Rousseau is considered to be at once the most modern political thinker of the 18th century and the most ancient in his allegiance to classical republicanism. These essays address the place of liberty in his moral and political philosophy, and the origins, meaning, strength, weakness and significance of his argument.

Is the common cause principle merely one of a set of useful heuristics for discovering causal relations, or is it rather a piece of heavy duty metaphysics, capable of grounding the direction of causation itself? Since the principle was introduced in Reichenbach’s groundbreaking work The Direction of Time (1956), there have been a series of attempts to pursue the latter program—to take the probabilistic relationships constitutive of the principle of the common cause and use them to ground the direction of (...) causation. These attempts have not all explicitly appealed to the principle as originally formulated; it has also appeared in the guise of independence conditions, counterfactual overdetermination, and, in the causal modelling literature, as the causal markov condition. In this paper, I identify a set of difficulties for grounding the asymmetry of causation on the principle and its descendents. The first difficulty, concerning what I call the vertical placement of causation, consists of a tension between considerations that drive towards the macroscopic scale, and considerations that drive towards the microscopic scale—the worry is that these considerations cannot both be comfortably accommodated. The second difficulty consists of a novel potential counterexample to the principle based on the familiar Einstein Podolsky Rosen (EPR) correlations in quantum mechanics. (shrink)

Biologists studying complex causal systems typically identify some factors as causes and treat other factors as background conditions. For example, when geneticists explain biological phenomena, they often foreground genes and relegate the cellular milieu to the background. But factors in the milieu are as causally necessary as genes for the production of phenotypic traits, even traits at the molecular level such as amino acid sequences. Gene-centered biology has been criticized on the grounds that because there is parity among causes, the (...) “privileging” of genes reflects a reductionist bias, not an ontological difference. The idea that there is an ontological parity among causes is related to a philosophical puzzle identified by John Stuart Mill: what, other than our interests or biases, could possibly justify identifying some causes as the actual or operative ones, and other causes as mere background? The aim of this paper is to solve this conceptual puzzle and to explain why there is not an ontological parity among genes and the other factors. It turns out that solving this puzzle helps answer a seemingly unrelated philosophical question: what kind of causal generality matters in biology? (shrink)

Hunting Causes and Using Them argues that causation is not one thing, as commonly assumed, but many. There is a huge variety of causal relations, each with different characterizing features, different methods for discovery and different uses to which it can be put. In this collection of new and previously published essays, Nancy Cartwright provides a critical survey of philosophical and economic literature on causality, with a special focus on the currently fashionable Bayes-nets and invariance methods - and it exposes (...) a huge gap in that literature. Almost every account treats either exclusively how to hunt causes or how to use them. But where is the bridge between? It's no good knowing how to warrant a causal claim if we don't know what we can do with that claim once we have it. This book will interest philosophers, economists and social scientists. (shrink)

People generally accept that there is causation by omission—that the omission of some events cause some related events. But this acceptance elicits the selection problem, or the difficulty of explaining the selection of a particular omissive cause or class of causes from the causal conditions. Some theorists contend that dependence theories of causation cannot resolve this problem. In this paper, we argue that the appeal to norms adequately resolves the selection problem for dependence theories, and we provide novel experimental evidence (...) for it. (shrink)

In Causes, Laws, and Free Will, Kadri Vihvelin argues that we can have free will even if everything we do is predictable given the laws of nature and the past. The belief that determinism robs us of free will springs from mistaken beliefs about the metaphysics of causation, the nature of laws, and the logic of counterfactuals.

Causation is everywhere in the world: it features in every science and technology. But how much do we understand it? Mumford and Anjum develop a new theory of causation based on an ontology of real powers or dispositions. They provide the first detailed outline of a thoroughly dispositional approach, and explore its surprising features.

Evolutionary Biology has two principal explananda, fit and diversity (Lewontin 1978). Natural selection theory stakes its claim to being the central unifying concept in biology on the grounds that it demonstrates both phenomena to be the consequence of a single process. By now the standard story hardly needs reiterating: Natural selection is a force that operates over a population, preserving the better fit, culling the less fit, and along the way promoting novel solutions to adaptive problems. Amundson’s historical survey of (...) the concept of adaptation captures the idea succinctly: The phenomenon of adaptation is at the core of modern evolutionary biology. Natural selection, the mechanism universally regarded as the primary causal influence on phenotypic evolutionary change, is first and foremost an explanation of adaptation. (1996: 11) At the same time, it appears that the capacity of natural selection to cause adaptations cannot account for every feature of the nature and distribution of biological form. Reason to believe this devolves from the simple fact that the bearers of biological form are organisms and a salient fact about organisms is that each faces the tribunal of the environment as a corporate entity, not as a loose aggregate of independent traits. One consequence of this fact is that at each stage of its development from egg to adult an organism must be an integrated, functioning whole. Another is that for any form to arise in an organism at a time, it must develop from the materials and processes at the organism’s disposal. The requirement of integration and the processes of development that produce it leave their distinctive marks on form. It seems reasonable, then, to suppose that one might appeal to the processes of development and the principles by which they operate in explaining the nature and distribution of biological form. (See here Gould 1977; Rose and Lauder 1996). (shrink)

Michael T. Ferejohn presents a new analysis of Aristotle's theory of explanation and scientific knowledge, in the context of its Socratic roots. Ferejohn shows how Aristotle resolves the tension between his commitment to the formal-case model of explanation and his recognition of the role of efficient causes in explaining natural phenomena.

Paul Pietroski presents an original philosophical theory of actions and their mental causes. We often act for reasons: we deliberate and choose among options, based on our beliefs and desires. However, bodily motions always have biochemical causes, so it can seem that thinking and acting are biochemical processes. Pietroski argues that thoughts and deeds are in fact distinct from, though dependent on, underlying biochemical processes within persons.

Negative causation occurs when an absence serves as cause, effect, or causal intermediary. Negative causation is genuine causation, or so I shall argue. It involves no physical connection between cause and effect. Thus causes need not be physically connected to their effects.

Most people are skeptical of the claim that the expectation that a person would have a life that would be well worth living provides a reason to cause that person to exist. In this essay I argue that to cause such a person to exist would be to confer a benefit of a noncomparative kind and that there is a moral reason to bestow benefits of this kind. But this conclusion raises many problems, among which is that it must be (...) determined how the benefits conferred on people by causing them to exist weigh against comparable benefits conferred on existing people. In particular, might the reason to cause people to exist ever outweigh the reason to save the lives of existing people? (shrink)

The leading accounts of the nature of causation divide into probability-raising and process-linkage views. On the probability-raising view, causation is rooted in the comparative probability of the effect with the cause versus without. On the process-linkage view, causation is rooted in the existence of a connecting line from cause to effect. I propose a third alternative which synthesizes these views while solving their problem cases. On this alternative, causation is rooted in the comparative probability of the connecting line to the (...) effect with the cause versus without: causes as probability raisers of processes. In the first two sections, I (briefly) outline the probability-raising and process-linkage views and identify a space of problems, and then I focus on developing the alternative probability-raisers-of-processes solution. I identify problems for this view as well, though I conclude that it is at least in important respects a step forward. (shrink)

Philosophers have long been fascinated by the connection between cause and effect: are 'causes' things we can experience, or are they concepts provided by our minds? The study of causation goes back to Aristotle, but resurged with David Hume and Immanuel Kant, and is now one of the most important topics in metaphysics. Most of the recent work done in this area has attempted to place causation in a deterministic, scientific, worldview. But what about the unpredictable and chancey world we (...) actually live in: can one theory of causation cover all instances of cause and effect? _Cause and Chance: Causation in an Indeterministic World _is a collection of specially written papers by world-class metaphysicians. Its focus is the problem facing the 'reductionist' approach to causation: the attempt to cover all types of causation, deterministic and indeterministic, with one basic theory. Contributors: Stephen Barker, Helen Beebee, Phil Dowe, Dorothy Edgington, Doug Ehring, Chris Hitchcock, Igal Kwart, Paul Noordhof, Murali Ramachandran and Michael Tooley. (shrink)

Since the appearance of a widely influential book, Self-Knowledge and Self-ldentity, Sydney Shoemaker has continued to work on a series of interrelated issues in the philosophy of mind and metaphysics. This volume contains a collection of the most important essays he has published since then. The topics that he deals with here include, among others, the nature of personal and other forms of identity, the relation of time to change, the nature of properties and causality and the relation between the (...) two, dualism and immortality, and the nature of mental states. All the essays show the same care and precision in argument as the earlier book, but they also reveal a substantial shift in Professor Shoemaker's position to a form of materialism. In fact, a number of papers together constitute what is probably the most subtle and rigourous defence yet of a sophisticated functionalism in the account of the mind. (shrink)

A just cause for war is a type of wrong that may make those responsible for it morally liable to military attack as a means of preventing or rectifying it. This claim has implications that conflict with assumptions of the current theory of just war.

When is a cause of a cause of an effect also a cause of that effect? The right answer is either Sometimes or Always . In favour of Always , transitivity is considered by some to be necessary for distinguishing causes from redundant non-causal events. Moreover transitivity may be motivated by an interest in an unselective notion of causation, untroubled by principles of invidious discrimination. And causal relations appear to add up like transitive relations, so that the obtaining of the (...) overarching relation is not independent of the obtaining of the intermediaries. On the other hand, in favour of Sometimes , often we seem not to treat events that are very spatiotemporally remote from an effect as its causes, even when connected to the effect in question by a chain of counterfactual or chance-raising dependence. Moreover cases of double prevention provide counterexamples to causal transitivity even over short chains. According to the argument of this paper, causation is non-transitive. Transitizing causation provides no viable account of causal redundancy. An unselective approach to causation may motivate resisting the distance counterexamples to transitivity, but it does not help with double prevention, and even makes it more intractable. The strongest point in favour of transitivity is the adding up of causal relations, and this is the point that extant non-transitizing analyses have not adequately addressed. I propose a necessary condition on causation that explains the adding up phenomenon. In doing so it also provides a unifying explanation of distance and double prevention counterexamples to transitivity. (shrink)

A discussion of Aristotle’s thought on determinism and culpability, Necessity, Cause, and Blame also reveals Richard Sorabji’s own philosophical commitments. He makes the original argument here that Aristotle separates the notions of necessity and cause, rejecting both the idea that all events are necessarily determined as well as the idea that a non-necessitated event must also be non-caused. In support of this argument, Sorabji engages in a wide-ranging discussion of explanation, time, free will, essence, and purpose in nature. He also (...) provides historical perspective, arguing that these problems remain intimately bound up with modern controversies. “ Necessity, Cause and Blame would be counted by all as one of Sorabji’s finest. The book is essential for philosophers—both specialists on the Greeks and modern thinkers about free will—and also compelling for non-specialists.”—Martha Nussbaum “Original and important . . . The book relates Aristotle’s discussions to both the contemporary debates on determinism and causation and the ancient ones. It is especially detailed on Stoic arguments about necessity . . . and on the social and legal background to Aristotle’s thought.”— Choice “It is difficult to convey the extraordinary richness of this book. . . . A Greekless philosopher could read it with pleasure . . . At the same time, its learning and scholarship are enormous.”—G. E. M. Anscombe, Times Literary Supplement. (shrink)

The causal theory of action is widely recognized in the literature of the philosophy of action as the "standard story" of human action and agency -- the nearest approximation in the field to a theoretical orthodoxy. This volume brings together leading figures working in action theory today to discuss issues relating to the CTA and its applications, which range from experimental philosophy to moral psychology. Some of the contributors defend the theory while others criticize it; some draw from historical sources (...) while others focus on recent developments; some rely on the tools of analytic philosophy while others cite the latest empirical research on human action. All agree, however, on the centrality of the CTA in the philosophy of action. The contributors first consider metaphysical issues, then reasons-explanations of action, and, finally, new directions for thinking about the CTA. They discuss such topics as the tenability of some alternatives to the CTA; basic causal deviance; the etiology of action ; teleologism and anticausalism; and the compatibility of the CTA with theories of embodied cognition. Two contributors engage in an exchange of views on intentional omissions that stretches over four essays, directly responding to each other in their follow-up essays. As the action -oriented perspective becomes more influential in philosophy of mind and philosophy of cognitive science, this volume offers a long-needed debate over foundational issues. Contributors: Fred Adams, Jesús H. Aguilar, John Bishop, Andrei A. Buckareff, Randolph Clarke, Jennifer Hornsby, Alicia Juarrero, Alfred R. Mele, Michael S. Moore, Thomas Nadelhoffer, Josef Perner, Johannes Roessler, David-Hillel Ruben, Carolina Sartorio, Michael Smith, Rowland Stout The hardcover edition does not include a dust jacket. (shrink)

This paper is about the history of a question in ancient Greek philosophy and medicine: what holds the parts of a whole together? The idea that there is a single cause responsible for cohesion is usually associated with the Stoics. They refer to it as the synectic cause (αἴτιον συνεκτικόν), a term variously translated as ‘cohesive cause,’ ‘containing cause’ or ‘sustaining cause.’ The Stoics, however, are neither the first nor the only thinkers to raise this question or to propose a (...) single answer. Many earlier thinkers offer their own candidates for what actively binds parts together, with differing implications not only for why we are wholes rather than heaps, but also why our bodies inevitably become diseased and fall apart. This paper assembles, up to the time of the Stoics, one part of the history of such a cause: what is called ‘the synechon’ (τὸ συνέχον) – that which holds things together. Starting with our earliest evidence from Anaximenes (sixth century BCE), the paper looks at different candidates and especially the models and metaphors for thinking about causes of cohesion which were proposed by different philosophers and doctors including Empedocles, early Greek doctors, Diogenes of Apollonia, Plato and Aristotle. My goal is to explore why these candidates and models were proposed and how later philosophical objections to them led to changes in how causes of cohesion were understood. (shrink)

This is the earliest critical discussion in the context of modern/contemporary philosophy in the analytical tradition arguing that somebody with a reasonably stable character and the company of the right people would be able to enjoy eternity.

We propose a new definition of actual causes, using structural equations to model counterfactuals. We show that the definition yields a plausible and elegant account of causation that handles well examples which have caused problems for other definitions and resolves major difficultiesn in the traditional account.

Causal pluralists hold that that there is not just one determinate kind of causation. Some causal pluralists hold that ‘cause’ is ambiguous among these different kinds. For example, Hall (2004) argues that ‘cause’ is ambiguous between two causal relations, which he labels dependence and production. The view that ‘cause’ is ambiguous, however, wrongly predicts zeugmatic conjunction reduction, and wrongly predicts the behaviour of ellipsis in causal discourse. So ‘cause’ is not ambiguous. If we are to disentangle causal pluralism from the (...) ambiguity claim, we need to consider what other linguistic approaches are available to the causal pluralist. I consider and reject proposals that ‘cause’ is a general term, that the term is an indexical, and that the term conveys different kinds of causation through implicature or presupposition. Finally, I argue that causal pluralism is better handled by treating ‘cause’ as a univocal term within a dynamic interpretation framework. (shrink)

It may seem obvious that causing disability in another person is morally problematic in a way that removing or preventing a disability is not. This suggests that there is a moral asymmetry between causing disability and causing non-disability. This chapter investigates whether there are any differences between these two types of actions that might explain the existence of a general moral asymmetry. After setting aside the possibility that having a disability is almost always bad or harmful for a person (a (...) view that we have critiqued at length elsewhere), seven putative differences are considered. Ultimately, it is concluded that none of these seven factors can ground a general moral asymmetry between causing disability and causing non-disability, though each factor can provide some moral reason to avoid causing disability in certain particular cases. (shrink)

The financial crisis was caused by the complex, constantly growing web of regulations designed to constrain and redirect modern capitalism. This complexity made investors, bankers, and perhaps regulators themselves ignorant of regulations previously promulgated across decades and in different “fields” of regulation. These regulations interacted with each other to foster the issuance and securitization of subprime mortgages; their rating as AA or AAA; and their concentration on the balance sheets (and off the balance sheets) of many commercial and investment banks. (...) As a practical matter, it was impossible to predict the disastrous outcome of these interacting regulations. This fact calls into question the feasibility of the century-old attempt to create a hybrid capitalism in which regulations are supposed to remedy economic problems as they arise. (shrink)

There is an initial presumption against disjunctive causes. First of all, for some people causation is a relation between events. But, arguably, there are no disjunctive events, since events are particulars and thus they have spatiotemporal locations, while it is unclear what the spatiotemporal location of a disjunctive event could be.1 More importantly, even if one believes that entities like facts can enter in causal relations, and even if there are disjunctive facts, it is still hard to see how disjunctive (...) facts could be causes. Imagine, for instance, the following scenario. I have a gun filled with red paint and another gun filled with blue paint, and I fire both guns at my neighbor’s white wall. A moment later, there is a graffiti on the wall and my neighbor notifies the police. He would have done so regardless of the graffiti’s color, since all he cares about is the existence of a graffiti on his wall. Is it plausible to claim that a disjunctive fact is a cause of his notifying the police? In particular, is it plausible to claim that he notified the police because I fired the red-paint gun or the blue-paint gun (the thought being that my firing paint of either color would have sufficed)? It seems not. The police was notified because of the actual graffiti on the wall, and the actual graffiti on the wall is made of a certain pattern of colored patches. Imagine, that, as it turns out, there are patches of both colors on the wall. Then it seems that both my firing the red-paint gun and my firing the blue-paint gun were causes of my neighbor’s notifying the police. In other words, my firing the red-paint gun and my firing the blue-paint gun jointly caused the outcome: each of them was a contributory cause of the outcome’s occurrence. On the other hand, imagine that there are only patches of one color on the wall. Then it seems that my firing only one of the guns was a cause. Either way, the disjunction fails to be a cause: either my firing the red-paint gun was a cause, or my firing the blue-paint gun was a cause, or they were both causes, but their disjunction was not.. (shrink)

This chapter presents a series of experiments that elicit causal judgments using statements that do not include the verb "to cause". In particular, our interest is in exploring the extent to which previously observed effects of normative considerations on agreement with what we call "cause"-statements, i.e. those of the form "X caused ..." extend as well to those of the form "X V-ed Y", where V is a lexical causative. Our principal finding is that in many cases the effects do (...) not extend in this way, and moreover that the cases where we do find the same pattern are those where the causal verb used has a negative valence of its own. We draw two main conclusions from this finding. First, it reveals how the almost exclusive focus on "cause"-statements in the experimental study of causal judgment has led to findings that are unrepresentative of the full range of ordinary causal thinking, and provides a proof of concept as to how this thinking can be studied in its full variety. Second, the results of our experiments provide significant indirect support for the contention that the effect of moral considerations on agreement with "cause"-statements reflect the fact that these statements are most often used to assign responsibility for an event, and not just to describe the causal structure of what happened. It is not causal judgments in general that result from a process in which normative considerations play a role, but perhaps only those judgments that express a determination of moral responsibility. (shrink)

Many philosophers are persuaded by familiar arguments that free will is incompatible with causal determinism. Yet, notoriously, past attempts to articulate how the right type of indeterminism might secure the capacity for autonomous action have generally been regarded as either demonstrably inadequate or irremediably obscure. This volume gathers together the most significant recent discussions concerning the prospects for devising a satisfactory indeterministic account of freedom of action. These essays give greater precision to traditional formulations of the problems associated with indeterministic (...) accounts and to the range of theoretical avenues for pursuing resolutions. The first four essays set out different challenges (from both compatibilists and those skeptical of the possibility of free will) to the adequacy of standard indeterministic theories. The next seven essays meet one or more of these challenges. Each of the fundamental types of approach--simple indeterminism, causal indeterminism, and agent causation--is represented in these novel and sophisticated proposals. The collection finishes with two essays that debate whether compatibilism entails that freedom of choice is a comparatively rare phenomenon within an individual's life. Eloquently presenting some of the most compelling and accessible arguments surrounding this central philosophical issue, Agents, Causes, and Events makes a valuable contribution to courses in free will/action theory and metaphysics. (shrink)

The interventionist account of causation offers a criterion to distinguish causes from non-causes. It also aims at defining various desirable properties of causal relationships, such as specificity, proportionality and stability. Here we apply an information-theoretic approach to these properties. We show that the interventionist criterion of causation is formally equivalent to non-zero specificity, and that there are natural, information-theoretic ways to explicate the distinction between potential and actual causal influence. We explicate the idea that the description of causes should be (...) proportional to that of their effects. Then we draw a distinction between two ideas in the existing literature, the range of invariance of a causal relationship and its stability. The range of invariance is related to specificity and range of causal values. Stability concerns the effect of additional variables on the relationship between some focal pair of cause and effect variables. We show how to distinguish and measure the direct influence of background variables on the effect variable, and their influence on the relationship between the focal cause and the effect variable. Finally, we discuss the limitations of the information-theoretic approach, and offer prospects for complementary approaches. (shrink)

Mechanistic explanations are often said to explain because they reveal the causal structure of the world. Conversely, dynamical models supposedly lack explanatory power because they do not describe causal structure. The only way for dynamical models to produce causal explanations is via the 3M criterion: the model must be mapped onto a mechanism. This framing of the situation has become the received view around the viability of dynamical explanation. In this paper, I argue against this position and show that dynamical (...) models can themselves reveal causal structure and consequently produce nonmechanistic, dynamical explanations. Taking the example of cell fates from systems biology, I show how dynamical models, and specifically the attractor landscapes they describe, identify the causes of cell differentiation and explain why cells select particular fates. These dynamical features of the system better fit Woodward’s (2010, 2018) criteria of specificity and proportionality and make them the best candidate causes of cell fates than mechanisms. I also show how these causes are irreducible and inaccessible to mechanistic models, making 3M unworkable and counterproductive in this case. Dynamical models can reveal dynamical causes and thereby provide causal explanations. (shrink)

In an important departure from theories of causation, David Owens proposes that coincidences have no causes, and that a cause is something which ensures that its effects are no coincidence. In Causes and Coincidences, he elucidates the idea of a coincidence as an event which can be analysed into constituent events, the nomological antecedents of which are independent of each other. He also suggests that causal facts can be analysed in terms of non-causal facts, including relations of necessity. Thus, causation (...) is defined in terms of coincidence, and coincidence without reference to causation. David Owens challenges the ideas associated with Hume, Davidson and Lewis, constructing a theory which distinguishes nomological necessity and sufficiency from their logical counterparts. He is able to offer novel solutions to the major problems of causation, including the direction of causation, the logical form of causal statements, the distinction betwen causal connections and logical connections, and the relationship between psychological and physical causation. (shrink)

We propose a new definition of actual causes, using structural equations to model counterfactuals. We show that the definition yields a plausible and elegant account of causation that handles well examples which have caused problems for other definitions and resolves major difficulties in the traditional account.

This book provides the first comprehensive account of Hume’s conception of objects in Book I of the Treatise. What, according to Hume, are objects? Ideas? Impressions? Mind-independent objects? All three? None of the above? Through a close textual analysis, I show that Hume thought that objects are imagined ideas. However, I argue that he struggled with two accounts of how and when we imagine such ideas. On the one hand, Hume believed that we always and universally imagine that objects are (...) the causes of our perceptions. On the other hand, he thought that we only imagine such causes when we reach a “philosophical” level of thought. This tension manifests itself in Hume’s account of personal identity; a tension that, I argue, Hume acknowledges in the Appendix to the Treatise. As a result of presenting a detailed account of Hume’s conception of objects, we are forced to accommodate new interpretations of, at least, Hume’s notions of belief, personal identity, justification and causality. (shrink)

It is instructive to view the scientific revolution from the point of view of Robert Boyle’s distinction between intermediate and ultimate causes. From this point of view, the scientific revolution involved the identification of intermediate causes and their investigation by way of experiment as opposed to the specification of ultimate causes of the kind involved in the corpuscular matter theories of the mechanical philosophers. The merits of this point of view are explored in this paper by focussing on the hydrostatics (...) of Pascal and Boyle, understood as the experimental investigation of the action of the intermediate causes weight and pressure. The distinctive features of this new science are highlighted by comparing it with two alternative versions of hydrostatics, that of Stevin and that of Descartes. (shrink)

I argue that the criterion of just cause is not independent of proportionality and other valid jus ad bellum criteria. One cannot know whether there is a just cause without knowing whether the other (valid) criteria (apart from ‘right intention’) are satisfied. The advantage of this account is that it is applicable to all wars, even to wars where nobody will be killed or where the enemy has not committed a rights violation but can be justifiably warred against anyway. This (...) account also avoids the inefficiency of having proportionality considerations come up at two different points: in a separate criterion of just cause and in the criterion of proportionality proper. ‘Right intention’, the subjective element of the justification of a war, on the other hand, is not to be subsumed under the criterion of just cause: there can be a just cause without anybody knowing it. Conversely, however, the subjective element requires that those responsible for waging the war do know that the justifying objective conditions are fulfilled. This is in one sense more demanding than traditional just war theory; in another sense, however, it is less demanding: nobody needs to intend to fight for a ‘just aim’. (shrink)

Within evolutionary biology a distinction is frequently made between proximate and ultimate causes. One apparently plausible interpretation of this dichotomy is that proximate causes concern processes occurring during the life of an organism while ultimate causes refer to those processes (particularly natural selection) that shaped its genome. But ultimate causes are not sought through historical investigations of an organisms lineage. Rather, explanations referring to ultimate causes typically emerge from functional analyses. But these functional analyses do not identify causes of any (...) kind, much less ultimate ones. So-called ultimate explanations are not about causes in any sense resembling those of proximate explanations. The attitude, implicit in the term ultimate cause, that these functional analyses are somehow superordinate to those involving proximate causes is unfounded. Ultimate causes are neither ultimate nor causes. (shrink)