and Chance in Modern Physics_ continues to make possible further insight into the meaning of the quantum theory and to suggest ways of extending the theory into new directions.

I discuss an important feature of the notion of cause in Post. An. 1. 13, 78b13–28, which has been either neglected or misunderstood. Some have treated it as if Aristotle were introducing a false principle about explanation; others have understood the point in terms of coextensiveness of cause and effect. However, none offers a full exegesis of Aristotle's tangled argument or accounts for all of the text's peculiarities. My aim is to disentangle Aristotle's steps to show that he is arguing (...) in favour of a logical requirement for a middle term's being the appropriate cause of its explanandum. Coextensiveness between the middle term and the attribute it explains is advanced as a sine qua non condition of a middle term's being an appropriate or primary cause. This condition is not restricted either to negative causes or to middle terms in second‐figure syllogisms, but ranges over all primary causes qua primary. (shrink)

Applied Evolutionary Epistemology is a scientific-philosophical theory that defines evolution as the set of phenomena whereby units evolve at levels of ontological hierarchies by mechanisms and processes. This theory also provides a methodology to study evolution, namely, studying evolution involves identifying the units that evolve, the levels at which they evolve, and the mechanisms and processes whereby they evolve. Identifying units and levels of evolution in turn requires the development of ontological hierarchy theories, and examining mechanisms and processes necessitates theorizing (...) about causality. Together, hierarchy and causality theories explain how biorealities form and diversify with time. This paper analyzes how Applied EE redefines both hierarchy and causality theories in the light of the recent explosion of network approaches to causal reasoning associated with studies on reticulate and macroevolution. Causality theories have often been framed from within a rigid, ladder-like hierarchy theory where the rungs of the ladder represent the different levels, and the elements on the rungs represent the evolving units. Causality then is either defined reductionistically as an upward movement along the strands of a singular hierarchy, or holistically as a downward movement along that same hierarchy. Upward causation theories thereby analyze causal processes in time, i.e. over the course of natural history or phylogenetically, as Darwin and the founders of the Modern Synthesis intended. Downward causation theories analyze causal processes in space, ontogenetically or ecologically, as the current eco-evo-devo schools are evidencing. This work demonstrates how macroevolution and reticulate evolution theories add to the complexity by examining reticulate causal processes in space–time, and the interactional hierarchies that such studies bring forth introduce a new form of causation that is here called reticulate causation. Reticulate causation occurs between units and levels belonging to different as well as to the same ontological hierarchies. This article concludes that beyond recognizing the existence of multiple units, levels, and mechanisms or processes of evolution, also the existence of multiple kinds of evolutionary causation as well as the existence of multiple evolutionary hierarchies needs to be acknowledged. This furthermore implies that evolution is a pluralistic process divisible into different kinds. (shrink)

We argue that the health sciences make causal claims on the basis of evidence both of physical mechanisms, and of probabilistic dependencies. Consequently, an analysis of causality solely in terms of physical mechanisms or solely in terms of probabilistic relationships, does not do justice to the causal claims of these sciences. Yet there seems to be a single relation of cause in these sciences - pluralism about causality will not do either. Instead, we maintain, the health sciences require (...) a theory of causality that unifies its mechanistic and probabilistic aspects. We argue that the epistemic theory of causality provides the required unification. (shrink)

In the present article, I introduce Husserl’s analyses of ‘natural causality’ and ‘volitional causality’, which are collected in the volume ‘Wille und Handlung’ of the Husserliana edition Studien zur Struktur des Bewußtseins. My aim is to show that Husserl’s insight into these phenomena enables us to understand more clearly both the specificity of, and the relation between, the motivational nexus belonging to the sphere of the will in contrast with the causal laws of nature. In light of this (...) understanding, in the last part of the article I reflect on whether and to what extent there is, in fact, an ontological and epistemological compatibility between volitional causality and natural causality. (shrink)

Philosophers of mind have long been interested in the relation between two ideas: that causality plays an essential role in our understanding of the mental; and that we can gain an understanding of belief and desire by considering the ascription of attitudes to people on the basis of what they say and do. Many have thought that those ideas are incompatible. William Child argues that there is in fact no tension between them, and that we should accept both. He (...) shows how we can have a causal understanding of the mental without having to see attitudes and experiences as internal, causally interacting entities and he defends this view against influential objections. The book offers detailed discussions of many of Donald Davidson's contributions to the philosophy of mind, and also considers the work of Dennett, Anscombe, McDowell, and Rorty, among others. Issues discussed include: the nature of intentional phenomena; causal explanation; the character of visual experience; psychological explanation; and the causal relevance of mental properties. (shrink)

I IT is often declared or evidently assumed that causality is some kind of necessary connexion, or alternatively, that being caused is — non-trivially ...

This paper presents a drastically revised version of the theory of causality, based on analyses of causal processes and causal interactions, advocated in Salmon (1984). Relying heavily on modified versions of proposals by P. Dowe, this article answers penetrating objections by Dowe and P. Kitcher to the earlier theory. It shows how the new theory circumvents a host of difficulties that have been raised in the literature. The result is, I hope, a more satisfactory analysis of physical causality.

After clarifying the probabilistic conception of causality suggested by Good (1961-2), Suppes (1970), Cartwright (1979), and Skyrms (1980), we prove a sufficient condition for transitivity of causal chains. The bearing of these considerations on the units of selection problem in evolutionary theory and on the Newcomb paradox in decision theory is then discussed.

This paper discusses several distinct process theories of causality offered in recent years by Phil Dowe and me. It addresses problems concerning the explication of causal process, causal interaction, and causal transmission, whether given in terms of transmission of marks, transmission of invariant or conserved quantities, or mere possession of conserved quantities. Renouncing the mark-transmission and invariant quantity criteria, I accept a conserved quantity theory similar to Dowe's--differing basically with respect to causal transmission. This paper also responds to several (...) fundamental constructive criticisms contained in Christopher Hitchcock's discussion of both the mark-transmission and the conserved quantity theories. (shrink)

A theory of causality based upon physical processes is developed. Causal processes are distinguished from pseudo-processes by means of a criterion of mark transmission. Causal interactions are characterized as those intersections of processes in which the intersecting processes are mutually modified in ways which persist beyond the point of intersection. Causal forks of three kinds (conjunctive, interactive, and perfect) are introduced to explicate the principle of the common cause. Causal forks account for the production of order and modifications of (...) order; causal processes account for the propagation of causal influence. (shrink)

David Hume and other philosophers doubt that causality can be perceived directly. Instead, observers become aware of it through inference based on the perception of the two events constituting cause and effect of the causal relation. However, Hume and the other philosophers primarily consider causal relations in which one object triggers a motion or change in another. In this paper, I will argue against Hume’s assumption by distinguishing a kind of causal relations in which an agent is controlling the (...) motion or change of an object. I will call this kind of causal relations ‘causation-as-control’. In instances of causation-as-control, the observer does not become aware of the causality through inference based on the perception of two events (cause and effect). Rather, she perceives the two events directly and without further inference as cause and effect of a causal relation, and, therefore, the causality at work. (shrink)

Why is understanding causation so important in philosophy and the sciences? Should causation be defined in terms of probability? Whilst causation plays a major role in theories and concepts of medicine, little attempt has been made to connect causation and probability with medicine itself. Causality, Probability, and Medicine is one of the first books to apply philosophical reasoning about causality to important topics and debates in medicine. Donald Gillies provides a thorough introduction to and assessment of competing theories (...) of causality in philosophy, including action-related theories, causality and mechanisms, and causality and probability. Throughout the book he applies them to important discoveries and theories within medicine, such as germ theory; tuberculosis and cholera; smoking and heart disease; the first ever randomized controlled trial designed to test the treatment of tuberculosis; the growing area of philosophy of evidence-based medicine; and philosophy of epidemiology. This book will be of great interest to students and researchers in philosophy of science and philosophy of medicine, as well as those working in medicine, nursing and related health disciplines where a working knowledge of causality and probability is required. (shrink)

"I regard it as a truly seminal work in this field." — Professor William A. Wallace, author of Causality and Scientific Explanation. Non-technical and clearly written, this book focuses on the place of the casual principle in modern science. The author defines the terminology, describes various formulations, examines the two primary critiques of causality, and more.

The anti-causal prophecies of last century have been disproved. Causality is neither a ‘relic of a bygone’ nor ‘another fetish of modern science’; it still occupies a large part of the current debate in philosophy and the sciences. This investigation into causal modelling presents the rationale of causality, i.e. the notion that guides causal reasoning in causal modelling. It is argued that causal models are regimented by a rationale of variation, nor of regularity neither invariance, thus breaking down (...) the dominant Human paradigm. The notion of variation is shown to be embedded in the scheme of reasoning behind various causal models: e.g. Rubin’s model, contingency tables, and multilevel analysis. It is also shown to be latent – yet fundamental – in many philosophical accounts. Moreover, it has significant consequences for methodological issues: the warranty of the causal interpretation of causal models, the levels of causation, the characterisation of mechanisms, and the interpretation of probability. This book offers a novel philosophical and methodological approach to causal reasoning in causal modelling and provides the reader with the tools to be up to date about various issues causality rises in social science. (shrink)

In a recent paper (1994) Wesley Salmon has replied to criticisms (e.g., Dowe 1992c, Kitcher 1989) of his (1984) theory of causality, and has offered a revised theory which, he argues, is not open to those criticisms. The key change concerns the characterization of causal processes, where Salmon has traded "the capacity for mark transmission" for "the transmission of an invariant quantity." Salmon argues against the view presented in Dowe (1992c), namely that the concept of "possession of a conserved (...) quantity" is sufficient to account for the difference between causal and pseudo processes. Here that view is defended, and important questions are raised about the notion of transmission and about gerrymandered aggregates. (shrink)

Hall has recently argued that there are two concepts of causality, picking out two different kinds of causal relation. McGrath, and Hitchcock and Knobe, have recently argued that the facts about causality depend on what counts as a “default” or “normal” state, or even on the moral facts. In the light of these claims you might be tempted to agree with Skyrms that causal relations constitute, metaphysically speaking, an “amiable jumble”, or with Cartwright that ‘causation’, though a single (...) word, encompasses many different kinds of things. This paper argues, drawing on the author’s recent work on explanation, that the evidence adduced in support of causal pluralism can be accommodated easily by a unified theory of causality—a theory according to which all singular causal claims concern the same fundamental causal network. (shrink)

This article is focused on the theory of nature (ṭabʽ) advocated by some of the early Muʽtazilī scholars such as Muʻammar b. ʽAbbād al-Sulamī (d. 215/830), Abū Isḥāq al-Naẓẓām (d. 231/845), Abū ʽUthmān al-Jāḥiẓ (d. 255/869) and Abū al-Qāsim al-Kaʽbī (d. 319/931) and its consequences about causality and miracle. The supporters of the ṭabʽ theory argue that Allah creates all beings with innate and permanent natures and these natures determine all movements and events in universe, and that necessary causal (...) relationships based on it prevail in the universe. But this understanding was criticized and rejected by the majority of Muslim theologians. The focal point of the criticism is God’s agency and His relationship with the universe. The acceptance of such necessary causality in the universe would make it impossible for God to intervene in nature, that is to create a miracle. So, miracles will be the work of the place where it occurs like all other acts when the concept of nature is accepted. In this case, it will not be possible to prove the omnipotence of God and belief in prophethood, which forms the basis of two principles of Islamic belief. This study deals with the dimensions of compulsory causality in the thought of naturalist Muslim theologians by addressing two questions: how the issue of miracle is grounded, and whether a divine intervention in nature of entities is possible. (shrink)

"A rich collection. Since it holds a number of introductory pieces along with advanced essays and review articles, the volume will be accessible to a broad audience and will work well in philosophy of science courses....Essential."--Lawrence Sklar, University of Michigan.

In the last decades, Systems Biology (including cancer research) has been driven by technology, statistical modelling and bioinformatics. In this paper we try to bring biological and philosophical thinking back. We thus aim at making diferent traditions of thought compatible: (a) causality in epidemiology and in philosophical theorizing—notably, the “sufcient-component-cause framework” and the “mark transmission” approach; (b) new acquisitions about disease pathogenesis, e.g. the “branched model” in cancer, and the role of biomarkers in this process; (c) the burgeoning of (...) omics research, with a large number of “signals” and of associations that need to be interpreted. In the paper we summarize frst the current views on carcinogenesis, and then explore the relevance of current philosophical interpretations of “cancer causes”. We try to ofer a unifying framework to incorporate biomarkers and omic data into causal models, referring to a position called “evidential pluralism”. According to this view, causal reasoning is based on both “evidence of diference-making” (e.g. associations) and on “evidence of underlying biological mechanisms”. We conceptualize the way scientists detect and trace signals in terms of information transmission, which is a generalization of the mark transmission theory developed by philosopher Wesley Salmon. Our approach is capable of helping us conceptualize how heterogeneous factors such as micro and macro-biological and psycho-social—are causally linked. This is important not only to understand cancer etiology, but also to design public health policies that target the right causal factors at the macro-level. (shrink)

In this thesis, I give a metascientific account of causality in medicine. I begin with two historical cases of causal discovery. These are the discovery of the causation of Burkitt’s lymphoma by the Epstein-Barr virus, and of the various viral causes suggested for cervical cancer. These historical cases then support a philosophical discussion of causality in medicine. This begins with an introduction to the Russo- Williamson thesis (RWT), and discussion of a range of counter-arguments against it. Despite these, (...) I argue that the RWT is historically workable, given a small number of modifications. I then expand Russo and Williamson’s account. I first develop their suggestion that causal relationships in medicine require some kind of evidence of mechanism. I begin with a number of accounts of mechanisms and produce a range of consensus features of them. I then develop this consensus position by reference to the two historical case studies with an eye to their operational competence. In particular, I suggest that it is mechanistic models and their representations which we are concerned with in medicine, rather than the mechanism as it exists in the world. -/- I then employ these mechanistic models to give an account of the sorts of evidence used in formulating and evaluating causal claims. Again, I use the two human viral oncogenesis cases to give this account. I characterise and distinguish evidence of mechanism from evidence of difference-making, and relate this to mechanistic models. I then suggest the relationship between types of evidence presents us with a means of tackling the reference-class problem. This sets the scene for the final chapter. Here, I suggest the manner in which these two different classes of evidence become integrated is also reflected in the way that developing research programmes change as their associated causal claims develop. (shrink)

Process theories of causality seek to explicate causality as a property of individual causal processes. This paper examines the capacity of such theories to account for the asymmetry of causation. Three types of theories of asymmetry are discussed; the subjective, the temporal, and the physical, the third of these being the preferred approach. Asymmetric features of the world, namely the entropic and Kaon arrows, are considered as possible sources of causal asymmetry and a physical theory of asymmetry is (...) subsequently developed with special reference to the questions of objectivity and backwards causation. (shrink)

Recently, it has been argued that the use of Big Data transforms the sciences, making data-driven research possible and studying causality redundant. In this paper, I focus on the claim on causal knowledge by examining the Big Data project EXPOsOMICS, whose research is funded by the European Commission and considered capable of improving our understanding of the relation between exposure and disease. While EXPOsOMICS may seem the perfect exemplification of the data-driven view, I show how causal knowledge is necessary (...) for the project, both as a source for handling complexity and as an output for meeting the project’s goals. Consequently, I argue that data-driven claims about causality are fundamentally flawed and causal knowledge should be considered a necessary aspect of Big Data science. In addition, I present the consequences of this result on other data-driven claims, concerning the role of theoretical considerations. I argue that the importance of causal knowledge and other kinds of theoretical engagement in EXPOsOMICS undermine theory-free accounts and suggest alternative ways of framing science based on Big Data. (shrink)

Human freedom is often characterised as a unique power of self-determination. Accordingly, free human action is often thought to be determined by the agent in some distinctive manner. What is more, this determination is widely assumed to be a kind of efficient-causal determination. In reaction to this efficient-causal-deterministic conception of free human action, this paper argues that if one takes up the understanding of determination and causality that is offered by Anscombe in ‘Causality and Determination’, and moreover takes (...) up an understanding of free human action that is constrained by Anscombe’s account of intentional action in Intention, then an account of free human action as distinctively caused or determined by the agent is untenable. However, the notion of necessitation that Anscombe presents in ‘Causality and Determination’, which implies neither causality nor determination, offers an attractive alternative account. This alternative account pushes us to reconsider the sense in which human freedom is a power of self-determination, and to acknowledge the limits of our control in free action. (shrink)

It is claimed that the `problem of the arrow of time in classical dynamics' has been solved. Since all classical particles have a self-field (gravitational and in some cases also electromagnetic), their dynamics must include self-interaction. This fact and the observation that the domain of validity of classical physics is restricted to distances not less than of the order of a Compton wavelength (thus excluding point particles), leads to the conclusion that the fundamental classical equations of motion are not invariant (...) under time reversal: retarded self-interactions lead to different equations than advanced ones. Since causality (the time order of cause and effect) requires retarded rather than advanced self-interaction, it is causality which is ultimately responsible for the arrow of time. Classical motions described by equations with advanced self-interactions differ from retarded ones and do not occur in nature. (shrink)

The article presents the question of understanding divine causality and its analogical character in the context of Thomas Aquinas’s teaching on Divine Providence. Analyzing Aquinas’s texts concerning the relation of God’s action towards nature and its activities it is necessary to emphasize the proper understanding of mutual relations between secondary causes and the primary cause which are not on the same level. Influenced by the reflection of M. Dodds and I Silva, the author of the article refers to Aquinas’s (...) biblical commentaries which have not been discussed so far from the perspective of the character of God’s action. In the final part of the article, metaphors used by Thomas in reference to the relation of God towards the world will be presented. (shrink)

The finding of fractal scaling (FS) in behavioral sequences has raised a debate on whether FS is a pervasive property of the cognitive system or is the result of specific processes. Inferences about the origins of properties in time sequences are causal. That is, as opposed to correlational inferences reflecting instantaneous symmetrical relations, causal inferences concern asymmetric relations lagged in time. Here, I integrate Granger-causality with inferences about FS. Four simulations illustrate that causal analyses can isolate distinct FS sources, (...) whereas correlational techniques cannot. I then analyze three simultaneous sequences of responses from a database of word-naming trials. I find that two, or perhaps three, distinct sources account for the presence of FS in these sequences, but FS is not a general property of the system. This suggests that FS arises due to the properties of a limited number of identifiable psychological and/or neural processes. Finally, I reanalyze a previously published dataset of acoustic frequency spectra using the new tools. The causality/criticality combination introduced here offers a new important perspective in the study of cognition. (shrink)

Bayesian nets are widely used in artificial intelligence as a calculus for causal reasoning, enabling machines to make predictions, perform diagnoses, take decisions and even to discover causal relationships. This book, aimed at researchers and graduate students in computer science, mathematics and philosophy, brings together two important research topics: how to automate reasoning in artificial intelligence, and the nature of causality and probability in philosophy.

In the philosophical tradition, the notions of determinism and causality are strongly linked: it is assumed that in a world of deterministic laws, causality may be said to reign supreme; and in any world where the causality is strong enough, determinism must hold. I will show that these alleged linkages are based on mistakes, and in fact get things almost completely wrong. In a deterministic world that is anything like ours, there is no room for genuine causation. (...) Though there may be stable enough macro-level regularities to serve the purposes of human agents, the sense of “causality” that can be maintained is one that will at best satisfy Humeans and pragmatists, not causal fundamentalists. (shrink)

The idea that causation can be reduced to transmission of an amount of some conserved quantity between events is spelled out and defended against important objections. Transmission is understood as a symmetrical relation of copresence in two distinct events. The actual asymmetry of causality has its origin in the asymmetrical character of certain irreversible physical processes and then spreads through the causal net. This conception is compatible with the possibility of backwards causation and with a causal theory of time. (...) Genidentity, the persistence of concrete objects, can be given an explanation in causal terms. The transmission theory is shown to escape difficulties faced by two important alternative theories of causation: Salmon's (1984) Mark Transmission Theory and Dowe's (1992a) Conserved Quantities Theory. (shrink)

Causality violations are typically seen as unrealistic and undesirable features of a physical model. The following points out three reasons why causality violations, which Bonnor and Steadman identified even in solutions to the Einstein equation referring to ordinary laboratory situations, are not necessarily undesirable. First, a space-time in which every causal curve can be extended into a closed causal curve is singularity free—a necessary property of a globally applicable physical theory. Second, a causality-violating space-time exhibits a nontrivial (...) topology—no closed timelike curve (CTC) can be homotopic among CTCs to a point, or that point would not be causally well behaved—and nontrivial topology has been explored as a model of particles. Finally, if every causal curve in a given space-time passes through an event horizon, a property which can be called “causal censorship”, then that space-time with event horizons excised would still be causally well behaved. (shrink)

Ernst Mayr’s concept of dual causality in biology with the two forms of causes continues to provide an essential foundation for the philosophy of biology. They are equivalent to functional and evolutionary causes with both required for full biological explanations. The natural sciences can be classified into nomological, historical nomological and historical dual causality, the last including only biology. Because evolutionary causality is unique to biology and must be included for all complete biological explanations, biology is autonomous (...) from the physical sciences. (shrink)

D. M. Armstrong is an eminent Australian philosopher whose work over many years has dealt with such subjects as: the nature of possibility, concepts of the particular and the general, causes and laws of nature, and the nature of human consciousness. This collection of essays explores the many facets of Armstrong's work, concentrating on his more recent interests. There are four sections to the book: possibility and identity, universals, laws and causality, and philosophy of mind. The contributors comprise an (...) international group of philosophers from the United States, England and Australia. An interesting feature of the volume is that Armstrong himself has written responses to each of the essays. There is also a complete bibliography of Armstrong's writings. (shrink)

A neuroscience-based approach has recently been proposed for the relation between the mind and the brain. The proposal is that events at the sub-neuronal, neuronal, and neuronal network levels take place simultaneously to perform a computation that can be described at a high level as a mental state, with content about the world. It is argued that as the processes at the different levels of explanation take place at the same time, they are linked by a non-causal supervenient relationship: (...) class='Hi'>causality can best be described in brains as operating within but not between levels. This mind-brain theory allows mental events to be different in kind from the mechanistic events that underlie them; but does not lead one to argue that mental events cause brain events, or vice versa: they are different levels of explanation of the operation of the computational system. Here, some implications are developed. It is proposed that causality, at least as it applies to the brain, should satisfy three conditions. First, interventionist tests for causality must be satisfied. Second, the causally related events should be at the same level of explanation. Third, a temporal order condition must be satisfied, with a suitable time scale in the order of 10 ms. Next, although it may be useful for different purposes to describe causality involving the mind and brain at the mental level, or at the brain level, it is argued that the brain level may sometimes be more accurate, for sometimes causal accounts at the mental level may arise from confabulation by the mentalee, whereas understanding exactly what computations have occurred in the brain that result in a choice or action will provide the correct causal account for why a choice or action was made. Next, it is argued that possible cases of “downward causation” can be accounted for by a within-levels-of-explanation account of causality. This computational neuroscience approach provides an opportunity to proceed beyond Cartesian dualism and physical reductionism in considering the relations between the mind and the brain. (shrink)

Given the importance of the principle of causality for the demonstration of God’s existence, this paper attempts to justify the evidence and necessity of the principle of causality, by following Fr. Fabro’s Thomistic defense—based on the notion of participation—but adding a particular emphasis on the notion of “being which is not per se,” this latter as an explanatory notion of the notion of “being which is by participation.” The introductory remarks touch upon two misunderstandings regarding the notion of (...) participation employed as a subject of the principle of causality: the first, considering “participated being” as a synonym of “received being,” and the second, the confusion, regarding the notion of being by participation, between its notional dependence on the notion of being as a whole and its real dependence on the real intensive Being. Then, in the first point, the defense of the principle of causality is distinguished from other related problems, like the particular applications of this principle. In the second and main point, we try to justify the necessity of being caused in the being which is experienced as being by participation, as follows: 1) that which is something by participation is not this something per se; 2) now, that which is not per se, either is not or is per aliud, 3) Therefore, that which is something by participation is per aliud. In the third and last point, we endeavor to justify the Thomism of our defense and of our emphasis on this notion of “being which is not per se” by considering two texts of St. Thomas. (shrink)

This paper surveys the issue of relativistic causality within the framework of algebraic quantum field theory . In doing so, we distinguish various notions of causality formulated in the literature and study their relationships, and thereby we offer what we hope to be a useful taxonomy. We propose that the most direct expression of relativistic causality in AQFT is captured not by the spectrum condition but rather by the axiom of local primitive causality, in that it (...) entails a form of local determinism for quantum fields which generalizes the constraint of no superluminal propagation of classical field theories to relativistic quantum field theory. We discuss the status of the axiom of micro-causality by locating its place within a large family of separability/independence/locality conditions developed for AQFT and also by relating it to so-called no-signalling theorems. And we also provide a critical survey of attempts to understand the implications for relativistic causality of the distant correlations endemic to the states in models of AQFT satisfying the standard axioms, and we provide an assessment of attempts to employ Reichenbach's common cause principle in AQFT to defuse worries that these distant correlations implicate direct causal connections between relatively spacelike events. (shrink)

This paper examines Dharmakīrti's arguments against Cārvāka physicalism in the Pramāṇasiddhi chapter of his magnum opus, the Pramāṇavārttika, with a focus on classical Indian philosophical attempts to address the mind-body problem. The key issue concerns the relation between cognition and the body, and the role this relation plays in causal-explanatory accounts of consciousness and cognition. Drawing on contemporary debates in philosophy of mind about embodiment and the significance of borderline states of consciousness, the paper proposes a philosophical reconstruction that builds (...) on two important features of the Buddhist account: an expanded conception of causality and a robust account of phenomenal content. (shrink)

D. M. Armstrong is an eminent Australian philosopher whose work over many years has dealt with such subjects as: the nature of possibility, concepts of the particular and the general, causes and laws of nature, and the nature of human consciousness. This collection of essays explores the many facets of Armstrong's work, concentrating on his more recent interests. There are four sections to the book: possibility and identity, universals, laws and causality, and philosophy of mind. The contributors comprise an (...) international group of philosophers from the United States, England and Australia. An interesting feature of the volume is that Armstrong himself has written responses to each of the essays. There is also a complete bibliography of Armstrong's writings. (shrink)

In his “Bridging mainstream and formal ontology”, Augusto (2021) gives an excellent analysis of Dietrich von Freiberg’s idea of using causality as a partitioning principle for upper ontologies. For this Dietrich’s notion of extrinsic principles is crucial. The question whether causation can and indeed should be used as a partitioning principle for ontologies is discussed using mathematics and physics as examples.

This paper discusses Simpson's paradox and the problem of positive relevance in probabilistic causality. It is argued that Cartwright's solution to Simpson's paradox fails because it ignores one crucial form of the paradox. After clarifying different forms of the paradox, it is shown that any adequate solution to the paradox must allow a cause to be both a negative cause and a positive cause of..