As illustrated by research Koehler himself cites (Dawes et al. 1993), the purpose of experiments is to choose between contrasting explanations of past observations – rather than to seek statistical generalizations about the prevalence of effects. True external validity results not from sampling various problems that are representative of “real world” decision making, but from reproducing an effect in the laboratory with minimal contamination (including from real world factors).

Recent studies of spider phobia have indicated thatfearof spiders is closely associated with the disease-avoidance response of disgust. It is argued that the disgust-relevant status of the spider resulted from its association with disease and illness in European cultures from the tenth century onward. The development of the association between spiders and illness appears to be linked to the many devastating and inexplicable epidemics that struck Europe from the Middle Ages onwards, when the spider was a suitable displaced target for (...) the anxieties caused by these epidemics. Such factors suggest that the pervasive fear of spiders that is commonly found in many Western societies may have cultural rather than biological origins, and may be restricted to Europeans and their descendants. (shrink)

Most phobias are focussed on a small number of fear-inducing stimuli (e.g., snakes, spiders). A review of the evidence supporting biological and cognitive explanations of this uneven distribution of phobias suggests that the readiness with which such stimuli become associated with aversive outcomes arises from biases in the processing of information about threatening stimuli rather than from phylogenetically based associative predispositions or “biological preparedness.” This cognitive bias, consisting of a heightened expectation of aversive outcomes following fear-relevant stimuli, generates and maintains (...) robust learned associations between them. Some of the features of such stimuli which determine this expectancy bias are estimates of how dangerous they are, the semiotic similarity between them and their aversive outcomes, and the degree of prior fear they elicit. Ontogenetic and cultural factors influence these features of fear-relevant stimuli and are hence important in determining expectancy bias. The available evidence does not exclude the possibility that both expectancy biases and specific evolved predispositions coexist, but the former can explain a number of important findings that the latter cannot. (shrink)

Some thoughts just come to mind together. This is usually thought to happen because they are connected by associations, which the mind follows. Such an explanation assumes that there is a particular kind of simple psychological process responsible. This view has encountered criticism recently. In response, this paper aims to characterize a general understanding of associative simplicity, which might support the distinction between associative processing and alternatives. I argue that there are two kinds of simplicity that are treated as characteristic (...) of association, and as a result three possible versions of associative processing. This provides a framework that informs our understanding of association as a current and historical concept, including how various specific versions in different parts of psychology relate to one another. This framework can also guide debates over normative evaluations of actions produced by processes thought to be associative. (shrink)

Some thoughts just come to mind together. This is usually thought to happen because they are connected by associations, which the mind follows. Such an explanation assumes that there is a particular kind of simple psychological process responsible. This view has encountered criticism recently. In response, this paper aims to characterize a general understanding of associative simplicity, which might support the distinction between associative processing and alternatives. I argue that there are two kinds of simplicity that are treated as characteristic (...) of association, and as a result three possible versions of associative processing. This provides a framework that informs our understanding of association as a current and historical concept, including how various specific versions in different parts of psychology relate to one another. This framework can also guide debates over normative evaluations of actions produced by processes thought to be associative. (shrink)

When reasoning about deontic rules (what one may, should, or should not do in a given set of circumstances), reasoners adopt a violation‐detection strategy, a strategy they do not adopt when reasoning about indicative rules (descriptions of purported state of affairs). I argue that this indicative‐deontic distinction constitutes a primitive in the cognitive architecture. To support this claim, I show that this distinction emerges early in development, is observed regardless of the cultural background of the reasoner, and can be selectively (...) disrupted at the neurological level. I also argue that this distinction emerged as a result of selective pressure favouring the evolution of reasoning strategies that determine survival within dominance hierarchies. (shrink)

Research from ethology and evolutionary biology indicates the following about the evolution of reasoning capacity. First, solving problems of social competition and cooperation have direct impact on survival rates and reproductive success. Second, the social structure that evolved from this pressure is the dominance hierarchy. Third, primates that live in large groups with complex dominance hierarchies also show greater neocortical development, and concomitantly greater cognitive capacity. These facts suggest that the necessity of reasoning effectively about dominance hierarchies left an indelible (...) mark on primate reasoning architectures, including that of humans. In order to survive in a dominance hierarchy, an individual must be capable of (a) making rank discriminations, (b) recognizing what is forbidden and what is permitted based one's rank, and (c) deciding whether to engage in or refriin from activities that will allow one to move up in rank. The first problem is closely tied to the capacity for transitive reasoning, while the second and third are intimately related to the capacity for deontic reasoning. I argue that the human capacity for these types of reasoning have evolutionary roots that reach deeper into our ancestral past than the emergence of the hominid line, and the operation of these evolutionarily primitive reasoning systems can be seen in the development of human reasoning and domain-specific effects in adult reasoning. (shrink)

Mealey's evolutionary reasoning is logically flawed. Furthermore, the evidence presented in favor of a genetic contribution to the causation of sociopathy is overinterpreted. Given the potentially large societal impact of sociobiological speculation on the roots of criminality, more-than-usual caution in interpreting data is called for.

The base rate fallacy is directly dependent on a particular judgment paradigm in which information may be unambiguously designated as either “base rate” or “individuating,” and in which subjects make two-stage sequential judgments. The paradigm may be a poor match for real world settings, and the fallacy may thus be undefined for natural ecologies of judgment.

Mealey's interesting interpretation of sociopathy is based on an inappropriate two-person game model. A multiperson, compound game version of Chicken would be more suitable, because a population engaging in random pairwise interactions with that structure would evolve to an equilibrium in which a fixed proportion of strategic choices was exploitative, antisocial, and risky, as required by Mealey's interpretation.

There is a familiar risk of antinomy if fromxisEand p(xisH/xisE) =rit is permissible to infer p(xisH) =r, and what Carnap (1950) called “The requirement of total evidence” will not prevent such antinomies satisfactorily. What is needed instead is a properly developed theory of evidential weight.

The object of this paper is to show why recent research in the psychology of deductive and probabilistic reasoning does not have.

In the Frank (1988) model, a small increase in the number of cheaters will soon be reversed. It is not clear that this prediction holds for sociopathy. There are also many attractive evolutionary models that do not admit a small, stable proportion of cheaters. Hence, without definitive evidence about the character of early human society, we cannot conclude that sociopathy has an evolutionary origin.

Definitional slippage threatens to equate secondary sociopathy with mere criminality and leaves the status of noncriminal sociopaths ambiguous. Primary sociopathy appears to show more environmental contingency than would be implied by a strong genetic trait approach. A reinterpretation in terms of hypermasculinity and hypofemininity is compatible with the data.

Koehler's work will assist the effort to understand legal fact finding. It leaves two questions somewhat open: (i) the extent to which empirical research can measure correctness of fact-finding, a function that involves the resolution of normative questions and (ii) the standards judges should use in the absence of the research he advocates.

For decades, interdisciplinary research efforts have accumulated insights that diminish the significance of the classic nature versus nurture dichotomy, instead calling for a nuanced, multifactorial approach to ontogeny. Similarly, the role of genes in both phylogeny and ontogeny, once seen as rather deterministic, is now conceptualized as highly dependent on environmental factors, including behavior. Linguistic theories have, in principle, made an effort to incorporate these changing views. However, the central claim of the given paper is that this apparent compliance with (...) biological insights remains superficial. As such, considerable disconnects between linguistic theory and what is known about the biological underpinnings of complex traits persist, negatively impacting pertinent views on language acquisition, language universals and the evolution of language. Given the breadth of these fields of study, the aim of this paper is to tackle the root of the problem: It begins by sketching out linguistic nativism as conceptualized within generativism, pointing to aspects within this position that stand in conflict with the interdisciplinary literature. It will then review select areas of research in a succinct manner in order to substantiate the criticism and characterize the counterposition as found within the biological sciences. The paper will culminate in addressing these disconnects on conceptual grounds, i.e. invoking the term emergence as employed in neuroscience as a possible means to reconcile those biological insights with linguistic nativism. (shrink)