David Bohm's interpretation of quantum mechanics yields a quantum potential, Q. In his early work, the effects of Q are understood in causal terms as acting through a real (quantum) field which pushes particles around. In his later work (with Basil Hiley), the causal understanding of Q appears to have been abandoned. The purpose of this paper is to understand how the use of certain metaphors leads Bohm away from a causal treatment of Q, and to evaluate (...) the use of those metaphors. (shrink)

In defining physical (i.e. causal dynamic) units to which conscious experience is to be ascribed, integrated information theory (IIT) raises three notable requirements: (1) that a unit to which consciousness is ascribed must be defined, or circumscribed, by some intrinsic aspect or property, where intrinsic implies existing 'for itself' or 'from its point of view'; (2) that the intrinsic aspect that defines the unit to which consciousness is ascribed must be dynamic (i.e. involve causal power) rather than purely (...) structural or kinematic; (3) that this dynamic aspect must involve the integration of elements of information in such a way that semantic values of elements are interdependent, allowing rich meaning. It will be argued that, although these requirements are all well-motivated, IIT falls short on them within a testable natural science framework. Concerns raised about the three requirements all point towards one central problem: that the concept of 'integrated information' does not involve any specific causal relation. Although IIT starts from strictly causal premises, the final analysis appears to invoke two incompatible accounts of the same events, with 'integration' having no causal relational basis. It will also be argued that the way IIT attempts to provide a testable theory of dynamic units to which consciousness is ascribed mustcausal reason inherent in any means of testing, be at best redundant for in the human case and, as a general principle, untenable. (shrink)

RESUMEN Las teorías de la referencia puramente descriptivistas o causales fracasan a la hora de dar cuenta del modo en que se fija y puede rastrearse la referencia de los términos teóricos. Psillos propuso dos versiones del descriptivismo causal que recogen argumentos presentes en defensas previas de dicha posición. Se trata de una teoría mixta que pretende solucionar el problema y acomodarse a intuiciones presentes en enfoques alternativos, como el que apela a oraciones de Ramsey. El artículo se propone (...) mostrar la insuficiencia del descriptivismo causal como solución al problema de la referencia de los términos teóricos. ABSTRACT Purely descriptivist or purely causal reference theories fail to account for the manner in which the reference of theoretical terms is fixed and can be traced. S. Psillos proposed two versions of causal descriptivism that take up arguments set forth in previous defenses of said position. His is a mixed theory that attempts to solve the problem by adopting insights from alternative approaches, such as those that resort to Ramsey sentences. The objective of the article is to show the insufficiency of causal descriptivism as a solution to the problem of reference of theoretical terms. (shrink)

According to the causal-historical theory of reference, natural kind terms refer in virtue of complicated causal relations the speakers have to their environment. A common objection to the theory is that purely causal relations are insufficient to fix reference in a determinate fashion. The so-called hybrid view holds that what is also needed for successful fixing are true descriptions associated in the mind of the speaker with the referent. The main claim of this paper is that the (...) objection fails: reference fixing of natural kind terms can be purely causal. The main argument draws inspiration from recent theoretical advances made in metaphysics of kinds by Marion Godman, Antonella Mallozzi, and David Papineau. The main claim is that their notion of super-explanatory properties may explain how reference of many kind terms can be fixed purely causally. (shrink)

In this paper I argue for a novel interpretation of Shepherd’s notion of selfhood. In distinction to Deborah Boyle’s interpretation, I contend that Shepherd differentiates between the mind and the self. The latter, for Shepherd, is an effect arising from causal interactions between mind and body – specifically those interactions that give rise to our present stream of consciousness, our memories, and that can unite these two. Thus, the body plays a constitutive role in the formation of the self. (...) The upshot of this interpretation is that it can dissolve the problem of individuating mind that Boyle identifies. Briefly, the problem consists in Shepherd seemingly individuating minds in terms of their associated bodies and bodies in terms of the minds they are united with. My interpretation, however, allows to see that Shepherd neither wants nor needs to individuate the mind in isolation of the body and to read the passages, in which the problem seems to arise, as being about what makes living beings individual – with mind and body both playing a crucial role in this context. (shrink)

This book offers a discussion about how people think, talk, learn, and explain things in causal terms in terms of action and manipulation. Sloman also reviews the role of causality, causal models, and intervention in the basic human cognitive functions: decision making, reasoning, judgement, categorization, inductive inference, language, and learning.

The Causal Exclusion Problem is raised in many domains, including in the metaphysics of macroscopic objects. If there is a complete explanation of macroscopic effects in terms of the microscopic entities that compose macroscopic objects, then the efficacy of the macroscopic will be threatened with exclusion. I argue that we can avoid the problem if we accept that macroscopic objects are ontically vague. Then, it is indeterminate which collection of microscopic entities compose them, and so information about microscopic entities (...) is insufficient to provide a complete explanation of certain properties of macroscopic objects. After outlining this solution, I consider several objections. (shrink)

The Causal Theory of Quantum Mechanics provides a better understanding of the fundamentals of quantum mechanics than is provided by Orthodox (i.e. Copenhagen) Quantum Theory by describing micro-phenomena in terms of entities and processes in space and time, thereby embracing causality at the quantum level. The book focuses especially on finding solutions to conceptual issues about the nature of energy, the conservation of energy, forces, and the Exclusion Principle within the context of the Causal Theory of Quantum Mechanics.

Causal selection is the task of picking out, from a field of known causally relevant factors, some factors as elements of an explanation. The Causal Parity Thesis in the philosophy of biology challenges the usual ways of making such selections among different causes operating in a developing organism. The main target of this thesis is usually gene centrism, the doctrine that genes play some special role in ontogeny, which is often described in terms of information-bearing or programming. This (...) paper is concerned with the attempt of confronting the challenge coming from the Causal Parity Thesis by offering principles of causal selection that are spelled out in terms of an explicit philosophical account of causation, namely an interventionist account. I show that two such accounts that have been developed, although they contain important insights about causation in biology, nonetheless fail to provide an adequate reply to the Causal Parity challenge: Ken Waters's account of actual-difference making and Jim Woodward's account of causal specificity. A combination of the two also doesn't do the trick, nor does Laura Franklin-Hall's account of explanation (in this volume). We need additional conceptual resources. I argue that the resources we need consist in a special class of counterfactual conditionals, namely counterfactuals the antecedents of which describe biologically normal interventions. (shrink)

One of Stuart Glennan's most prominent contributions to the new mechanist debate consists in his reductive analysis of higher-level causation in terms of mechanisms (Glennan, 1996). In this paper I employ the causal Bayes net framework to reconstruct his analysis. This allows for specifying general assumptions which have to be satis ed to get Glennan's approach working. I show that once these assumptions are in place, they imply (against the background of the causal Bayes net machinery) that higher-level (...) causation indeed reduces to interactions between component parts of mechanisms. I also briefly discuss the plausibility of these assumptions and some consequences for the mechanism debate. (shrink)

Causal claims in biomedical contexts are ubiquitous albeit they are not always made explicit. This paper addresses the question of what causal claims mean in the context of disease. It is argued that in medical contexts causality ought to be interpreted according to the epistemic theory. The epistemic theory offers an alternative to traditional accounts that cash out causation either in terms of “difference-making” relations or in terms of mechanisms. According to the epistemic approach, causal claims tell (...) us about which inferences (e.g., diagnoses and prognoses) are appropriate, rather than about the presence of some physical causal relation analogous to distance or gravitational attraction. It is shown that the epistemic theory has important consequences for medical practice, in particular with regard to evidence-based causal assessment. (shrink)

Some non-reductionists claim that so-called ‘exclusion arguments’ against their position rely on a notion of causal sufficiency that is particularly problematic. I argue that such concerns about the role of causal sufficiency in exclusion arguments are relatively superficial since exclusionists can address them by reformulating exclusion arguments in terms of physical sufficiency. The resulting exclusion arguments still face familiar problems, but these are not related to the choice between causal sufficiency and physical sufficiency. The upshot is that (...) objections to the notion of causal sufficiency can be answered in a straightforward fashion and that such objections therefore do not pose a serious threat to exclusion arguments. (shrink)

Recent attempts to resolve the Paradox of the Gatecrasher rest on a now familiar distinction between individual and bare statistical evidence. This paper investigates two such approaches, the causal approach to individual evidence and a recently influential (and award-winning) modal account that explicates individual evidence in terms of Nozick's notion of sensitivity. This paper offers counterexamples to both approaches, explicates a problem concerning necessary truths for the sensitivity account, and argues that either view is implausibly committed to the impossibility (...) of no-fault wrongful convictions. The paper finally concludes that the distinction between individual and bare statistical evidence cannot be maintained in terms of causation or sensitivity. We have to look elsewhere for a solution of the Paradox of the Gatecrasher. (shrink)

In his recent article, ``Self-Consciousness'’, George Bealer has set outa novel and interesting argument against functionalism in the philosophyof mind. I shall attempt to show, however, that Bealer's argument cannotbe sustained.In arguing for this conclusion, I shall be defending three main theses.The first is connected with the problem of defining theoreticalpredicates that occur in theories where the following two features arepresent: first, the theoretical predicate in question occurswithin both extensional and non-extensional contexts; secondly, thetheory in question asserts that the relevant (...) theoretical states enterinto causal relations. What I shall argue is that a Ramsey-styleapproach to the definition of such theoretical terms requires twodistinct quantifiers: one which ranges over concepts, and theother which ranges over properties in the world.My second thesis is a corollary: since the theories on whichBealer is focusing have both of the features just mentioned, and sincethe method that he employs to define theoretical terms in his argumentagainst functionalism does not involve both quantifiers that range overproperties and quantifiers that range over concepts, that method isunsound.My final thesis is that when a sound method is used, Bealer's argumentagainst functionalism no longer goes through.The structure of my discussion is as follows. I begin by setting out twoarguments – the one, a condensed version of Bealer's argument, andthe other, an argument that parallels Bealer's argument very closely.The parallel argument leads to a conclusion, however, that, rather thanbeing merely somewhat surprising, seems very implausible indeed. Forwhat the second argument establishes, if sound, is that there can betheoretical terms that apply to objects by virtue of their first-orderphysical properties, but whose meaning cannot be defined via aRamsey-style approach.Having set out the two parallel arguments, I then go on to focus uponthe second, to determine what is wrong with it. My diagnosis will bethat the problem with the argument arises from the fact that it involvesdefining a theoretical term that occurs both inside and outside ofopaque contexts, for the method employed fails to take into account thefact that the types of entities that are involved in the relevanttruthmakers are different when a sentence occurs within an extensionalcontext from those involved when a sentence occurs within anon-extensional context.I then go on to discuss how one should define a theoretical term thatoccurs within such theories, and I argue that in such a case one needstwo quantifiers, ranging over different types of entities – on theone hand, over properties and relations, and the other, over concepts. Ithen show that, when such an approach is followed, the argument inquestion collapses.I then turn to Bealer's argument against functionalism, and I show,first, that precisely the same method of defining theoretical terms canbe applied there, and, secondly, that, when this is done, it turns outthat that argument is also unsound.Next, I consider two responses that Bealer might make to my argument,and I argue that those responses would not succeed.Finally, I conclude by asking exactly where the problem lies in the caseof Bealer's argument. My answer will be that it is not simply the factthat one is dealing with a theoretical term that occurs in bothextensional and non-extensional contexts. It is rather the combinationof that feature together with the fact that the theory in questionasserts that the relevant type of theoretical state enters into causalrelations. For the first of these features means that the Ramseysentence for the theory must involve quantification over concepts, whilethe presence of the second feature means that the Ramsey sentence mustinvolve quantification over properties in the world, and so no attemptto offer a Ramsey-style account of the meaning of the relevanttheoretical term can succeed unless one employs both quantification overconcepts and quantification over properties. Bealer, however, in hisargument against functionalism, uses a method of defining theoreticalterms that does not involve both types of quantification, and it isprecisely because of this that his argument does not in the end succeed. (shrink)

The paper examines definitions of ‘cause’ in the epidemiological literature. Those definitions all describe causes as factors that make a difference to the distribution of disease or to individual health status. In the philosophical jargon, causes in epidemiology are difference-makers. Two claims are defended. First, it is argued that those definitions underpin an epistemology and a methodology that hinge upon the notion of variation, contra the dominant Humean paradigm according to which we infer causality from regularity. Second, despite the fact (...) that causes be defined in terms of ‘difference-making’, this cannot fixes the causal metaphysics. Causality in epidemiology ought to be interpreted according to the epistemic theory. In this approach relations are deemed causal depending on the evidence and on the available methods. Indeed, evidence to establish causal claims requires difference-making considerations; furthermore, those definitions of cause reflect the ‘variational’ epistemology and methodology of epidemiology. (shrink)

The term ‘realism’ and its contrasting terms have various related senses, although often they occlude as much as they illuminate, especially if ontological and epistemological issues and their tenable combinations are insufficiently clarified. For example, in 1807 the infamous ‘idealist’ Hegel argued cogently that any tenable philosophical theory of knowledge must take the natural and social sciences into very close consideration, which he himself did. Here I argue that Hegel ably and insightfully defends Newton’s causal realism about gravitational (...) force, in part by exposing a fatal equivocation in the traditional concept of substance, by criticizing some still-standard empiricist misconceptions of force, by emphasizing the role of explanatory integration in Newtonian mechanics, and by using his powerful semantics of singular, specifically cognitive reference to justify fallibilism regarding empirical justification, together with the semantic core of Newton’s Rule Four of (experimental) Philosophy—in a way that highlights a key fallacy in many arguments against realism, both in epistemology and within philosophy of science. (shrink)

The idea that causation can be reduced to transmission of an amount of some conserved quantity between events is spelled out and defended against important objections. Transmission is understood as a symmetrical relation of copresence in two distinct events. The actual asymmetry of causality has its origin in the asymmetrical character of certain irreversible physical processes and then spreads through the causal net. This conception is compatible with the possibility of backwards causation and with a causal theory of (...) time. Genidentity, the persistence of concrete objects, can be given an explanation in causal terms. The transmission theory is shown to escape difficulties faced by two important alternative theories of causation: Salmon's (1984) Mark Transmission Theory and Dowe's (1992a) Conserved Quantities Theory. (shrink)

Causal realism is the view that causation is a structural feature of reality; a power inherent in the world to produce effects, independently of the existence of minds or observers. This article suggests that certain problems in the philosophy of mind are artefacts of causal realism; because they presuppose the existence or possibility of a mind-independent causal nexus between the ‘physical’ and the ‘mental’. These dilemmas include the 'hard problem' of consciousness, and the problems of free will (...) and mental causality. Since the ostensible causal nexus cannot be directly perceived, it is sublimated into obscure and elusive phenomena along the purported mental causal chain. The antithesis of causal realism, and the proposed solution to the problems above, is causal anti-realism: the view that causation is not a fundamental property of the world, but of how observers purposively interpret ‘the world’. Causal anti-realism is compatible with causal pragmatism, which allows for the practical use of causal terms. Causal anti-realism denies the possibility of ontological reduction and is therefore incompatible with materialism. The article concludes that causal anti-realism is at least prima facie reconcilable with idealism. (shrink)

Minimally, causal realism (as understood here) is the view that accounts of causation in terms of mere, regular or probabilistic conjunction are unsatisfactory, and that causal phenomena are correctly associated with some form of de re necessity. Classic arguments, however, some of which date back to Sextus Empiricus and have appeared many times since, including famously in Russell, suggest that the very notion of causal realism is incoherent. In this paper I argue that if such objections seem (...) compelling, it is only because everyday expressions concerning causal phenomena are misleading with respect to certain metaphysical details. These expressions generally make reference to the relations of events or states of affairs, but ignore or obscure the role played by causal properties. I argue that on a proposed alternative, an analysis in terms of causal processes, more refined descriptions of causal phenomena escape the charge of incoherence. Causal necessity is here located in the relations of causal properties. I distinguish this view from the recent process theories of Salmon and Dowe, which are disinterested in causal realism. (shrink)

The belief is widely held that the physical world is causally-driven. The world is one because a tangled web of causally-driven processes keeps it together. However, both the psychological and the social worlds cannot be articulated in causal terms only. Hereby, motivation is used as the most general term referring to whatever keeps (synchronically) together and provides (diachronic) reasons explaining the behavior of psychological and social systems. In order to systematically address these problems, a categorical framework is needed (...) for understanding the various types of realities populating the world and their multifarious interrelations. The papers collected in this volume dig into some of the intricacies presented by these problems. The papers here presented have been selected from those presented at the workshops bearing the very same name, Causality and Motivation organized in Bolzano and Rome.". (shrink)

Anscombe’s “Causality and Determination” is often cited in the contemporary free will debate, but rarely discussed in much detail. It’s main contribution, it is thought, is the defense of an alternative to deterministic causation, thus clearing the way for an incompatibilist analysis of free actions in terms of probabilistic causation. However, in this paper I will show that the contemporary probabilistic analysis of free action actually stands in direct conflict with Anscombe’s lecture. Instead, I will argue, its true value for (...) incompatibilist accounts of free will lies in Anscombe’s thought that there are various fundamentally different kinds of causality. This variety, I argue, allows for a revised conception indeterminism and an understanding of free will as the manifestation of a particular sort of agent-causal power. (shrink)

It is a common view that Descartes' causal principle is to be understood in light of a similarity condition that accounts for how finite causes contribute to an explanation of their effects. This paper challenges this common view and offers a sui generis reading of Descartes' views on causation that has also the advantage of solving the two exegetical issues of whether Descartes thought of the body-to-mind relation in occasionalist or causal terms and of whether Descartes regarded sensory (...) ideas innate or caused by bodies. (shrink)

Causal essentialists hold that a property essentially bears its causal and nomic relations. Further, as many causal essentialists have noted, the main motivations for causal essentialism also motivate holding that properties are individuated in terms of their causal and nomic relations. This amounts to a kind of identity of indiscernibles thesis; properties that are indiscernible with respect to their causal and nomic relations are identical. This can be compared with the more well-known identity of (...) indiscernibles thesis, according to which particulars that are qualitatively indiscernible are identical. Robert Adams has developed a well-known objection to this thesis by considering a series of possibilities involving nearly qualitatively indiscernible particulars that naturally leads to a possibility involving qualitatively indiscernible particulars. I argue that we can construct parallel cases involving a series of possibilities involving properties that are nearly indiscernible with respect to their causal and nomic relations that naturally lead to possibilities involving properties that are indiscernible with respect to their causal and nomic relations. The same features that make Adams’ argument forceful also carry over to my cases, giving us a powerful objection to the causal essentialist identity of indiscernibles thesis. (shrink)

Among the factors necessary for the occurrence of some event, which of these are selectively highlighted in its explanation and labeled as causes — and which are explanatorily omitted, or relegated to the status of background conditions? Following J. S. Mill, most have thought that only a pragmatic answer to this question was possible. In this paper I suggest we understand this ‘causal selection problem’ in causal-explanatory terms, and propose that explanatory trade-offs between abstraction and stability can provide (...) a principled solution to it. After sketching that solution, it is applied to a few biological examples, including to a debate concerning the ‘causal democracy’ of organismal development, with an anti-democratic (though not a gene-centric) moral. (shrink)

Causal reasoning is an aspect of learning, reasoning, and decision-making that involves the cognitive ability to discover relationships between causal relata, learn and understand these causal relationships, and make use of this causal knowledge in prediction, explanation, decision-making, and reasoning in terms of counterfactuals. Can we fully automate causal reasoning? One might feel inclined, on the basis of certain groundbreaking advances in causal epistemology, to reply in the affirmative. The aim of this paper is (...) to demonstrate that one still has good skeptical grounds for resisting any conclusions in favour of the automation of causal reasoning. If by causal reasoning is meant the entirety of the process through which we discover causal relationships and make use of this knowledge in prediction, explanation, decision-making, and reasoning in terms of counterfactuals, then one relies besides on tacit knowledge, as might be constituted by or derived from the epistemic faculty virtues and abilities of the causal reasoner, the value systems and character traits of the causal reasoner, the implicit knowledge base available to the causal reasoner, and the habits that sustain our causal reasoning practices. While certain aspects of causal reasoning may be axiomatized and formalized and algorithms may be implemented to approximate causal reasoning, one has to remain skeptical about whether causal reasoning may be fully automated. This demonstration will involve an engagement with Meno’s Paradox. (shrink)

Causal selection and priority are at the heart of discussions of the causal parity thesis, which says that all causes of a given effect are on a par, and that any justified priority assigned to a given cause results from causal explanatory interests. In theories of causation that provide necessary and sufficient conditions for the truth of causal claims, status as cause is an either/or issue: either a given cause satisfies the conditions or it does not. (...) Consequently, assessments of causal parity and priority require more resources, which can either be additional or part of the causal analysis itself. While adding resources in terms of a theory of causal explanation has been standard, here we develop a unified conceptual analysis that includes a range of different precise causal concepts that allows for assessments of causal priority in terms of different kinds of causal relevance. (shrink)

Baumard suggests that the advent, through phenotypic plasticity mechanisms, of future-oriented preferences and creative mindsets in eighteenth-century Great Britain explains the wave of innovations that drove the British Industrial Revolution. We argue that, although this approach is promising, Baumard's model would benefit from being supplemented by demographic, economic, and sociological explanations independent of Life History Theory.

For those who maintain that free will is incompatible with causal determinism, a persistent problem is to give a coherent characterization of action that is neither determined by prior events nor random, arbitrary, lucky or in some way insufficiently under the control of the agent to count as free action. One approach—that of Roderick Chisholm and others—is to say that a third alternative is for an action to be caused by an agent in a way that is not reducible (...) to event causal terms. A different approach than the Chisholmian appeal to primitive substance causation is one that, instead, involves causal relations purely among events. This paper presents a particular event-causal indeterminist account of free action, describing both its attractions and recent objections to it, and then proposes a revised version, with the aim of supporting the plausibility of an event-causal indeterminist approach to free will. (shrink)

I argue that one central aspect of the epistemology of causation, the use of causes as evidence for their effects, is largely independent of the metaphysics of causation. In particular, I use the formalism of Bayesian causal graphs to factor the incremental evidential impact of a cause for its effect into a direct cause-to-effect component and a backtracking component. While the “backtracking” evidence that causes provide about earlier events often obscures things, once we our restrict attention to the cause-to-effect (...) component it is true to say promoting (inhibiting) causes raise (lower) the probabilities of their effects. This factoring assumes the same form whether causation is given an interventionist, counterfactual or probabilistic interpretation. Whether we think about causation in terms of interventions and causal graphs, counterfactuals and imaging functions, or probability raising against the background of causally homogenous partitions, if we describe the essential features of a situation correctly then the incremental evidence that a cause provides for its effect in virtue of being its cause will be the same. (shrink)

Heritability is routinely interpreted causally. Yet, what such an interpretation amounts to is often unclear. Here, I provide a causal interpretation of this concept in terms of range of causal influence, one of several causal dimensions proposed within the interventionist account of causation. An information-theoretic measure of range of causal influence has recently been put forward in the literature. Starting from this formalization and relying upon Woodward’s analysis, I show that an important problem associated with interpreting (...) heritability causally, namely the locality problem, amounts, at least partly, to a low invariance and low stability between the genotype/environment and the phenotype of individuals. In light of this, I plead for a causal interpretation of heritability that takes the notions of Woodward’s invariance and stability into consideration. In doing so, I defuse naive causal interpretations of heritability. (shrink)

This dissertation defends the view that concepts encode causal information and, for the first time, applies this view to a range of topics in the philosophy of language and social philosophy. In my first chapter (“Cognitive Essentialism and the Structure of Concepts”), I survey the current empirical and theoretical literature on causal-essentialist theories of concepts. In my second chapter (“Meaning Externalism and Causal Model Theory”), I propose an account of natural kind concepts according to which they encode (...) statistical information of features of a natural kind, and represents these features as causally related to each other. I show that this internalist model of concepts correctly predicts intuitions about Putnam’s twin earth scenario and Kripke’s conceivability cases that historically motivated philosophers of language to accept externalist accounts of meaning. The defended theory of concepts also informs topics that go beyond traditional issues in philosophy of language. In my third chapter (“An Essentialist Theory of the Meaning of Slurs”), I defend the view that slurs are, too, a species of kind terms: slur concepts encode mini-theories which represent an essence-like element that is causally connected to a set of negatively-valenced stereotypical features of a social group. This explains both the peculiar linguistic behavior of slurs and slurs’ dehumanizing effects. In my fourth chapter, I build on this insight, showing that the explicit language in and around pornography depicts women as ‘kinds’ or ‘breeds’ that are naturally made to enjoy certain sexual acts, and argue that this deterministic picture of women dehumanizes them. (shrink)

In this contribution, I explore the possibility of characterizing the emergence of time in causal set theory (CST) in terms of the growing block universe (GBU) metaphysics. I show that although GBU seems to be the most intuitive time metaphysics for CST, it leaves us with a number of interpretation problems, independently of which dynamics we choose to favor for the theory —here I shall consider the Classical Sequential Growth and the Covariant model. Discrete general covariance of the CSG (...) dynamics does not allow us to individuate a single history of the universe (defined by a causal history of different causal sets), thereby making the claim that ‘the past exists’ at best problematic. In addition, because the evolution of the universe in CSG dynamics leads to an outward branching causal tree, it becomes impossible to determine a proper ‘line of becoming’, thereby blurring the presentists’ claim that only the present exists. Similarly, the covariant approach runs into the same, if not even more severe problems, since each configuration of the universe would amount to a set of possible causal sets, thereby making the individuation of a single configuration of the universe —and thus the physical interpretation of the theory— implausible. (shrink)

I discuss an important feature of the notion of cause in Post. An. 1. 13, 78b13–28, which has been either neglected or misunderstood. Some have treated it as if Aristotle were introducing a false principle about explanation; others have understood the point in terms of coextensiveness of cause and effect. However, none offers a full exegesis of Aristotle's tangled argument or accounts for all of the text's peculiarities. My aim is to disentangle Aristotle's steps to show that he is arguing (...) in favour of a logical requirement for a middle term's being the appropriate cause of its explanandum. Coextensiveness between the middle term and the attribute it explains is advanced as a sine qua non condition of a middle term's being an appropriate or primary cause. This condition is not restricted either to negative causes or to middle terms in second‐figure syllogisms, but ranges over all primary causes qua primary. (shrink)

It is natural to think of remembering in terms of causation: I can recall a recent dinner with a friend because I experienced that dinner. Some fifty years ago, Martin and Deutscher (1966) turned this basic thought into a full-fledged theory of memory, a theory that came to dominate the landscape in the philosophy of memory. Remembering, Martin and Deutscher argue, requires the existence of a specific sort of causal connection between the rememberer's original experience of an event and (...) his later representation of that event: a causal connection sustained by a memory trace. In recent years, it has become apparent that this reference to memory traces may be out of step with memory science. Contemporary proponents of the causal theory are thus confronted with the question: is it possible to develop an empirically adequate version of the theory, or is it time to move beyond it? This chapter traces the recent history of the causal theory, showing how increased awareness of the theory’s problems has led to the development of modified version of the causal theory and ultimately to the emergence of postcausal theories. (shrink)

With the advent of evolutionary developmental research, or EvoDevo, there is hope of discovering the roles that the genetic bases of development play in morphological evolution. Studies in EvoDevo span several levels of organismal organization. Low-level studies identify the ultimate genetic changes responsible for morphological variation and diversity. High-level studies of development focus on how genetic differences affect the dynamics of gene networks and epigenetic interactions to modify morphology. Whereas an increasing number of studies link independent acquisition of homoplastic or (...) convergent morphologies to similar changes in the genomes, homoplasies are not always found to have identical low-level genetic underpinnings. This suggests that a combination of low- and high-level approaches may be useful in understanding the relationship between genetic and morphological variation. Therefore, as an empirical and conceptual framework, we propose the causality horizon to signify the lowest level that allows linking homoplastic morphologies to similar changes in the development. A change in a system below the causality horizon cannot be generalized. In more concrete terms, homoplastic morphologies cannot be reduced to the same change in gene regulation when that change occurs below the causality horizon; rather, a higher-level mechanism should be identified. (shrink)

Causal Dispositionalism provides an account of causation based on an ontology of causal powers, properties with causal essence. According to the account, causation can be analysed in terms of the interaction of powers and its subsequent production of their effect. Recently, Baltimore, J. A. has raised a challenge against two competing approaches, the compositional view and the mutual manifestation view, to explain what makes powers interactive – the interaction gap. In this paper, we raise the challenge of (...) explaining what makes powers productive – the production gap. While Baltimore’s verdict is tentatively favouring, we find both approaches wanting. Our conclusion is that Causal Dispositionalists should take Baltimore’s and our critique seriously. Powers cannot cause their effects just by bearing the name “causal”. To deserve their names, more metaphysical details are needed. (shrink)

Talk of powers is muddled. Building upon Powers and capacities in philosophy: The new aristotelianism, Routledge, London, 2012a, pp 207–227), I disambiguate four senses of the term: powers construed as activity, as capacity/potentiality, as essence and as necessity, respectively, in an attempt to clarify what it is that realists about causal powers take themselves to be realists about.

In the last two decades few topics in philosophy of science have received as much attention as mechanistic explanation. A significant motivation for these accounts is that scientists frequently use the term “mechanism” in their explanations of biological phenomena. While scientists appeal to a variety of causal concepts in their explanations, many philosophers argue or assume that all of these concepts are well understood with the single notion of mechanism. This reveals a significant problem with mainstream mechanistic accounts– (...) although philosophers use the term “mechanism” interchangeably with other causal concepts, this is not something that scientists always do. This paper analyses two causal concepts in biology–the notions of “mechanism” and “pathway”–and how they figure in biological explanation. I argue that these concepts have unique features, that they are associated with distinct strategies of causal investigation, and that they figure in importantly different types of explanation. (shrink)

Jaegwon Kim’s causal exclusion argument says that if all physical effects have sufficient physical causes, and no physical effects are caused twice over by distinct physical and mental causes, there cannot be any irreducible mental causes. In addition, Kim has argued that the nonreductive physicalist must give up completeness, and embrace the possibility of downward causation. This paper argues first that this extra argument relies on a principle of property individuation, which the nonreductive physicalist need not accept, and second (...) that once we get clear on overdetermination, there is a way to reject the exclusion principle upon which the causal exclusion argument depends, but third that this should not lead to the belief that mental causation is easily accounted for in terms of counterfactual dependencies. (shrink)

The causal closure of physics is usually discussed in a context free way. Here I discuss it in the context of engineering systems and biology, where strong emergence takes place due to a combination of upwards emergence and downwards causation. Firstly, I show that causal closure is strictly limited in terms of spatial interactions because these are cases that are of necessity strongly interacting with the environment. Effective Spatial Closure holds ceteris parabus, and can be violated by Black (...) Swan Events. Secondly, I show that causal closure in the hierarchy of emergence is a strictly interlevel affair, and in the cases of engineering and biology encompasses all levels from the social level to the particle physics level. However Effective Causal Closure can usefully be defined for a restricted set of levels, and one can experimentally determine Effective Theories that hold at each level. This does not however imply those effective theories are causally complete by themselves. In particular, the particle physics level is not causally complete by itself in the contexts of solid state physics, digital computers, or biology. Furthermore Inextricably Intertwined Levels occur in all these contexts. (shrink)

Powers, capacities and dispositions (in what follows I will use these terms synonymously) have become prominent in recent debates in metaphysics, philosophy of science and other areas of philosophy. In this paper I will analyse in some detail a well-known argument from scientific practice to the existence of powers/capacities/dispositions. According to this argument the practice of extrapolating scientific knowledge from one kind of situation to a different kind of situation requires a specific interpretation of laws of nature, namely as attributing (...) dispositions to systems. My main interest will be to discuss what characteristics these dispositions need to have in order to account for the scientific practice in question. I will furthermore assess whether the introduction of dispositions in the context of the extrapolation argument can be described as a ‘revitalization’ or as a ‘return’ to those notions repudiated by early modern philosophers. More particularly I will argue for the following claims: I. In repudiating scholastic terminology, including substantial forms with their causal powers, post-cartesian philosophers focussed on a concept of causation that was much stronger than 21st century conceptions of causation. For this reason alone, whatever ‘causal’ is supposed to mean in today’s causal powers, embracing causal powers is not a simple return to a pre-cartesian notion. II. The dispositions presupposed in scientific practice need not (and should not) be construed in causal terms (whether strong or weak). III. While some early modern philosophers contrasted the characterisation of the natural world in terms of substantial forms (and their causal powers) on the one hand and a mathematical characterization on the other and suggested that these approaches are incompatible, the dispositions postulated by the extrapolation argument to account for scientific practice are themselves characterized in mathematical terms. More precisely: The behaviour the systems are disposed to display is – at least in physics – often characterized in mathematical terms. IV. The dispositions assumed in the law-statements in scientific practice are determinable rather than determinate properties. (shrink)

Abstract In this paper I consider an easier-to-read and improved to a certain extent version of the causal chance-based analysis of counterfactuals that I proposed and argued for in my A Theory of Counterfactuals. Sections 2, 3 and 4 form Part I: In it, I survey the analysis of the core counterfactuals (in which, very roughly, the antecedent is compatible with history prior to it). In section 2 I go through the three main aspects of this analysis, which are (...) the following. First, it is a causal analysis, in that it requires that intermediate events to which the antecedent event is not a cause be preserved in the main truth-condition schema. Second, it highlights the central notion to the semantics of counterfactuals on the account presented here -- the notion of the counterfactual probability of a given counterfactual, which is the probability of the consequent given the following: the antecedent, the prior history, and the preserved intermediate events. Third, it considers the truth conditions for counterfactuals of this sort as consisting in this counterfactual probability being higher than a threshold. In section 3, I re-formulate the analysis of preservational counterfactuals in terms of the notion of being a cause, which ends up being quite compact. In section 4 I illustrate this analysis by showing how it handles two examples that have been considered puzzling – Morgenbesser's counterfactual and Edgington's counterfactual. Sections 5 and on constitute Part II: Its main initial thrust is provided in section 5, where I present the main lines of the extension of the theory from the core counterfactuals (analyzed in part I) to counterfactuals (roughly) whose antecedents are not compatible with their prior history. In this part II, I elaborate on counterfactuals that don't belong to the core, and more specifically on so-called reconstructional counterfactuals (as opposed to the preservational counterfactuals, which constitute the core counterfactual-type). The heart of the analysis is formulated in terms of processes leading to the antecedent (event/state), and more specifically in terms of processes likely to have led to the antecedent, a notion which is analyzed entirely in terms of chance. It covers so-called reconstructional counterfactuals as opposed to the core, so-called preservational counterfactuals, which are analyzed in sections 2 and 3 of part I. The counterfactual probability of such reconstructional counterfactuals is determined via the probability of possible processes leading to the antecedent weighed, primarily and roughly, by the conditional probability of the antecedent given such process: The counterfactual probability is thus, very roughly, a weighted sum for all processes most likely to have led to the antecedent, diverging at a fixed time. In section 6 I explain and elaborate further on the main points in section 5. In section 7 I illustrate the reconstructional analysis. I specify counterfactuals which are so-called process-pointers, since their consequent specifies stages in processes likely to have led to their antecedent. I argue that so-called backtracking counterfactuals are process-pointers counterfactuals, which fit into the reconstructional analysis, and do not call for a separate reading. I then illustrate cases where a speaker unwittingly employs a certain counterfactual while charitably construable as intending to assert (or ‘having in mind’) another. Here I also cover the issue of how to construe what one can take as back-tracking counterfactuals, or counterfactuals of the reconstructional sort, and more specifically, which divergence point they should be taken as alluding to (prior to which the history is held fixed). Some such cases also give rise to what one can take as a dual reading of a counterfactual between preservational and reconstructional readings. Such cases may yield an ambiguity, where in many cases one construal is dominant. In section 8 I illustrate the analysis by applying it to the famous Bizet-Verdi counterfactuals. This detailed analysis of counterfactuals (designed for the indeterministic case) has three main distinctive elements: its being chance-based, its causal aspect, and the use it makes of processes most likely to have led to the antecedent-event. This analysis is couched in a very different conceptual base from, and is an alternative account to, analyses in terms of the standard notion of closeness or distance of possible worlds, which is the main feature of the Stalnaker-Lewis-type analyses of counterfactuals. This notion of closeness or distance plays no role whatsoever in the analysis presented here. (This notion of closeness has been left open by Stalnaker, and to significant extent also by Lewis's second account.) . (shrink)

When evaluating the success of causal theories of explanation the focus has typically been on the legitimacy of causal relations and on putative examples of explanations that we cannot capture in causal terms. Here I motivate the existence of a third kind of problem: the difficulty of accounting for explanatory disputes. Moreover, I argue that this problem remains even if the first two are settled and that it threatens to undercut one of the central motivations for (...) class='Hi'>causal accounts of explanation, namely, the causal account of the directionality of scientific explanation. (shrink)

In the social constructionist literature, little has been said about what it means for social factors to cause X in such a way that X would count as causally socially constructed. In this paper, I argue that being caused by social factors – and thus being causally socially constructed – is best defined in terms of a contrastive counterfactual notion of causation. Unlike some plausible alternatives, this definition captures what is at stake in actual social constructionist debates. It makes transparent (...) which factors the truth of a causal constructionist claim may depend on. By doing so, it sheds light on what the disagreements over whether X is causally socially constructed may turn on. It also helps us to see under which condition the claim that X is socially causally constructed is compatible with the claim that X is caused by biological factors. (shrink)