The prevention, treatment and management of disease are closely linked to how the causes of a particular disease are explained. For multi-factorial conditions, the causal explanations are inevitably complex and competing models may exist to explain the same condition. Selecting one particular causal explanation over another will carry practical and ethical consequences that are acutely relevant for health policy. In this paper our focus is two-fold; the different models of causal explanation that are put forward within current scientific literature for (...) the high and rising prevalence of the common complex conditions of coronary artery disease and type 2 diabetes mellitus ; and how these explanations are taken up within national health policy guidelines. We examine the causal explanations for these two conditions through a systematic database search of current scientific literature. By identifying different causal explanations we propose a three-tier taxonomy of the most prominent models of explanations: evolutionary, lifecourse, and lifestyle and environment. We elaborate this taxonomy with a micro-level thematic analysis to illustrate how some explanations are semantically and rhetorically foregrounded over others. We then investigate the uptake of the scientific causal explanations in health policy documents with regard to the prevention and management recommendations of current National Service Frameworks for CAD and T2D. Our findings indicate a lack of congruence between the complexity and frequent overlap of causal explanations evident in the scientific literature and the predominant focus on lifestyle recommendations found in the mainstream health policy documents. (shrink)

Fundamental Causation addresses issues in the metaphysics of deterministic singular causation, the metaphysics of events, property instances, facts, preventions, and omissions, as well as the debate between causal reductionists and causal anti-reductionists. The book also pays special attention to causation and causal structure in physics. Weaver argues that causation is a multigrade obtaining relation that is transitive, irreflexive, and asymmetric. When causation is singular, deterministic and such that it relates purely contingent events, the relation is (...) also universal, intrinsic, and well-founded. He shows that proper causal relata are events understood as states of substances at ontological indices. He then proves that causation cannot be reduced to some non-causal base, and that the best account of that relation should be unashamedly primitivist about the dependence relation that underwrites its very nature. The book demonstrates a distinctive realist and anti-reductionist account of causation by detailing precisely how the account outperforms reductionist and competing anti-reductionist accounts in that it handles all of the difficult cases while overcoming all of the general objections to anti-reductionism upon which other anti-reductionist accounts falter. This book offers an original and interesting view of causation and will appeal to scholars and advanced students in the areas of metaphysics, philosophy of science, and philosophy of physics. (shrink)

This paper explores and defends the idea that mental properties and their physical bases jointly cause their physical effects. The paper evaluates the view as an emergentist response to the exclusion problem, comparing it with a competing nonreductive physicalist solution, the compatibilist solution, and argues that the joint causation view is more defensible than commonly supposed. Specifically, the paper distinguishes two theses of closure, Strong Closure and Weak Closure, two causal exclusion problems, the overdetermination problem and the supervenience problem, (...) and argues that emergentists can avoid the overdetermination problem by denying Strong Closure and respond to the supervenience problem by accepting the joint causation view. (shrink)

There is ample evidence that violations of injunctive norms impact ordinary causal attributions. This has struck some as deeply surprising, taking the ordinary concept of causation to be purely descriptive. Our explanation of the findings—the responsibility view—rejects this: we contend that the concept is in fact partly normative, being akin to concepts like responsibility and accountability. Based on this account, we predicted a very different pattern of results for causal attributions when an agent violates a statistical norm. And this (...) pattern has been borne out by the data. These predictions were based on the responsibility attributions that we would make. In this paper, I extend these previous findings, testing responsibility attributions. The results confirm the basis of our predictions, showing the same pattern of effects previously found for causal attributions for both injunctive norms and statistical norms. In fact, the results for responsibility attributions are not statistically significantly different from those previously found for causal attributions. I argue that this close correspondence lends further credence to the responsibility view over competing explanations of the impact of norms on causal attributions. (shrink)

Extant research has shown that ordinary causal judgments are sensitive to normative factors. For instance, agents who violate a norm are standardly deemed more causal than norm-conforming agents in identical situations. In this paper, we explore two competing explanations for the Norm Effect: the Responsibility View and the Bias View. According to the former, the Norm Effect arises because ordinary causal judgment is intimately intertwined with moral responsibility. According to the alternative view, the Norm Effect is the result of a (...) blame-driven bias. In a series of five preregistered experiments (N = 2688), we present evidence that predominantly favours the Bias View. In particular, and against predictions made by the Responsibility View, we show that participants deem agents who violate nonpertinent or silly norms – norms that do not relate to the outcome at hand – as more causal, and that this effect cannot be explained in terms of plausible mediators such as the agent’s foreknowledge and desire, or the foreseeability of harm. We close with a discussion regarding the implications of these findings, in as regards the just assessment of proximate cause in the law. (shrink)

In taking properties to have powerful or dispositional essences, dispositionalism is primed to provide an account of causation. This paper lays out a challenge confronting the dispositionalist’s ability to account for how powers causally interact with one another so as to bring about collective results. The challenge, here labeled the “interaction gap,” is raised for two competing kinds of approaches to dispositional interaction: contribution combinationist and mutual manifestationist. After carefully highlighting and testing potential resources for closing the interaction gap, (...) it is concluded that the mutual manifestationist approach holds a significant advantage. In turn, the importance of the interaction gap itself is highlighted. While powers prime an ontology to yield an account of causation, how far that account can actually go depends on the metaphysical details of one’s view of powers and their causal interaction. (shrink)

A growing body of literature has revealed ordinary causal judgement to be sensitive to normative factors, such that a norm-violating agent is regarded more causal than their non-norm-violating counterpart. In this paper, we explore two competing explanations for this phenomenon: the Responsibility View and the Bias View. The Bias View, but not the Responsibility View, predicts features peripheral to the agent’s responsibility to impact causal attributions. In a series of three preregistered experiments (N = 1162), we present new evidence that (...) the Norm Effect arises from such peripheral features, namely from nonpertinent or entirely silly norm violations. Furthermore, we show that this effect cannot be explained by recourse to the agent’s foreknowledge or desire of the outcome, nor by its foreseeability: the Norm Effect arises even when participants judge the norm-violating agent’s doing as equally foreseeable. This, we argue, provides evidence in favour of the Bias View. (shrink)

In taking properties to have powerful or dispositional essences, dispositionalism is primed to provide an account of causation. This paper lays out a challenge confronting the dispositionalist’s ability to account for how powers causally interact with one another so as to bring about collective results. The challenge, here labeled the “interaction gap,” is raised for two competing kinds of approaches to dispositional interaction: contribution combinationist and mutual manifestationist. After carefully highlighting and testing potential resources for closing the interaction gap, (...) it is concluded that the mutual manifestationist approach holds a significant advantage. In turn, the importance of the interaction gap itself is highlighted. While powers prime an ontology to yield an account of causation, how far that account can actually go depends on the metaphysical details of one’s view of powers and their causal interaction. (shrink)

How can mental properties bring about physical effects, as they seem to do, given that the physical realizers of the mental goings-on are already sufficient to cause these effects? This question gives rise to the problem of mental causation (MC) and its associated threats of causal overdetermination, mental causal exclusion, and mental causal irrelevance. Some (e.g., Cynthia and Graham Macdonald, and Stephen Yablo) have suggested that understanding mental-physical realization in terms of the determinable/determinate relation (henceforth, 'determination') provides the key (...) to solving the problem of MC: if mental properties are determinables of their physical realizers, then (since determinables and determinates are distinct, yet don't causally compete) all three threats may be avoided. Not everyone agrees that determination can do this good work, however. Some (e.g., Douglas Ehring, Eric Funkhauser, and Sven Walter) object that mental-physical realization can't be determination, since such realization lacks one or other characteristic feature of determination. I argue that on a proper understanding of the features of determination key to solving the problem of MC these arguments can be resisted. (shrink)

Yablo suggests that we can understand the possibility of mental causation by supposing that mental properties determine physical properties, in the classic sense of determination according to which red determines scarlet. Determinates and their determinables do not compete for causal relevance, so if mental and physical properties are related as determinable and determinates, they should not compete for causal relevance either. I argue that this solution won''t work. I first construct a more adequate account of determination than that provided (...) by Yablo. I then consider two common accounts of the mental, token identity theories and dispositional theories, and argue that on neither do mental and physical properties satisfy the requirements for determination. (shrink)

Arguments for an extended evolutionary synthesis often center on the concept of “reciprocal causation.” Proponents argue that reciprocal causation is superior to standard models of evolutionary causation for at least two reasons. First, it leads to better scientific models with more predictive power. Second, it more accurately represents the causal structure of the biological world. Simply put, proponents of an extended evolutionary synthesis argue that reciprocal causation is empirically and explanatorily apt relative to competing causal frameworks. (...) In this paper, I present quantitative survey data from faculty members in biology departments at universities across the United States to evaluate this claim. The survey data indicate that a majority of the participants do not agree (i.e., most either disagree or neither agree nor disagree) that the concept of reciprocal causation confers a larger advantage on research practices. However, a majority of the participants agree that the causal framework of the extended evolutionary synthesis more accurately represents the structure of the biological world. These results demonstrate that the explanatory merits of a conceptual framework and its practical utility can come apart in interesting and informative ways. (shrink)

In this paper I do three things. First, I argue that Stephen Yablo’s influential account of mental causation is susceptible to counterexamples involving what I call disproportional mental causation. Second, I argue that similar counterexamples can be generated for any alternative account of mental causation that is like Yablo’s in that it takes mental states and their physical realizers to causally compete. Third, I show that there are alternative nonreductive approaches to mental causation which reject the (...) idea of causal competition, and which thus are able to allow for disproportional mental causation. This, I argue, is a significant advantage for such noncompetitive accounts. (shrink)

I argue that high level causal relationships are often more fundamental than low level causal relationships. My argument is based on some general principles governing when one causal relationship will metaphysically ground another—a phenomenon I term derivative causation. These principles are in turn based partly on our intuitive judgments concerning derivative causation in a series of representative examples, and partly on some powerful theoretical considerations in their favour. I show how these principles entail that low level causation (...) can derive from high level causation, and in particular that neural causation can derive from mental causation. I then draw out several important consequences of this result. Most immediate among these are the implications the result has for aspirations to reduce high level causation to its low level counterpart. But the result also bears on the possibility of downward causation, the relationship between counterfactuals and causation, and the idea—familiar from both the literature on the exclusion problem and the literature on proportionality constraints on causation—that causal relationships at different levels compete for their existence. (shrink)

Leibniz argued that in natural world, only intra-substantial or immanent causation is possible— the causation that takes place within an individual, when an individual brings about a change in itself. In this dissertation, I address issues arising from Leibniz’s arguments against the rival view that posits a world of causally interacting substances and issues pertaining to Leibniz’s own positive metaphysics of immanent causation and change. -/- Chapter 1 is devoted to stage setting for the remainder of the (...) dissertation. I first offer a historically informed overview of efficient causation and change before introducing Leibniz’s novel views, including his criticisms of competing accounts and his own positive account. After presenting a detailed roadmap of my project, I articulate the idealistic interpretation of Leibniz assumed in this dissertation, where the only genuine substances are simple monads. Finally, I articulate the methodological approaches I employ. -/- In Chapters 2 and 3, I reconstruct and assess Leibniz’s most frequent argument against transeunt causation (the causation that occurs when a substance produces a property in a numerically distinct substance), what I call the “Transference Argument.” Leibniz argued that a created substance’s causing an accident in a numerically distinct substance is possible only if the agent transfers the accident from itself to the patient, where upon transference the agent no longer possesses the accident it transferred. Call the transeunt causal requirement that the agent transfer the accident produced from itself to the patient the “Transference Condition.” -/- Chapter 2 is devoted to two problems with Leibniz’s transference condition. First, Leibniz stated the transference condition throughout his career, but offered little argument for it. Second, God is a transeunt cause in Leibniz’s metaphysics yet God’s causation does not consist in transference. Thus, Leibniz needs a principled way to require transference for creaturely causation while denying that divine transeunt causation consists in transference. Finally, I close off chapter 2 by drawing attention to an important weakness with the transference condition that has not yet been recognized by Leibniz scholars. -/- In Chapter 3, I argue that there is nothing in Leibniz’s ontology that could be transferred from the cause to the recipient of the effect. I first argue that Leibniz’s ontology consists of simple non-corporeal substances and their modifications. Second, I present and articulate a number of important theses Leibniz affirmed about substances and their modifications, which entail that neither could be transferred. I also show that most of these theses were not unique to Leibniz, but were in fact widely endorsed by his predecessors who defended the possibility of creaturely transeunt causation. -/- In chapter 4, I continue the study of the nature of Leibnizian accidents, shifting the focus from their role in Leibniz’s critique of creaturely transeunt causation to their positive role in change and as causal relata, where such accidents are the effects of immanent causation. I shall argue that Leibniz’s thesis that accidents are modifications or limitations allowed him to posit mereologically simple substance that have a multitude of accidents at a time and change accidents over time. -/- In Chapter 5, I address an issue that has divided Leibniz scholars concerning the precise relata in Leibnizian immanent efficient causation. In many passages, Leibniz writes as if it is the substance or individual itself that efficiently causes its later properties or accidents. Call this the “Efficacious-substance” account. The efficacious-substance account is difficult to reconcile with Leibniz’s requirements that change be intelligible and deterministic. In plenty of other passages, he writes as if it is the substances earlier properties or accidents that cause its later accidents. Call this the “Efficacious-accident” account. The efficacious-accident account explains how change is intelligible and deterministic but it faces a “plurality of agents” objection. If a substance’s accidents are the efficient causes of later accidents, then prima facie there is a plurality of efficient causal agents in a substance. This view is incompatible with Leibniz’s requirement that substances be simple, unified entities. -/- Drawing upon a Scholastic distinction made between two kinds of efficient causes— principle quod efficient causes and principle quo efficient causes, I shall argue that for Leibniz, substances are principle quod efficient causes and their appetitions are principle quo efficient causes. This interpretation combines the strengths of the Efficacious-substance and Efficacious-accident accounts while overcoming their weaknesses. There is just one causal agent, the substance, but change is both intelligible and deterministic because as what an agent produces is explained by its appetitions. (shrink)

In this thesis, I give a metascientific account of causality in medicine. I begin with two historical cases of causal discovery. These are the discovery of the causation of Burkitt’s lymphoma by the Epstein-Barr virus, and of the various viral causes suggested for cervical cancer. These historical cases then support a philosophical discussion of causality in medicine. This begins with an introduction to the Russo- Williamson thesis (RWT), and discussion of a range of counter-arguments against it. Despite these, I (...) argue that the RWT is historically workable, given a small number of modifications. I then expand Russo and Williamson’s account. I first develop their suggestion that causal relationships in medicine require some kind of evidence of mechanism. I begin with a number of accounts of mechanisms and produce a range of consensus features of them. I then develop this consensus position by reference to the two historical case studies with an eye to their operational competence. In particular, I suggest that it is mechanistic models and their representations which we are concerned with in medicine, rather than the mechanism as it exists in the world. -/- I then employ these mechanistic models to give an account of the sorts of evidence used in formulating and evaluating causal claims. Again, I use the two human viral oncogenesis cases to give this account. I characterise and distinguish evidence of mechanism from evidence of difference-making, and relate this to mechanistic models. I then suggest the relationship between types of evidence presents us with a means of tackling the reference-class problem. This sets the scene for the final chapter. Here, I suggest the manner in which these two different classes of evidence become integrated is also reflected in the way that developing research programmes change as their associated causal claims develop. (shrink)

Nonreductive physicalism provides an appealing solution to the nature of mental properties. But its success as a theory of mental properties has been called into doubt by claims that it cannot adequately handle the problems of mental causation, as it leads either to epiphenomenalism or to thoroughgoing overdetermination. I argue that these apparent problems for the nonreductivist are based in fundamental confusion about causation and explanation. I distinguish two different types of explanation and two different relations to which (...) they appeal: causation and determination. I argue that these types of explanation do not compete with one another, nor do these relations jointly result in overdetermination. In closing I develop a nonreductivist solution to mental causation which avoids both the hazards of epiphenomenalism and of overdetermination and so demonstrates a way to save nonreductive physicalism from the problems of mental causation. (shrink)

Gold & Stoljar's argument rejecting the “explanatory sufficiency” of the radical neuron doctrine depends on distinguishing it from the trivial neuron doctrine. This distinction depends on the thesis of “supervenience,” which depends on Hume's regularity theory of causation. In contrast, the radical neuron doctrine depends on a physical theory of causation, which denies the supervenience thesis. Insofar as the target article argues by drawing implications from the premise of Humean causation, whereas the radical doctrine depends on the (...) competing premise of physical causation, the resulting critique of the neuron doctrine amounts largely to begging the question of causation. (shrink)

Social systems theory has been dominated in recent years by the work of Niklas Luhmann, but there is another strand of systems thinking, which is receiving increasing attention in sociology: emergentism. For emergentism, the core problems of systems thinking are concerned with causation and reductionism; for Luhmann, they are questions of meaning and self-reference. Arguing from an emergentist perspective, the article finds that emergentism addresses its own core problem successfully, while Luhmann's approach is incapable of resolving questions of (...) class='Hi'>causation and reductionism. On the other hand, neither paradigm yet has a convincing response to the challenges of meaning and self-reference. Key Words: social systems • emergence • paradigms • autopoiesis • critical realism. (shrink)

We often face a bewildering array of different explanations for the same social facts (e.g. biological, psychological, economic, and historical accounts). But we have few guidelines for whether and when we should think of different accounts as competing or compatible. In this paper, I offer some guidelines for understanding when custom or norm accounts do and don’t compete with other types of accounts. I describe two families of non-competing accounts: (1) explanations of different (but similarly described) facts, and (2) accounts (...) which seem to differ but are really different parts or versions of the same underlying explanation. I argue that, while many types of apparent competitors don’t really compete with customs, there are some that do. I also describe some of the central problems, which suggest that custom accounts will compete poorly with their rivals. (shrink)

This paper pulls together three debates fundamental in metaphysics and proposes a novel unified approach to them. The three debates are (i) between bundle theory and substrate theory about the nature of objects, (ii) dispositionalism and categoricalism about the nature of properties, and (iii) regularity theory and production theory about the nature of causation. The first part of the paper (§§2-4) suggests that although these debates are metaphysical, the considerations motivating the competing approaches in each debate tend to be (...) epistemological. The second part (§§5-6) argues that the two underlying epistemological pictures supporting competing views lead to highly unsatisfying conceptions of the world. The final part (§§7-10) proposes an alternative epistemological picture, which I call ‘introverted empiricism,’ and presents the way it provides for a more satisfying grasp of the ultimate nature of objects, properties, and causation. It is a consequence of this alternative picture that there is a kind of intimate self-understanding that underlies our understanding of the deep nature of reality. (shrink)

The term "social science" promises more than its practitioners can deliver: it promises knowledge. This knowledge is to consist of statements of empirical regularities of such quality as will enhance predictive power and inform public and private policy. Boundaries of Competence illuminates obstacles to this aspiration. Chapter 1 grounds knowledge in perception. Chapter 2 challenges the assumption that ordinary language necessarily describes reality and reveals the mischief words can do. Chapter 3 proposes a continuum of perceiving-conceiving involved in different ways (...) of "knowing" worlds. Chapter 4 lays out requirements of measurement, arguing that assigning numbers to dubious observations gives false assurance that mathematical manipulations necessarily rep- resent events. Chapter 5 shows how choice of unit affects the correlations we find in our search for causes. Chapter 6 holds that, given deficiencies in knowledge and perhaps because of the way Nature works, we assess probabilities rather than seek certainties. Chapter 7 notes difficulties in counting events that consist of "social facts," which "depend on us," and those that refer to "brute facts," that exist independently of us. Chapter 8 criticizes the practice of employing proxies for observations of what we're talking about. In particular, it demonstrates the error produced by relying on what people say as measure of what they do. Chapter 9 criticizes ways of explaining conduct by characterizing actors and their acts, and by "understanding" them through empathy. Chapter 10 discusses the important and never-ending quarrel about causation. Chapters 11 and 12 argue that in social affairs, decision is regularly torn between doing what is effective (rational) and doing what is right (moral). Nettler's writing is crisp, and his argument balanced. He employs research from several disciplines to challenge ideologically driven descriptions of our condition and explanations of our conduct. Gwynne Nettler is professor emeritus at the University of Alberta in Edmonton, Canada, where he taught psychology, sociology, and criminology. His previous books include Explanations, Social Concerns, and Explaining Crime. (shrink)

Abstract: Situations that social scientists and others explain by using concepts like "custom" and "norm" often tend to be situations in which many other kinds of explanations (for example, biological, psychological, economic, historical) seem plausible as well. Do these other explanations compete with the custom or norm explanations, or do they complement them? We need to consider this question carefully and not just assume that various accounts are all permissible at different levels of analysis. In this article I describe two (...) families of noncompeting accounts: (1) explanations of different (but similarly described) facts, and (2) accounts that seem to differ but are really different parts or versions of the same underlying explanation. I argue that while many types of apparent competitors don't really compete with norms, there are usually some that do. These competing accounts will usually undermine the norm account. (shrink)

Nonreductive physicalism provides an appealing solution to the nature of mental properties. But its success as a theory of mental properties has been called into doubt by claims that it cannot adequately handle the problems of mental causation, as it leads either to epiphenomenalism or to thoroughgoing overdetermination. I argue that these apparent problems for the nonreductivist are based in fundamental confusion about causation and explanation. I distinguish two different types of explanation and two different relations to which (...) they appeal: causation and determination. I argue that these types of explanation do not compete with one another, nor do these relations jointly result in overdetermination. In closing I develop a nonreductivist solution to mental causation which avoids both the hazards of epiphenomenalism and of overdetermination and so demonstrates a way to save nonreductive physicalism from the problems of mental causation. (shrink)

In a recent article, “Wayward Modeling: Population Genetics and Natural Selection,” Bruce Glymour claims that population genetics is burdened by serious predictive and explanatory inadequacies and that the theory itself is to blame. Because Glymour overlooks a variety of formal modeling techniques in population genetics, his arguments do not quite undermine a major scientific theory. However, his arguments are extremely valuable as they provide definitive proof that those who would deploy classical population genetics over natural systems must do so with (...) careful attention to interactions between individual population members and environmental causes. Glymour’s arguments have deep implications for causation in classical population genetics. (shrink)

Stephen Yablo offers a solution to the problem of mental causation by claiming that the physical is a determinate of the mental's determinable, and therefore the mental and physical do not compete for causal relevance. I present Yablo's solution and argue that the mental‐physical relation cannot meet three necessary conditions for determination. That relation fails to meet the requirements that determinates of the same determinable be incompatible and that no property can be a determinate of more than one determinable. (...) Further, the mental and physical do not fall under the same series of scope inclusions as determinables and their determinates must. Yablo's conception of determination might be rendered more general to avoid these objections, but doing so would prevent it from functioning as a competitor to extant theories of the mental‐physical relation. Thus, Yablo's solution to the problem of mental causation cannot be both adequately specific and correct. (shrink)

Stephen Yablo offers a solution to the problem of mental causation by claiming that the physical is a determinate of the mental's determinable, and therefore the mental and physical do not compete for causal relevance. I present Yablo's solution and argue that the mental‐physical relation cannot meet three necessary conditions for determination. That relation fails to meet the requirements that determinates of the same determinable be incompatible and that no property can be a determinate of more than one determinable. (...) Further, the mental and physical do not fall under the same series of scope inclusions as determinables and their determinates must. Yablo's conception of determination might be rendered more general to avoid these objections, but doing so would prevent it from functioning as a competitor to extant theories of the mental‐physical relation. Thus, Yablo's solution to the problem of mental causation cannot be both adequately specific and correct. (shrink)

Knobe cites both relevant alternatives and defaults on a continuum to explain how moral judgments influence intuitions about certain apparently non-moral notions. I ask (1) how these two accounts are related, (2) whether they exclude or supplement supposedly competing theories, and (3) how to get positive evidence that people consider relevant alternatives when applying such notions.

Underdevelopment is the most controversial issue of our time. In a world which apparently exhibits so much power and yet does so little to drive it back, it represents the challenge par excellence. However, concerning this most pressing and controversial issue of underdevelopment, of all the disciplines which study man, philosophy is the one which until now said the least. At first sight, to mark off in the topic of underdevelopment an area of real philosophical concern does not seem feasible (...) indeed. Underdevelopment understood as a mere failure of development appears to be within the competence of the disciplines of the social sciences, especially of economics, rather than that of philosophy. Viewed as a technical problem, it could thereby be declared outside the sphere of direct philosophical inquiry.But this reason is no sooner accepted than it comes up against itself by the very nature of the conflicts in which the social sciences got bogged down in their attempt to understand underdevelopment. Although not directly spelt out, the conflicts are unmistakably pervaded throughout by philosophical questions. It suffices to point out the main cause of the theoretical split which divides scholars, in the general issue of tradition and modernity, to really bring out the eminently philosophical nature of the debate. We are obviously referring to the conflict between modernization theory and the neo-Marxist school. The purpose of this paper is precisely to show that the topic of underdevelopment, not only raises one of the basic and oldest problems of philosophy, namely the nature of the connection between the spiritual and the material, but also helps positively to reformulate it. (shrink)

As you scroll through this review, you move your hand; this causes the mouse to move; in turn this causes, via a series of intermediary events, changes on your screen. A bit more reflection shows that this case is entirely mundane: causal relations are a ubiquitous feature of the physical world. Causal relations are also, according to many philosophers, at the center of phenomena like knowledge, perception, linguistic meaning, mental content, belief, free action, and right action. In fact, one is (...) hard put to think of an important philosophical notion that has not received a causal analysis, especially in recent analytic philosophy. Consider a few from the theory of knowledge. According to the causal theory of knowledge, knowledge is true belief caused by what makes the belief true. Or, according to a competing view, knowledge is true belief caused by a reliable belief forming process, where a reliable process is one that causes a high ratio of true to false beliefs. According to the causal theory of perception, seeing that the cup is on the table consists in being in a perceptual state that is appropriately caused by the cup and the table. Causation, it seems, is absolutely central. We will need to understand causation itself if we are to understand either causal theories in philosophy or the nature of the surrounding world. (shrink)

Ethics consultation has become an integral part of the fabric of U.S. health care delivery. This article summarizes the second edition of the Core Competencies for Health Care Ethics Consultation report of the American Society for Bioethics and Humanities. The core knowledge and skills competencies identified in the first edition of Core Competencies have been adopted by various ethics consultation services and education programs, providing evidence of their endorsement as health care ethics consultation (HCEC) standards. This revised report was prompted (...) by thinking in the field that has evolved since the original report. Patients, family members, and health care providers who encounter ethical questions or concerns that ethics consultants could help address deserve access to efficient, effective, and accountable HCEC services. All individuals providing such services should be held to the standards of competence and quality described in the revised report. (shrink)

Why is understanding causation so important in philosophy and the sciences? Should causation be defined in terms of probability? Whilst causation plays a major role in theories and concepts of medicine, little attempt has been made to connect causation and probability with medicine itself. Causality, Probability, and Medicine is one of the first books to apply philosophical reasoning about causality to important topics and debates in medicine. Donald Gillies provides a thorough introduction to and assessment of (...) competing theories of causality in philosophy, including action-related theories, causality and mechanisms, and causality and probability. Throughout the book he applies them to important discoveries and theories within medicine, such as germ theory; tuberculosis and cholera; smoking and heart disease; the first ever randomized controlled trial designed to test the treatment of tuberculosis; the growing area of philosophy of evidence-based medicine; and philosophy of epidemiology. This book will be of great interest to students and researchers in philosophy of science and philosophy of medicine, as well as those working in medicine, nursing and related health disciplines where a working knowledge of causality and probability is required. (shrink)

The Exclusion Argument has afflicted non-reductionists for decades. In this article, I attempt to show that emergentism—the view that mental entities can downwardly cause physical entities in a non-overdetermining way—is the most plausible approach to solving the exclusion problem. The emergentist approach is largely absent in contemporary philosophy of mind, because emergentism rejects the Causal Closure of Physics, a doctrine embraced by almost all physicalists. This article, however, challenges the consensus on causal closure and defends a physicalist version of emergentism. (...) On closer examination, I find that neither physical evidence nor physicalist considerations can satisfactorily support causal closure. Moreover, I argue that two competing approaches that accept causal closure, compatibilism and autonomism, involve ad hoc postulations of mental causation. Therefore, all things considered, emergentism is the best option available to non-reductive physicalists. (shrink)

We engage with the nature and the value of achievement through a critical examination of an argument according to which biomedical “enhancement” of our capacities is impermissible because enhancing ourselves in this way would threaten our achievements. We call this the argument against enhancement from achievement. We assess three versions of it, each admitting to a strong or a weak reading. We argue that strong readings fail, and that weak readings, while in some cases successful in showing that enhancement interferes (...) with the nature or value of achievement, fail to establish that enhancement poses an unusual threat to achievement. (shrink)

Causal Dispositionalism provides an account of causation based on an ontology of causal powers, properties with causal essence. According to the account, causation can be analysed in terms of the interaction of powers and its subsequent production of their effect. Recently, Baltimore, J. A. has raised a challenge against two competing approaches, the compositional view and the mutual manifestation view, to explain what makes powers interactive – the interaction gap. In this paper, we raise the challenge of explaining (...) what makes powers productive – the production gap. While Baltimore’s verdict is tentatively favouring, we find both approaches wanting. Our conclusion is that Causal Dispositionalists should take Baltimore’s and our critique seriously. Powers cannot cause their effects just by bearing the name “causal”. To deserve their names, more metaphysical details are needed. (shrink)

Denis Walsh has written a striking new defense in this journal of the statisticalist (i.e., noncausalist) position regarding the forces of evolution. I defend the causalist view against his new objections. I argue that the heart of the issue lies in the nature of nonadditive causation. Detailed consideration of that turns out to defuse Walsh’s ‘description‐dependence’ critique of causalism. Nevertheless, the critique does suggest a basis for reconciliation between the two competing views. *Received December 2009; revised December 2009. †To (...) contact the author, please write to: Department of Philosophy, 599 Lucas Hall, One University Boulevard, University of Missouri, St. Louis, MO 63121; e‐mail: [email protected]. (shrink)

It is natural to think of causes as difference-makers. What exact difference causes make, however, is an open question. In this paper, I argue that the right way of understanding difference-making is in terms of causal processes: causes make a difference to a causal process that leads to the effect. I will show that this way of understanding difference-making nicely captures the distinction between causing an outcome and helping determine how the outcome happens and, thus, explains why causation is (...) not transitive. Moreover, the theory handles tricky cases that are problematic for competing accounts of difference-making. (shrink)

Newsome ((2003). The debate between current versions of covariation and mechanism approaches to causal inference. Philosophical Psychology, 16, 87-107.) recently published a critical review of psychological theories of human causal inference. In that review, he characterized covariation and mechanism theories, the two dominant theory types, as competing, and offered possible ways to integrate them. I argue that Newsome has misunderstood the theoretical landscape, and that covariation and mechanism theories do not directly conflict. Rather, they rely on distinct sets of reliable (...) indicators of causation, and focus on different types of causation (type vs. token). There are certainly debates in the research field, but the theoretical landscape is not as fractured as Newsome suggests, and a potential unifying framework has already emerged using causal Bayes nets. Philosophical work on causal epistemology matters for psychologists, but not in the way Newsome suggests. (shrink)

The interventionist theory of causation has been advertised as an empirically informed and more nuanced approach to causality than the competing theories. However, previous literature has not yet analyzed the regression discontinuity (hereafter, RD) and the difference-in-differences (hereafter, DD) within an interventionist framework. In this paper, I point out several drawbacks of using the interventionist methodology for justifying the DD and RD designs. However, I argue that the first step towards enhancing our understanding of the DD and RD designs (...) from an interventionist perspective is to take advantage of the assumptions of common trend and continuity. (shrink)